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MARYVILLE NURS 611 PATHO EXAM 2 |COMPLETE QUESTIONS WITH EXPERT SOLUTIONS| 2026 LATEST UPDATED| A+

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MARYVILLE NURS 611 PATHO EXAM 2 |COMPLETE QUESTIONS WITH EXPERT SOLUTIONS| 2026 LATEST UPDATED| A+

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MARYVILLE NURS 611 PATHO EXAM 2 |COMPLETE QUESTIONS

WITH EXPERT SOLUTIONS| 2026 LATEST UPDATED| A+

Which body system is responsible for conserving energy and body resources -

(answer)Parasympathetic nervous system




which system responds to stress by preparing the body to defend itself - (answer)Sympathetic

nervous system (SNS)




how is blood flow redistributed by the sympathetic nervous system (SNS) - (answer)blood flow

to the muscles is increased while blood flow to GI and integumentary is decreased




how are primary brain injuries classified - (answer)focal or diffuse (aka multifocal)




focal brain injuries - (answer)specific, grossly observable brain lesions that occur in a precise

location

Epidural and subdural hemorrhages




diffuse brain injuries - (answer)include brain injury due to hypoxia, meningitis, encephalitis, and

damage to blood vessels

,The brain is confined in a limited space so increased pressure can cause collateral dysfunction:

Diabetes Insipidus (ADH not secreted thus polyuria)




autonomic hyperreflexia - (answer)characterized by paroxysmal HTN (up to 300 mmHg

systolic), a pounding headache, blurred vision, sweating above the level of the lesion with

flushing of the skin, nasal congestion, nausea, piloerection caused by pilomotor spasm, and

bradycardia (30-40 beats/min)




location of lesions in cases of autonomic hyperreflexia - (answer)individual most likely to be

affected have lesions at the T5-T6 level or above




sequence of events that lead to hyperreflexia induced bradycardia - (answer)bradycardia (30-

40bpm) is a sx of hyperreflexia

Stimulation of the carotid sinus -->vagus nerve -->sinoatrial (SA) node. The intact ANS

reflexively responds with an arteriolar spasm that increases blood pressure. Baroreceptors in the

cerebral vessels, the carotid sinus, and the aorta sense the HTN and stimulate the PNS. The heart

rate decreases, but the visceral and peripheral vessels do not dilate because efferent impulses

cannot pass through the cord




Alzheimer's disease - (answer)leading cause of dementia and one of the most common causes of

severe cognitive dysfunction in older adults

,what are the greatest risk factors for Alzheimer's disease - (answer)age, family history




what are the proposed protective factors for Alzheimer's disease - (answer)low calorie diets,

estrogen replacement at time of menopause, NSAIDs, physical activity, antioxidants, the

presence of apoE2




what genetic susceptibility tests are used to screen for early-onset AD - (answer)PSEN 1

(presenilin) on chromosome 14, PSEN 2, and APP (amyloid precursor protein) on chromosome

21




When can a specific diagnosis of AD be given - (answer)postmortem examination




what is the single greatest risk factor for stroke - (answer)hypertension




what are common risk factors for stroke - (answer)arterial HTN, insulin resistance and DM,

elevated cholesterol or low high density lipoprotein (HDL), elevated lipoprotein- A level,

hyperhomocysteinemia, congestive heart disease and PVD, asymptomatic carotid stenosis,

polycythemia and thrombocythemia, a-fib, postmenopausal hormone therapy, high sodium intake

, above 2300mg, low potassium intake less than 4700mg, smoking, lack of physical activity,

obesity, chronic sleep deprivation




which autoimmune disease typically presents 2-4 weeks following a bacterial/viral infection such

as respiratory or GI illness - (answer)Guillain-Barre syndrome




Describe the progression of Guillain-Barre symptoms - (answer)typical first manifestations are

numbness, pain, paresthesias, or weakness in the limbs. Paresis/paralysis may present in an

ascending pattern




when can improvement be expected with Guillain-Barre - (answer)weakness usually plateaus or

improves by the 4th week in 90% of cases




Myasthenia Gravis - (answer)a chronic autoimmune disease that is mediated by Ach receptor

antibodies that act at the neuromuscular junction. The antibodies prevent normal reception for

muscle contraction




Graves disease - (answer)Graves disease is a result of autoantibodies binding to the TSH receptor

sites. This leads to hyperthyroidism

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