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NR 507 Advanced Pathophysiology Midterm Guide: Asthma (2026/2027) PDF | Nursing | Chamberlain

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INSTANT PDF DOWNLOAD. Complete NR 507 Advanced Pathophysiology midterm guide focusing on asthma pathophysiology. Covers airway inflammation, bronchial hyperreactivity, smooth muscle spasm, mucus production, bronchioles anatomy (3 layers, lamina propria), alveolar hyperinflation, and 5 signs/symptoms of asthma. Essential study resource for Chamberlain nursing students.

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NR 507 MIDTERM EXAM STUDY GUIDE PRACTICE

1.Which of the following are considered the "first responders" of the
innate
immune response?


A. Eosinophils


B. T-cells


C. IgM

D. Neutrophils: D. Neutrophils
2. Anaphylaxis is a hypersensitivity reaction.



A. type 1


B. type 2


C. type 3

D. type 4: A. Type 1
3. Type 2 (Cytotoxic) hypersensitivity reactions are mediated by:


A. IgE or IgG


B. IgA or IgE




,C. IgG or IgM

D. IgM or IgA: C. IgG or IgM
4. Hives (urticaria) are an example of a:


A. Type 1 hypersensitivity reaction.


B. Type 2 hypersensitivity reaction.


C. Type 3 hypersensitivity reaction.


D. Type 4 hypersensitivity reaction.: A. Type 1 hypersensitivity reaction.






,5. Allergic contact dermatitis is an example of hypersen-
sitivity reaction.


A. Type 4


B. Type 1


C. Type 2

D. Type 3: A. Type 4
6. Type 1 Hypersensitivity reaction Mechanism:



A. Cytotoxic T cell-mediated


B. IgE action on mast cells

C. Tissue-specific destruction or impairment because
of: Antibody binding followed by lysis via
complement Antibody binding followed by
macrophage phagocytosis Antibody binding
followed by neutrophil destruction Antibody-
dependent cell (NK)-mediated cytotoxicity, or
Antireceptor antibodies

D. Antigen-Antibody complex deposited in tissues: B. IgE action on mast
cells
7. Type 2 Hypersensitivity reaction Mechanism:



A. Cytotoxic T cell-mediated


, B. IgE action on mast cells

C. Tissue-specific destruction or impairment because
of: Antibody binding followed by lysis via
complement Antibody binding followed by
macrophage phagocytosis Antibody binding
followed by neutrophil destruction Antibody-
dependent cell (NK)-mediated cytotoxicity, or
Antireceptor antibodies

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