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PHARMACOLOGY II NUR 354 EXAM 3 LATEST 2026 ACTUAL SPRING-SUMMER SEMESTER ARIZONA COLLEGE OF NURSING |SCORE A

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PHARMACOLOGY II NUR 354 EXAM 3 LATEST 2026 ACTUAL SPRING-SUMMER SEMESTER ARIZONA COLLEGE OF NURSING |SCORE A

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PHARMACOLOGY II NUR 354 EXAM 3
LATEST 2026 ACTUAL SPRING-SUMMER
SEMESTER ARIZONA COLLEGE OF
NURSING |SCORE A
WBC differential

all blood parts + WBC parts (neutrophils, lymphocytes, monocytes, basophils,

eosinophils)

Anemia

the decrease in the number of RBC's, quantity of hemoglobin, and/or volume of packed

cells- decrease in any of these leads to signs of hypoxia

blood loss, decreased production of RBCs, Increased destrution of RBCs

What are three prevalent causes of anemia?

Blood loss

>>Chronic hemorrhage - Bleeding duodenal ulcer, Colon cancer>>Acute trauma -cuts

decreased production of RBCs

deficient nutrients (iron, vitamin B12, folic acid), decreased erythropoietin (kidneys not

functioning, epogen medication), Increased destruction of RBCs (sickle cell anemia,

medication, incompatible blood, trauma)

acute anemia clinical manifestations

gets really bad at 40-50% volume loss mild - exertional dyspnea & palpations

Clincal manifestations of Chronic Anemia

moderate - fatigue, bounding pulse, roaring in ears; severe- pallor, systolic murmur,

headache, anorexia, vertigo

,iron deficiency anemia S&S

initally symptom free, pallor, glossitis, chelisitis, headache, paresthesias

iron deficiency anemia causes

most common, decreased RBC production, chronic blood loss, inadequate intake,

malabsorption, hemolysis

pernicious anemia S&S

insidious inset, anorexia, N/V, abdominal pain, weakness, paresthesias, Red flags for

Vitamin B12 deficiency: altered mental status, somnolence, depression

Red flags for Vitamin B12 deficiency:

altered mental status, somnolence, depression

pernicious anemia causes

megaloblastic anemia (large RBCs), lack of gastric intrinsic factors resulting in

decreased vitamin B12 absorption, familial tendency, high risk populations: >40 y/o,

Northern European (scandinavian) ancestry

aplastic anemia causes

bone marrow suppression, pancytopenia, congenital birth defects; Acquired: idiopathic,

chemicals, medications, infections

Aplastic anemia S&S

onset can be abrupt or gradual, mild to severe, pallor, fatigue, dyspnea, neutropenia,

thrombocytopenia, petechiae, epitaxis, ecchymosis

hemolytic anemia causes

sickle cell anemia, hemodialysis, prosthetic valves, transfusion reactions, autoimmune

hemolytic, malaria, clostridium perfringens

, hemolytic anemia S&S

jaundice, pallor, pale conjunctive, tachycardia, hypotension, splenomegaly

Hemolytic anemia S&S

jaundice, pallor, pale conjunctivae, tachycardia, hypotension, splenomegaly

Sickle cell anemia etiology

genetic, autosomal recessive

sickle cell "sickling" is caused by

low oxygen levels, increased blood acidity, or low blood volume

common sickle cell crisis triggers include

sudden change in temperature (narrowing blood vessels), strenuous activity or

excessive exercise (oxygen shortage)

upper respiratory tract

nose, pharynx, adenoids, tonsils, epiglottis, larynx, trachea

Nose

has a septum, divides 2 cavities, responsible for warming & moistening the air that goes

to your lungs, filters smaller particles that are in the air, connected with sinuses & ears

adenoids & tonsils

part of the lymph system, helpful in getting rid of infections that come in through the

nose

epiglottis

keeps fluid & food from going into lungs, blocks off trachea, if not working=aspiration

apiration

things getting into the lungs that should not be there

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