Advanced Physiology and Pathophysiology Test Bank 2nd Edition - Chapters 1-17

Advanced Physiology and Pathophysiology Essentials for
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Clinical Practice 1st Edition Tkacs Test Bank -Questions
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,with Answer Key At the end of Every Chapter, All Updated
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Chapter 1. THE FOUNDATIONAL CONCEPTS OF
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CLINICALPRACTICE
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1. The s s characteristic, s s localized s s cardinal s s signs s s of s s acute s s inflammation
s s include:
A) fever.

B) fatigue.
C) redness.
D) granuloma.


2. The s s vascular, s s hemodynamic s s stage s s of ssacute s s inflammation s s is
ssinitiated s s by ssmomentary s s vasoconstriction s s followed s s by

s s vasodilation s s that s s causes sslocalized:



A) bleeding.
B) congestion.
C) pale s s skin.
D) coolness.


3. The s s cellular s s stage s s of ssacute s s inflammation s s is s s marked s s by ssthe
s s movement s s of ssleukocytes s s into s s the s s area. s s Which s s of s s the

s s following s s cells s s arrives s s early s s in ssgreatnumbers?



A) Basophils
B) Lymphocytes
C) Neutrophils
D) Platelets

,4. The s s phagocytosis s s process s s involves s s three s s distinct s s steps.
s s What s s is s s the ssinitialstep ssin ssthe ssprocess?



A) Engulfment
B) Intracellular s s killing
C) Antigen s s margination
D) Recognition s s and s s adherence


5. Which s s of s s the s s following s s mediators s s of s s inflammation s s causes
s s increased sscapillarypermeability ssand sspain?



A) Serotonin
B) Histamine

C) Bradykinin
D) Nitric s s oxide


6. Inflammatory s s exudates s s are s s a s s combination s s of s s several s s types.
s s Which s s of ssthefollowing ssexudates ssis sscomposed s s of ssenmeshed

s s necrotic s s cells?


A) Serous
B) Fibrinous
C) Suppurative

D) Membranous


7. The s s acute-phase s s systemic s s response s s usually s s begins s s within
s s hours s s of s s the ssonset of ssinflammation ssand ssincludes:
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A) fever s s and s s lethargy.
B) decreased s s C-reactive s s protein.
C) positive s s nitrogen s s balance.
D) low s s erythrocyte s s sedimentation s s rate.

, 8. In s s contrast s s to s s acute s s inflammation, s s chronic s s inflammation s s is
s s characterized ssbywhich ssof ssthe s s following ssphenomena?



A) Profuse s s fibrinous s s exudation

B) A s s shift s s to s s the s s left s s of s s granulocytes
C) Metabolic s s and s s respiratory s s alkalosis
D) Lymphocytosis s s and s s activated s s macrophages


9. Exogenous s s pyrogens s s (interleukin-1) s s and s s the s s presence s s of
s s bacteria s s in s s the ssblood lead ssto ssthe ssrelease ssof ssendogenous sspyrogens
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ssthat:


A) stabilize s s thermal s s control s s in s s the s s brain.
B) produce s s leukocytosis s s and s s anorexia.

C) block s s viral s s replication s s in s s cells.
D) inhibit s s prostaglandin s s release.


10 An s s older s s adult s s patient s s has s s just s s sheared s s the s s skin s s on s s her
ss elbow s s while ssattempted
. to s s boost s s herself s s up s s in s s bed, s s an s s event s s that s s has
s s precipitated s s acute ssinflammation in s s the s s region s s surrounding bb



s s the s s wound. s s Which s s of ssthe ssfollowing s s events s s will ssoccur

s s during ssthe s s vascular s s stage s s of ssthe s s patients

inflammation?
A) Outpouring s s of s s exudate s s into s s interstitial s s spaces
B) Chemotaxis
C) Accumulation s s of s s leukocytes s s along s s the s s epithelium
D) Phagocytosis s s of s s cellular s s debris


11 Which s s of s s the s s following s s individuals s s most s s likely s s has s s the s s highest
s s risk s s of

. experiencing s s chronic s s inflammation?
A) A s s patient s s who s s has s s recently s s been s s diagnosed s s with s s type s s 2
s s diabetes


B) A s s patient s s who s s is s s a s s carrier s s of s s an s s antibiotic-resistant s s organism