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NSG 3850 exam 4 Questions & Answers _ Latest Already Graded A+ _Questions with Correct Answers 2026 latest update!! .pdf

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NSG 3850 exam 4 Questions & Answers |
Latest Already Graded A+ |Questions with
Correct Answers 2026 latest update!!
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NSG 3850 exam 4 Questions & Answ... Exam 4 NSG 3850 LATEST VERSION... Patho Exam 4 review questions Exam 4

Teacher 104 terms Teacher 54 terms 50 terms 54 terms




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diagnostics: typically CT, US, or MRI, but can use HIDA, MRCA, ERCP



Choose an answer



1 a and b 2 pancreatitis



3 hepatitis 4 cholecystitis



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Terms in this set (104) Hide definitions




cholelithiasis three phases: supersaturation of bile with cholesterol causing precipitation of
cholesterol, nucleation of crystals, hypermobility (stasis of bile) allowing stone
growth

, true T/F: crystals of cholesterol may ignite gallstone formation


true T/F: excessive somatostatin stimulates the gallbladder




true T/F: crystals of cholesterol may initiate gallstone formation


cholelithiasis risk factors of this include prolonged fasting, rapid weight loss, oral
contraceptives, diabetes, spinal cord injury, TPN, pregnancy


cholecystitis inflammation of the gallbladder wall related to continued presence of gallstones


acute cholecystitis if untreated, escalates; gangrene may occur. can rupture gallbladder and cause
peritonitis


chronic cholecystitis predisposing factors: diabetes and obesity


chronic cholcystitis can lead to complications like biliary sepsis and scarring/calcified (porcelain
gallbladder); causes higher risk for cancer.


cholecystitis diagnostics: typically CT, US, or MRI, but can use HIDA, MRCA, ERCP


liver disease S/S: jaundice (from impaired bilirubin metabolism), decreased clotting factors,
hypoalbuminemia (from ascites/edema related to low serum oncotic pressure),
portal HTN, muscle wasting, ascites, impaired absorption of vit A, D, E, K,
hypertriglyceridemia


liver disease can disrupt lipoprotein metabolism causing dyslipidemias.




"anti" with hepatitis, what term means immunity or forming immunity to the specific hep
virus


hep A and E heps that are fecal oral route

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