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NUR 445 EXAM 2 UPDATED ACTUAL Questions and CORRECT Answers

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NUR 445 EXAM 2 UPDATED ACTUAL Questions and CORRECT Answers

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NUR 445 EXAM 2 UPDATED ACTUAL Questions and CORRECT Answers

palpitations, hypotension, diaphoresis, SOB, syncope, light-headedness, dizziness,
s/s of cardiac instability
anxiety

p wave depolarization of the atria

QRS complex ventricular depolarization

T wave ventricular repolarization

age
MI
HTN
HF
cardiomyopathy
infections
cardiac dysrhythmia r/f
DM
sleep apnea
heart surgery
electrolyte disturbances
drug use
digoxin toxicity

measure of time that it takes an electrical impulse to depolarize the atria and travel
PR interval to the ventricles
(0.12-0.20)

measure of time to depolarize the ventricles
QRS interval
(0.6-0.10)

QT interval measure of time that it takes the ventricle to depolarize and then repolarize

<60 bpm
sinus bradycardia hypoxia, hypothermia, sleep, ischemia, electrolyte imbalance, medication toxicity
can be normal in athletes

, sinus brady treatment symptomatic: atropine 1 mg IV push, pacemaker if not effective

>100 bpm
sinus tachycadia
fever, anemia, pain, hypovolemia, PE, MI

sinus tach treatment treat the cause, beta blockers, CCB, vagal maneuvers

atrial dysrhythmias atrial fibriliation, atrial flutter, supraventricular tachycardia, PACs

atrial fibrillation no P waves, narrow + irregular QRS complex

uncontrolled afib rapid rate (>100bpm) - rapid ventricular response

controlled afib regular rate

afib causes age, cardiomyopathy, pericarditis, HTN, valvular disease, obesity, DM, CKD, CAD

afib patho loss of cardiac output (atrial kick) causing the blood to pool in the atria

afib complications syncope, palpitations, SOB. clots

based on new onset or established diagnosis, anticoagulations, rate control in new
afib treatment onset using BB, CCB, digoxin, antiarrhythmic to control rhythm, cardiac ablation,
cardioversion

new onset + symptomatic afib treatment control HR first using CCB, BB, or dig, then initiate anticoagulation therapy

no P waves, F waves present (sawtooth), QRS complex present and regular
atrial flutter
atrial rate can range from 250-350 bpm (RVR) or have a normal HR

atrial flutter causes acute MI, severe mitral valve disease, thyrotoxicosis, COPD

atrial flutter treatment control the rate using BB, CCB, or digoxin, antiarrhythmic, cardioversion

cardioversion for a-flutter warranted if experiencing chest pain, hypotension, SOB

SVT fast HR - unable to determine the rhythm. Narrow QRS complex

ST, AT, a-fib w/ RVR, a-flutter w/ RVR, JT, ischemia, stimulants, infection, fever,
SVT causes
ischemia, hypovolemia, hypoxia, MI, electrolyte imbalances

SVT treatment adenosine, O2, BB, cardioversion

3 or more PVCs in a row, >150 bpm, can be life threatening b/c of reduced cardiac
output, will cause death if untreated
ventricular tachycardia

ALWAYS check if there is a pulse or not

hypovolemia, hypoxia, acidosis, hypo/hyperkalemia, hypoglycemia, hypothermia,
vtach causes
toxins, cardiac tamponade, MI, PE

antiarrhythmic medications (amiodarone), electrolyte replacements (K+ + Mg),
cardioversion
vtach w/ a pulse
cardioversion is typically reserved for patients who are symptomatic such as
hypotension, SOB, or complaining of chest pain

pulseless vtach treatment CPR + defibrilation, epinephrine, + amiodarone

shaky, coarse, fine
ventricular fibrillation
lethal dysrhythmia that requires immediate treatment

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