Prepare for NUR 355 Exam 1 (2026) with this comprehensive study guide featuring 350+ expertly compiled exam questions and 100% correct answers covering the foundational concepts of Pathophysiology and Pharmacology for nursing students. This resource provides an in-depth review of cell injury and cellular adaptation, plasma membrane injury, free radicals and antioxidants, oxidative stress, sodium-potassium pump, lysosomes, autolysis, ischemia, necrosis, apoptosis, stem cells, pharmacokinetics, pharmacodynamics, drug absorption, distribution, metabolism and excretion (ADME), first-pass metabolism, bioavailability, half-life, receptor theory, therapeutic index, drug interactions, pharmacogenetics, genomics, stress response, General Adaptation Syndrome (GAS), aging physiology, polypharmacy, infection prevention, innate and adaptive immunity, bacterial and viral pathogens, inflammatory response, wound healing, NSAIDs, corticosteroids, cytokines, fever, chemotaxis, and evidence-based nursing interventions. The structured question-and-answer format reinforces critical thinking, strengthens clinical reasoning, and prepares students for classroom examinations, comprehensive nursing assessments, and NCLEX-style testing.
Aligned with the learning outcomes of NUR 355, this study guide integrates essential concepts in pathophysiology, pharmacology, immunology, microbiology, inflammation, and nursing science into a comprehensive exam review resource. Learners will strengthen their understanding of cellular injury mechanisms, adaptive cellular responses, pharmacologic principles, host defense systems, infectious disease processes, inflammatory mediators, wound healing physiology, medication safety, stress adaptation, aging-related physiologic changes, and evidence-based patient care. The material combines high-yield review questions with clinically relevant scenarios, making it an excellent resource for exam preparation, nursing coursework, and NCLEX-RN review.
The content is consistent with current evidence-based nursing education and authoritative references, including:
McCance, K. L., & Huether, S. E. Pathophysiology: The Biologic Basis for Disease in Adults and Children.
Lewis, S. L., Bucher, L., Heitkemper, M. M., Harding, M., Kwong, J., & Roberts, D. Medical-Surgical Nursing: Assessment and Management of Clinical Problems.
Lilley, L. L., Rainforth Collins, S., & Snyder, J. S. Pharmacology and the Nursing Process.
Katzung, B. G. Basic & Clinical Pharmacology.
Ignatavicius, D. D., Workman, M. L., & Rebar, C. R. Medical-Surgical Nursing: Concepts for Interprofessional Collaborative Care.
National Council of State Boards of Nursing (NCSBN). NCLEX-RN® Test Plan.
This study resource is recommended for:
NUR 355 Students
BSN Nursing Students
ADN Nursing Students
Pre-Licensure Nursing Students
Registered Nurse (RN) Students
Licensed Practical/Vocational Nursing (LPN/LVN) Students
Pathophysiology Students
Pharmacology Students
Medical-Surgical Nursing Students
NCLEX-RN Candidates
ATI Examination Candidates
Healthcare students preparing for nursing examinations
Keywords
NUR 355 Exam 1 2026, NUR 355 Exam Questions, NUR 355 Study Guide, Pathophysiology, Pharmacology, Cell Injury, Cellular Adaptation, Free Radicals, Antioxidants, Oxidative Stress, Sodium Potassium Pump, Necrosis, Apoptosis, Ischemia, Stem Cells, Pharmacokinetics, Pharmacodynamics, Drug Absorption, Drug Distribution, Drug Metabolism, Drug Excretion, ADME, First Pass Effect, Bioavailability, Half Life, Therapeutic Index, Drug Interactions, Pharmacogenetics, Genomics, Stress Response, General Adaptation Syndrome, Aging Physiology, Polypharmacy, Infection Control, Innate Immunity, Adaptive Immunity, Inflammation, Wound Healing, NSAIDs, Corticosteroids, Cytokines, Nursing Pharmacology, Nursing Pathophysiology, NCLEX Review, Nursing Exam Questions, Verified Answers
Content preview
NUR 355 Exam 1 2026 Exam
Questions with 100% Correct
Answers | Latest Update
4 injurious agents of the plasma membrane: - ANSWER ✔✔1. free
radicals (substances with free electron on an oxygen molecule)
2. high pressure forces (ex: hypertension)
3. radiation (ex: sunlight)
4. high glucose concentrations (ex: uncontrolled diabetes)
,How are free radicals generated? - ANSWER ✔✔during inefficient
mitochondrial metabolism
Free radicals are sometimes called "_____________" molecules. The
free electron is attracted to cell membranes, attaches to membranes,
and damages them. The body has natural _______________ that
destroy these destructive molecules. - ANSWER ✔✔superoxide;
antioxidants
Free radicals _________ cell structures, disrupt the integrity of the cell
membrane and damage organelles and DNA, causing cell dysfunction,
therefore they can be called _________. - ANSWER ✔✔oxidize;
oxidants
4 examples of antioxidants: - ANSWER ✔✔1. Vitamin A, E, C
2. beta- carotene
The sodium potassium pump constantly pumps Na+ _____ in exchange
for K+ ____. - ANSWER ✔✔out; in
organelle that contains digestive enzymes and digests particles brought
in by endocytosis, pinocytosis, or phagocytosis. Also digests worn out
cell parts. - ANSWER ✔✔lysosomes
,In cell death,__________ occurs where the enzymes rupture from
lysosome and digest whole cell. WBC's have many of _________. -
ANSWER ✔✔autolysis; lysosomes
In a MI, autolysis occurs where the enzymes rupture from cardiac
muscle cell lysosomes to digest dead cardiac muscle cells. The 3 serum
cardiac enzymes are: - ANSWER ✔✔CPK/LDH/SGOT
Injured cells can develop ___________ or ____________ changes to
maintain homeostasis. - ANSWER ✔✔adaptive; maladaptive
terms that means the original cause of a cellular alteration or disease -
ANSWER ✔✔etiology
term that means unique histological findings that represent distinct
disease process. - ANSWER ✔✔pathognomonic changes
means condition or change in - ANSWER ✔✔-trophy
means development or formation - ANSWER ✔✔-plasia
Decrease in cell size due to not using the muscle. If the body doesn't
use that muscle, it's not going to supply nutrients to that muscle. ex:
paralysis, injury and lack of use - ANSWER ✔✔muscle atropy
COPYRIGHT©PROFFKERRYMARTIN 2025/2026. YEAR PUBLISHED 2026. COMPANY REGISTRATION NUMBER: 619652435. TERMS OF USE.
PRIVACY STATEMENT. ALL RIGHTS RESERVED
, Increase in cell size due to overusing muscle. ex: working out, steroids,
etc. - ANSWER ✔✔hypertrophy
Increase in cardiac muscle because it has to work harder. It has an
increased need for oxygen and blood show which causes inadequate
blood flow and can lead to ischemia. Can be caused by hypertension. -
ANSWER ✔✔ventricular hypertrophy
term that means increase in the number of cells. ex: pregnancy when
there is an increase of breast gland cells caused by estrogen
stimulation, callous, endometriosis (increase in uterine lining) -
ANSWER ✔✔hyperplasia
term that means one cell type is replaced by another. Can be an
indication of cancer due to diff. types of cells. ex: keloid on skin, cells at
gastroesophageal junction irritated by acid reflux from stomach -
ANSWER ✔✔metaplasia
term that means cells have lost normal arrangement (shape, function,
size), can be precancerous changes (not necessarily cancerous growth).
Ex: hpv virus causing precancerous cervical cells - ANSWER
✔✔dysplasia