NSG-318 Exam LATEST
QUESTIONS AND VERIFIED
CORRECT ANSWERS 100%
GUARANTEED PASS
sympathomimetic - CORRECT ANSWER-agent that mimics the effects of the sympathetic
nervous system
Parasympatholytic - CORRECT ANSWER-agent blocking or inhibiting the effects of the
parasympathetic nervous system
alpha 1 receptors - CORRECT ANSWER--inc cardiac contractility; vasoconstriction (inc BP)
-dilates pupils, dec salivary gland secretion
-inc bladder and prostate cx
alpha 2 receptors - CORRECT ANSWER--inhibits NE release (promotes vasodilation and dec BP)
-dec GI motility and tone
beta 1 receptors - CORRECT ANSWER--inc cardiac contractility, HR
-inc renin secretion and BP
beta 2 receptors - CORRECT ANSWER--dec GI tone and motility
-bronchodilation
-inc blood flow in skeletal muscles
,-relaxes smooth muscles of uterus
-activates liver glycogenolysis (inc blood glucose)
Direct acting adrenergic agonists - CORRECT ANSWER-directly activates adrenergic receptor
(epinephrine, norepinephrine, dopamine)
indirect acting adrenergic agonists - CORRECT ANSWER-stimulates release of NE from terminal
nerve endings (amphetamine)
mixed acting adrenergic agonists - CORRECT ANSWER-stimulates adrenergic receptor sites and
stimulates release of NE from terminal nerve endings (ephedrine)
adrenergic agonists - CORRECT ANSWER-Catecholamines: produce sympathetic response
- endogenous (epinephrine, NE, dopamine)
-synthetic (isoproterenol, dobutamine)
Noncatecholamines: stimulate adrenergic receptors. most have longer duration of action than
endogenous and synthetic (phenylephrine, metaproterneol, albuterol)
Epinephrine - CORRECT ANSWER--CLASSIFICATION: adrenergic agonist
-USES: anaphylaxis, bronchospasms, status asthmaticus, cardiac arrest
-ACTION: Alpha1 inc BP, Beta1 inc HR, Beta2 promotes bronchodilation
-CONTRAINDICATIONS: cardiac dysrhythmias, hypertension, hyperthyroidism, DM, pregnancy
-SIDE/ADVERSE EFFECTS: cardiac (palpitation, tachycardia, hypertension), restlessness,
hyperglycemia, dyspnea, tissue necrosis at IV site, vfib, PE
,-INTERACTIONS: beta blockers, digoxin, TCA and MAOIs
-INTERVENTIONS: monitor IV site, monitor vitals and EKG, monitor I&Os and assess for bladder
distention, avoid nausea and vomiting for pt, monitor blood glucose in pt with DM
adrenergic antagonists - CORRECT ANSWER-block effects of adrenergic neurotransmitter (block
alpha and beta receptor sites)
-alpha-adrenergic agonists and beta adrenergic agonists
alpha1 blockers - CORRECT ANSWER--vasodilation/dizziness
-orthostatic hypotension, reflex tachycardia
-pupil constrict
-suppress ejaculation
-dec cx of smooth muscles in bladder, neck, and prostate
beta1 blockers - CORRECT ANSWER--dec cardiac contractility
-dec pulse
beta2 blockers - CORRECT ANSWER--bronchoconstriction
-cx uterus
-inhibits glycogenolysis (leads to hypoglycemia)
alpha adrenergic antagonists - CORRECT ANSWER-drugs that inhibit a response at alpha-
adrenergic receptor site
-selective (block alpha1)
-nonselective (block alpha1 and alpha2)
, -action (promote vasodilation)
-use (dec symptoms of BPH)
albuterol - CORRECT ANSWER--CLASSIFICATION: beta2 adrenergic agonist
-USE: tx of asthma, prophylactic tx for asthma, tx of bronchospasms
-ACTION: stimulates beta2 adrenergic receptors in lungs (relaxes bronchial smooth muscle)
bronchodilation
Beta-adrenergic agonists - CORRECT ANSWER--dec BP and pulse
-nonselective beta blockers
blocks beta1: dec BP and pulse
blocks beta2: bronchoconstriction, use with caution w pt with COPD or asthma
atenolol - CORRECT ANSWER--CLASS: beta adrenergic agonist
-USE: tx of hypertension, angina, and prophylaxis and tx of acute MI
-ACTION: selectively blocks beta1-adrenergic receptor sites, dec sympathetic outflow to the
periphery, suppresses RAAS
cholinergic agonists - CORRECT ANSWER-drugs that stimulate PNS
-cholinergic receptors: muscarinic (smooth muscles, slow HR), nicotinic (skeletal muscles)
-direct acting (activate)
-indirect acting (inhibits)
QUESTIONS AND VERIFIED
CORRECT ANSWERS 100%
GUARANTEED PASS
sympathomimetic - CORRECT ANSWER-agent that mimics the effects of the sympathetic
nervous system
Parasympatholytic - CORRECT ANSWER-agent blocking or inhibiting the effects of the
parasympathetic nervous system
alpha 1 receptors - CORRECT ANSWER--inc cardiac contractility; vasoconstriction (inc BP)
-dilates pupils, dec salivary gland secretion
-inc bladder and prostate cx
alpha 2 receptors - CORRECT ANSWER--inhibits NE release (promotes vasodilation and dec BP)
-dec GI motility and tone
beta 1 receptors - CORRECT ANSWER--inc cardiac contractility, HR
-inc renin secretion and BP
beta 2 receptors - CORRECT ANSWER--dec GI tone and motility
-bronchodilation
-inc blood flow in skeletal muscles
,-relaxes smooth muscles of uterus
-activates liver glycogenolysis (inc blood glucose)
Direct acting adrenergic agonists - CORRECT ANSWER-directly activates adrenergic receptor
(epinephrine, norepinephrine, dopamine)
indirect acting adrenergic agonists - CORRECT ANSWER-stimulates release of NE from terminal
nerve endings (amphetamine)
mixed acting adrenergic agonists - CORRECT ANSWER-stimulates adrenergic receptor sites and
stimulates release of NE from terminal nerve endings (ephedrine)
adrenergic agonists - CORRECT ANSWER-Catecholamines: produce sympathetic response
- endogenous (epinephrine, NE, dopamine)
-synthetic (isoproterenol, dobutamine)
Noncatecholamines: stimulate adrenergic receptors. most have longer duration of action than
endogenous and synthetic (phenylephrine, metaproterneol, albuterol)
Epinephrine - CORRECT ANSWER--CLASSIFICATION: adrenergic agonist
-USES: anaphylaxis, bronchospasms, status asthmaticus, cardiac arrest
-ACTION: Alpha1 inc BP, Beta1 inc HR, Beta2 promotes bronchodilation
-CONTRAINDICATIONS: cardiac dysrhythmias, hypertension, hyperthyroidism, DM, pregnancy
-SIDE/ADVERSE EFFECTS: cardiac (palpitation, tachycardia, hypertension), restlessness,
hyperglycemia, dyspnea, tissue necrosis at IV site, vfib, PE
,-INTERACTIONS: beta blockers, digoxin, TCA and MAOIs
-INTERVENTIONS: monitor IV site, monitor vitals and EKG, monitor I&Os and assess for bladder
distention, avoid nausea and vomiting for pt, monitor blood glucose in pt with DM
adrenergic antagonists - CORRECT ANSWER-block effects of adrenergic neurotransmitter (block
alpha and beta receptor sites)
-alpha-adrenergic agonists and beta adrenergic agonists
alpha1 blockers - CORRECT ANSWER--vasodilation/dizziness
-orthostatic hypotension, reflex tachycardia
-pupil constrict
-suppress ejaculation
-dec cx of smooth muscles in bladder, neck, and prostate
beta1 blockers - CORRECT ANSWER--dec cardiac contractility
-dec pulse
beta2 blockers - CORRECT ANSWER--bronchoconstriction
-cx uterus
-inhibits glycogenolysis (leads to hypoglycemia)
alpha adrenergic antagonists - CORRECT ANSWER-drugs that inhibit a response at alpha-
adrenergic receptor site
-selective (block alpha1)
-nonselective (block alpha1 and alpha2)
, -action (promote vasodilation)
-use (dec symptoms of BPH)
albuterol - CORRECT ANSWER--CLASSIFICATION: beta2 adrenergic agonist
-USE: tx of asthma, prophylactic tx for asthma, tx of bronchospasms
-ACTION: stimulates beta2 adrenergic receptors in lungs (relaxes bronchial smooth muscle)
bronchodilation
Beta-adrenergic agonists - CORRECT ANSWER--dec BP and pulse
-nonselective beta blockers
blocks beta1: dec BP and pulse
blocks beta2: bronchoconstriction, use with caution w pt with COPD or asthma
atenolol - CORRECT ANSWER--CLASS: beta adrenergic agonist
-USE: tx of hypertension, angina, and prophylaxis and tx of acute MI
-ACTION: selectively blocks beta1-adrenergic receptor sites, dec sympathetic outflow to the
periphery, suppresses RAAS
cholinergic agonists - CORRECT ANSWER-drugs that stimulate PNS
-cholinergic receptors: muscarinic (smooth muscles, slow HR), nicotinic (skeletal muscles)
-direct acting (activate)
-indirect acting (inhibits)
