NBME CBSE Actual exam COMPREHENSIVE QUESTIONS AND VERIFIED ANSWERS 2025(GRADED A+) DETAILED ANSWERS!!

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NBME CBSE Actual exam COMPREHENSIVE QUESTIONS AND VERIFIED ANSWERS 2025(GRADED A+) DETAILED ANSWERS!!

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NBME CBSE Actual exam COMPREHENSIVE QUESTIONS AND VERIFIED ANSWERS 2025(GRADED A+) DETAILED ANSWERS!!NBME CBSE Actual exam COMPREHENSIVE QUESTIONS AND VERIFIED ANSWERS 2025(GRADED A+) DETAILED ANSWERS!!

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NBME CBSE Actual exam COMPREHENSIVE
QUESTIONS AND VERIFIED ANSWERS
2025(GRADED A+) DETAILED ANSWERS!!
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Type II pneumocytes surfactant (lecithin)
Proliferate after injury
Type I progenitors
Neonatal Respiratory Distress Syndrome

Polio live v killed vaccine Killed = Salk = IgG

Live = Sabin = IgG + IgA
- can be shed in feces

Neonatal Respiratory Distress: Maternal DM (high insulin)
Etiology + Tx or C-section (low cortisol)
TX: dexamethasone before birth

Lung maturity determined with Amniocentesis of Phospholipids (*type II
pneumocytes)
L >> S

Type I pneumocytes Squamous gas diffusion

Elastase in lungs macrophage: lysosomes
PMN: azuronphilic granules

, Elastin stretches and recoils due to Lysine interchain crosslinks

air pressure and Air pressure = 0
intrapleural pressure at FRC Intrapleural pressure = -5

Pulm Vasc Resistance is lowest Exhale of Tidal Volume
during

Lung Compliance is decreased by LHF, pulmonary edema,
pulmonary fibrosis

Lung Compliance is increased by emphysema, age

Obesity affects ERV and FRC DECREASE
ERV & FRC

Blood flow/min (pulmonary v pulmonary = systemic
systemic)

Anatomic pulmonary shunting Bronchial circulation causes
decreased PO2 in LA/LV
than in pulmonary capillaries

More ventilation is at the BASE

O2-Hgb dissociation LEFT shift basic, cold, low 2,3 BPG
low pO2 (compensatory erythrocytosis)

O2-Hgb dissociation RIGHT shift low pH, high 2,3BPG, high T
HOT, ACIDIC

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