GI Disorders – Part 1**
(Complete, organized, no topics missing)
2. Esophagogastroduodenoscopy (EGD)
Purpose
Visualize esophagus, stomach, duodenum.
Nursing Care After EGD
Administer prescribed antacids/meds
Position: reverse Trendelenburg (HOB elevated).
Assess response to treatment.
Watch for complications:
o Significant bleeding
o Aspiration
o Incarceration or strangulation of herniated stomach
o Perforation → signs:
Sudden pain
↓ BP
Rapid pulse
Shock
4. Gastroesophageal Reflux Disease (GERD)
Definition
Backflow of stomach acid into the esophagus.
Risk Factors
Diet (acidic, spicy, fatty foods)
Smoking
Medications
, Alcohol
NG tube
Clinical Manifestations
(Not listed in detail in slides, implied)
Heartburn, regurgitation, chest discomfort.
Labs/Diagnostics
Endoscopy
pH monitoring
Barium swallow
Medications
Antacids
Histamine blockers (H2 blockers)
PPIs
Nursing Teaching
Dietary modifications
Lifestyle changes (weight loss, no smoking)
Proper positioning
Take meds as prescribed
Complications
Precancerous cellular changes
Regeneration of damaged cells incorrectly
5. Hiatal Hernia
Definition
Protrusion of stomach through diaphragm opening.
Types
Sliding Hernia
Most common
Stomach slides in/out of thoracic cavity
Causes reflux
,Rolling (Paraesophageal) Hernia
Stomach rolls up next to esophagus
Does not slide back
Can become blocked
Higher risk complication
Risk Factors
Male sex
Advanced age
Pregnancy
Obesity
Genetics
Straining
Activities increasing abdominal pressure
Assessment
(Not detailed in slides) – Look for symptoms of reflux or obstruction.
Therapeutic Management
(Not detailed) – Typically PPIs, lifestyle changes, surgery if severe.
Postoperative Teaching
Soft diet for 1 week
Anti-reflux medications for life
Walk daily
Clean incisions
Continue lifestyle modifications
6. Gastritis & Peptic Ulcer Disease (PUD)
Gastritis
Inflammation of the gastric mucosa.
Types:
Acute vs Chronic
Erosive vs Non-erosive
, Peptic Ulcer Disease
Break in mucosal lining → acid causes damage.
Types:
Gastric ulcers
Duodenal ulcers
Stress ulcers
Shared Risk Factors
H. pylori (most common)
NSAIDs
Corticosteroids
Stress
Alcohol
Caffeine
Contaminated food/water
Pathophysiology
Gastritis
Damage to protective mucosal barrier
Acid injures small vessels
Leads to edema and inflammation
Ulcers
H. pylori toxins damage mucosa
Acid injures epithelium
Delayed gastric emptying worsens injury
Clinical Manifestations
Gastritis Symptoms
Upper abdominal pain/burning
Indigestion
Nausea, vomiting, hiccups