IFMCP CERTIFICATION EXAM
QUESTIONS AND ANSWERS 100% PASS
DIGIN - ANS D-Digestion
I-Intestinal permeability
G-Gastrointestinal Flora
I-Immune regulation
E-Enteric Nervous System
The function of Cortisol - ANS Stimulates the liver to convert AA to glucose to supply energy
to muscles of flight.
-Increase Glucagen in liver
-Mobilize FA in blood
-Increase coagulation
-Suppresses part of immune response
Prolong Cortisol - ANS Stimulates Fat deposits
Increase BP
Increase protein breakdown
Demineralization of bone
Suppression of immune system
1 @COPYRIGHT 2025/2026 ALLRIGHTS RESERVED.
,Memory Loss
Depression
Increase Blood sugar
Sterogenic Pathway - ANS CHL-Pregnelone- 17-OH Pregnenelone- Progesterone- 11 Dexy-
progesterone
Function of DHEA - ANS Precursor for Testosterone and Estrogen
Reverses immuno suppression caused by excessive cortisol levels
Stimulate bone depositions and remodeling
Lower total CHL and LDL level
Increase muscle mass
Involved in conversion of T4-T3
Accelerates recovery from acute stress
Stage one - ANS Arousal
Both Cortisol and DHEA increased but recovered
Asymptomatic
Adaptation Syndrome Stage two - ANS Adaptation
Cortisol chronically elevated but DHEA decreases
sx; stress, anxiety, mood swings, depression, anger
Stage three - ANS Adrenal depletion
Adrenal insufficiency decrease cortisol and DHEA decreased
Depression and Fatigue
2 @COPYRIGHT 2025/2026 ALLRIGHTS RESERVED.
, Adrenal Cortisol dysfunction symptoms - ANS Fatigue
Memory loss of brain fog
Depression
Over-reaction to stress
Any Sx of high adrenaline
Symptoms of too much adrenalin - ANS Weight loss
Anxiety
Palpitation tremors
Cold hand and fee
Systolic HTN
Glucose Fluctuation
Symptoms of low Adrenaline - ANS Orthostatic HTN
Lack of focus
Sleepiness, non restful sleep
Cortisol Steal - ANS Cortisol increase may decrease the manufacture and activity of
progesterone, estrogen, DHEA and Testosterone
Cortisol Steal in Women - ANS Less progestone and initially (estrogen dominance)
Estrogen Dominance
Breast CA
Fibroid
Endometreosis
3 @COPYRIGHT 2025/2026 ALLRIGHTS RESERVED.
QUESTIONS AND ANSWERS 100% PASS
DIGIN - ANS D-Digestion
I-Intestinal permeability
G-Gastrointestinal Flora
I-Immune regulation
E-Enteric Nervous System
The function of Cortisol - ANS Stimulates the liver to convert AA to glucose to supply energy
to muscles of flight.
-Increase Glucagen in liver
-Mobilize FA in blood
-Increase coagulation
-Suppresses part of immune response
Prolong Cortisol - ANS Stimulates Fat deposits
Increase BP
Increase protein breakdown
Demineralization of bone
Suppression of immune system
1 @COPYRIGHT 2025/2026 ALLRIGHTS RESERVED.
,Memory Loss
Depression
Increase Blood sugar
Sterogenic Pathway - ANS CHL-Pregnelone- 17-OH Pregnenelone- Progesterone- 11 Dexy-
progesterone
Function of DHEA - ANS Precursor for Testosterone and Estrogen
Reverses immuno suppression caused by excessive cortisol levels
Stimulate bone depositions and remodeling
Lower total CHL and LDL level
Increase muscle mass
Involved in conversion of T4-T3
Accelerates recovery from acute stress
Stage one - ANS Arousal
Both Cortisol and DHEA increased but recovered
Asymptomatic
Adaptation Syndrome Stage two - ANS Adaptation
Cortisol chronically elevated but DHEA decreases
sx; stress, anxiety, mood swings, depression, anger
Stage three - ANS Adrenal depletion
Adrenal insufficiency decrease cortisol and DHEA decreased
Depression and Fatigue
2 @COPYRIGHT 2025/2026 ALLRIGHTS RESERVED.
, Adrenal Cortisol dysfunction symptoms - ANS Fatigue
Memory loss of brain fog
Depression
Over-reaction to stress
Any Sx of high adrenaline
Symptoms of too much adrenalin - ANS Weight loss
Anxiety
Palpitation tremors
Cold hand and fee
Systolic HTN
Glucose Fluctuation
Symptoms of low Adrenaline - ANS Orthostatic HTN
Lack of focus
Sleepiness, non restful sleep
Cortisol Steal - ANS Cortisol increase may decrease the manufacture and activity of
progesterone, estrogen, DHEA and Testosterone
Cortisol Steal in Women - ANS Less progestone and initially (estrogen dominance)
Estrogen Dominance
Breast CA
Fibroid
Endometreosis
3 @COPYRIGHT 2025/2026 ALLRIGHTS RESERVED.
