NUR 633 Exam 1 Study Guide | Questions
and Answers | 2026 Update | 100%
Correct - D'Youville College
inflammation
reaction of vascularized tissue to injury that allows for repair of injured tissue.
Conditions are named by adding the suffix -itis to the affected organ/system
Cardinal signs of inflammation
Rubor (redness)
Tumor (swelling)
Calor (heat)
Dolor (pain)
Functio laesa (Loss of function)
may also include fever
acute inflammation
Short duration of non-specific response to injury. Involves the infiltration of neutrophils
and exudate is often present. Involves 2 major component; the vascular stage and the
cellular stage
vascular stage
The first stage of response to injury when blood flow increaes, blood vessels have
momentary vasoconstriction and then vasodilation
the cellular stage
,leukocytes/neutrophils consume debris; damaged cells are repaired. Aided by the
changes in the endothelial cells lining and the release of chemical mediators from mast
cells and macrophages.
immediate transient response
Vascular response that occurs rapidly and only lasts a short duration- typically
reversible
immediate sustained response
Vascular response that occurs with more serious injury and continues for several days
and damages the vessels in the area
delayed hemodynamic response
Involves an increase in capillary permeability that occurs 4 to 24 hours after injury due
to delayed cell damage. Often due to radiation like sunburn
chemotaxis
movement by a cell or organism in reaction to a chemical stimulus (chemokines for
leukocytes)
bands
immature white blood cells (granulocytes)
granulocytes
A neutrophil that destroys engulfed microbes and dead tissue
monocytes
Largest circulating leukocytes that engulf and digest microorganisms- essential in
clearing of bacteria
eosinophils
, A leukocyte that is responsible for antigen antibody complexes and virus response
basophils
a leukocyte that binds antibodies and immunoglobulin E which triggers release of
histamine and vasoactive substances. It is involved in parasitic and allergic responses
mast cells
leukocytes that releases histamine, cytokines, interleukin and other substances.
Involved in IGE triggered reactions.
histamine
Chemical stored in mast cells that triggers dilation of arterioles and increased
permeability of capillaries.
cytokines
Chemicals released by the immune system that control the immune response. They are
important in acute and chronic inflammation and communication between cells.
arachidonic acid metabolites
a chemical that causes inflammation through the production of prostaglandins and
leukotrienes
chronic inflammation
continuous injury or irritation to tissue that last days-years and is self-perpetuating. Can
be caused by TB, syphilis, foreign bodies and surgial materials
inflammatory phase
the initial phase of wound healing and involves the constriction of the injured vessel,
clotting, then vasodilation to allow plasma and blood to leak into the injured area. Also
and Answers | 2026 Update | 100%
Correct - D'Youville College
inflammation
reaction of vascularized tissue to injury that allows for repair of injured tissue.
Conditions are named by adding the suffix -itis to the affected organ/system
Cardinal signs of inflammation
Rubor (redness)
Tumor (swelling)
Calor (heat)
Dolor (pain)
Functio laesa (Loss of function)
may also include fever
acute inflammation
Short duration of non-specific response to injury. Involves the infiltration of neutrophils
and exudate is often present. Involves 2 major component; the vascular stage and the
cellular stage
vascular stage
The first stage of response to injury when blood flow increaes, blood vessels have
momentary vasoconstriction and then vasodilation
the cellular stage
,leukocytes/neutrophils consume debris; damaged cells are repaired. Aided by the
changes in the endothelial cells lining and the release of chemical mediators from mast
cells and macrophages.
immediate transient response
Vascular response that occurs rapidly and only lasts a short duration- typically
reversible
immediate sustained response
Vascular response that occurs with more serious injury and continues for several days
and damages the vessels in the area
delayed hemodynamic response
Involves an increase in capillary permeability that occurs 4 to 24 hours after injury due
to delayed cell damage. Often due to radiation like sunburn
chemotaxis
movement by a cell or organism in reaction to a chemical stimulus (chemokines for
leukocytes)
bands
immature white blood cells (granulocytes)
granulocytes
A neutrophil that destroys engulfed microbes and dead tissue
monocytes
Largest circulating leukocytes that engulf and digest microorganisms- essential in
clearing of bacteria
eosinophils
, A leukocyte that is responsible for antigen antibody complexes and virus response
basophils
a leukocyte that binds antibodies and immunoglobulin E which triggers release of
histamine and vasoactive substances. It is involved in parasitic and allergic responses
mast cells
leukocytes that releases histamine, cytokines, interleukin and other substances.
Involved in IGE triggered reactions.
histamine
Chemical stored in mast cells that triggers dilation of arterioles and increased
permeability of capillaries.
cytokines
Chemicals released by the immune system that control the immune response. They are
important in acute and chronic inflammation and communication between cells.
arachidonic acid metabolites
a chemical that causes inflammation through the production of prostaglandins and
leukotrienes
chronic inflammation
continuous injury or irritation to tissue that last days-years and is self-perpetuating. Can
be caused by TB, syphilis, foreign bodies and surgial materials
inflammatory phase
the initial phase of wound healing and involves the constriction of the injured vessel,
clotting, then vasodilation to allow plasma and blood to leak into the injured area. Also
