MU NURS 611: Adv Patho -
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Exam 4 Review -
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Reproductive System
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primary dysmenorrhea - correct answer✔ ✔
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excessive endometrial protaglandin production; painful perio
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ds; produces 10x as much prostaglandin F (potent myometrial
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stimulant and vasoconstrictor)
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elevated levels of prostaglandins - correct answer✔ ✔
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uterine hypercontractility, decreased blood flow to uterus, in
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creased nerve hypersensitivity = pain
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secondary dysmenorrhea - correct answer✔ ✔
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results from endometriosis, endometritis (infection), pelvic i
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nflammatory disease, obstructive uterine/vaginal anomalies, ut
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erine fibroids, pulps, tumors, ovarian cysts, pelvic congestion
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syndrome, nonhormonal IUDs v7 v7
polycystic ovarian syndrome (PCOS) - correct answer✔ ✔
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cause unknown, may be genetic; no single factor fully acco
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unts for abnormalities
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, hyperandrogenic state - correct answer✔ ✔ v7 v7 v7 v7
cardinal feature in pathogenesis of PCOS; glucose intoleran
v7 v7 v7 v7 v7 v7 v7
ce/insulin resistance, hyperinsulinemia often run parallel and
v7 v7 v7 v7 v7 v7 v7
markedly aggravate it; affects follicular growth, insulin affect
v7 v7 v7 v7 v7 v7 v7
s follicular decline by suppressing apoptosis and enabling foll
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icles to survive v7 v7
benign ovarian cysts - correct answer✔ ✔
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produced when a follicle or # of follicles are stimulated by
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no dominant follicle develops and complete maturity process
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normal early follicular phase of menstrual cycle -
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v7correct answer✔ ✔ v7
follicles of ovary respond to hormonal signals from pituitar
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y gland; pituitary produces FSH to mature follicles -
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> as it enlarges, granulosa cells in follicle multiply and secret
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e estradiol -
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> as dominant follicle develops, secretes higher levels of estr
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adiol -> stimulates LH surge that comes from pituitary -
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> stimulates follicle to rupture, releasing ova and transformin
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g the granulosa cells of dominant follicle into corpus luteum
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-
> corpus luteum becomes vascularized and secretes progester
v7 v7 v7 v7 v7 v7 v7
one - v7
> arrests development of other follicles in both ovaries; LH,
v7 v7 v7 v7 v7 v7 v7 v7 v7 v7
v7 v7 v7 v7 v7
Exam 4 Review -
v7 v7 v7 v7
Reproductive System
v7 v7
primary dysmenorrhea - correct answer✔ ✔
v7 v7 v7 v7
excessive endometrial protaglandin production; painful perio
v7 v7 v7 v7 v7
ds; produces 10x as much prostaglandin F (potent myometrial
v7 v7 v7 v7 v7 v7 v7 v7
stimulant and vasoconstrictor)
v7 v7 v7
elevated levels of prostaglandins - correct answer✔ ✔
v7 v7 v7 v7 v7 v7
uterine hypercontractility, decreased blood flow to uterus, in
v7 v7 v7 v7 v7 v7 v7
creased nerve hypersensitivity = pain
v7 v7 v7 v7
secondary dysmenorrhea - correct answer✔ ✔
v7 v7 v7 v7
results from endometriosis, endometritis (infection), pelvic i
v7 v7 v7 v7 v7 v7
nflammatory disease, obstructive uterine/vaginal anomalies, ut
v7 v7 v7 v7 v7
erine fibroids, pulps, tumors, ovarian cysts, pelvic congestion
v7 v7 v7 v7 v7 v7 v7 v7
syndrome, nonhormonal IUDs v7 v7
polycystic ovarian syndrome (PCOS) - correct answer✔ ✔
v7 v7 v7 v7 v7 v7
cause unknown, may be genetic; no single factor fully acco
v7 v7 v7 v7 v7 v7 v7 v7 v7
unts for abnormalities
v7 v7
, hyperandrogenic state - correct answer✔ ✔ v7 v7 v7 v7
cardinal feature in pathogenesis of PCOS; glucose intoleran
v7 v7 v7 v7 v7 v7 v7
ce/insulin resistance, hyperinsulinemia often run parallel and
v7 v7 v7 v7 v7 v7 v7
markedly aggravate it; affects follicular growth, insulin affect
v7 v7 v7 v7 v7 v7 v7
s follicular decline by suppressing apoptosis and enabling foll
v7 v7 v7 v7 v7 v7 v7 v7
icles to survive v7 v7
benign ovarian cysts - correct answer✔ ✔
v7 v7 v7 v7 v7
produced when a follicle or # of follicles are stimulated by
v7 v7 v7 v7 v7 v7 v7 v7 v7 v7 v7
no dominant follicle develops and complete maturity process
v7 v7 v7 v7 v7 v7 v7
normal early follicular phase of menstrual cycle -
v7 v7 v7 v7 v7 v7 v7
v7correct answer✔ ✔ v7
follicles of ovary respond to hormonal signals from pituitar
v7 v7 v7 v7 v7 v7 v7 v7
y gland; pituitary produces FSH to mature follicles -
v7 v7 v7 v7 v7 v7 v7 v7
> as it enlarges, granulosa cells in follicle multiply and secret
v7 v7 v7 v7 v7 v7 v7 v7 v7 v7
e estradiol -
v7 v7
> as dominant follicle develops, secretes higher levels of estr
v7 v7 v7 v7 v7 v7 v7 v7 v7
adiol -> stimulates LH surge that comes from pituitary -
v7 v7 v7 v7 v7 v7 v7 v7 v7
> stimulates follicle to rupture, releasing ova and transformin
v7 v7 v7 v7 v7 v7 v7 v7
g the granulosa cells of dominant follicle into corpus luteum
v7 v7 v7 v7 v7 v7 v7 v7 v7 v7
-
> corpus luteum becomes vascularized and secretes progester
v7 v7 v7 v7 v7 v7 v7
one - v7
> arrests development of other follicles in both ovaries; LH,
v7 v7 v7 v7 v7 v7 v7 v7 v7 v7
