NGR6605 Exam 1 - Neuro PPT Notes: – Questions &
Complete Verified Answers
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Terms in this set (131)
1. Muscle Contraction or Tension HA
2. Vascular HA (Migraine or Cluster)
Name 3 Types of Primary
3. Mixed (Combo of tension & Vascular)
Headaches:
Source: Neurology 1.pptx
-Traction or Inflammatory
Name type of Secondary
Headache:
Source: Neurology 1.pptx
- CVA
- intracranial infection
- mass
- sudden HTN
- metabolic disorders
Causes for Secondary
- drugs
headaches may include:
- drug withdrawals
- cranial nerve pain
- ear, eye, nose, sinus, teeth, or jaw disorders
Source: Neurology 1.pptx Slide 1 Notes
, - More common in women and whites
- Peak age 30-38 yrs
- Mild to moderate constant pain, bilateral or in a
band-like distribution, usually chronic
- Usually come on gradually and worsen as day
progresses
- NOT accompanied by nausea and vomiting
- NOT made worse by physical activity, light, sounds, or
smells.
Tension Headaches (HA)
Presentation:
Causes:
circuitry of head and neck are hypersensitive to external
stimuli and muscle strain; often related to anxiety and
depression
Source: Neurology 1.pptx
- circuitry of head and neck are hypersensitive to
external stimuli and muscle strain; often related to
anxiety and depression
- sleep disturbances
- stress
Causes and/or triggers for
- TNJ
Tension HA may include:
- neck pain
- and eyestrain
* Avoid triggers- bright lights, head position, stress
Source: Neurology 1.pptx, Slide Notes
, - may last 30 min to several days.
- typically start several hours after waking and
worsen as the day progresses.
- They rarely awaken patients from sleep.
- occur < 15 days/mo.
True about Episodic
- very common
headaches:
- most patients obtain relief with OTC analgesics
and do not seek medical attention.
Source: Neurology 1.pptx/ Slide Notes
- may vary in intensity throughout the day but are almost
always present.
True about Chronic
- occur ≥ 15 days/mo.
headaches -
Source: Neurology 1.pptx/ Slide Notes
- NSAIDS, ASA, Tylenol or Fioricet (Tylenol &
barbiturate);
- some pts respond to Triptans as well- good
product is Treximet (Imitrex & Aleve mix)
- Prevention: Tricyclic antidepressants
- Cool compresses
Treatment for Tension HA's:
- Hydration
- Stress reduction: restorative yoga, deep
breathing, relaxation, sleep
Source: Neurology 1.pptx
True
- If severe headaches are thought to be tension-type
Reconsider the diagnosis of
headaches, the diagnosis should be reconsidered
tension-type headache if
because severe tension-type headaches are rare.
headache is severe, T/F?
Source: Neurology 1.pptx/ Slide Notes
African Americans and True
Hispanics are twice as likely
to have Migraines than Source: Neurology 1.pptx
Caucasians or Asians, T/F?
, True
Migraines are Hereditary,
T/F?
Source: Neurology 1.pptx
________________ is thought to be Migraine
a neurovascular pain
syndrome with altered Source: Neurology 1.pptx/ Migraine Slide Notes
central neuronal
processing (activation of
brain stem nuclei, cortical
hyperexcitability, and
spreading cortical
depression) and
involvement of the
trigeminovascular system
(triggering neuropeptide
release, which causes
painful inflammation in
cranial vessels and the dura
mater).
Complete Verified Answers
Save
Terms in this set (131)
1. Muscle Contraction or Tension HA
2. Vascular HA (Migraine or Cluster)
Name 3 Types of Primary
3. Mixed (Combo of tension & Vascular)
Headaches:
Source: Neurology 1.pptx
-Traction or Inflammatory
Name type of Secondary
Headache:
Source: Neurology 1.pptx
- CVA
- intracranial infection
- mass
- sudden HTN
- metabolic disorders
Causes for Secondary
- drugs
headaches may include:
- drug withdrawals
- cranial nerve pain
- ear, eye, nose, sinus, teeth, or jaw disorders
Source: Neurology 1.pptx Slide 1 Notes
, - More common in women and whites
- Peak age 30-38 yrs
- Mild to moderate constant pain, bilateral or in a
band-like distribution, usually chronic
- Usually come on gradually and worsen as day
progresses
- NOT accompanied by nausea and vomiting
- NOT made worse by physical activity, light, sounds, or
smells.
Tension Headaches (HA)
Presentation:
Causes:
circuitry of head and neck are hypersensitive to external
stimuli and muscle strain; often related to anxiety and
depression
Source: Neurology 1.pptx
- circuitry of head and neck are hypersensitive to
external stimuli and muscle strain; often related to
anxiety and depression
- sleep disturbances
- stress
Causes and/or triggers for
- TNJ
Tension HA may include:
- neck pain
- and eyestrain
* Avoid triggers- bright lights, head position, stress
Source: Neurology 1.pptx, Slide Notes
, - may last 30 min to several days.
- typically start several hours after waking and
worsen as the day progresses.
- They rarely awaken patients from sleep.
- occur < 15 days/mo.
True about Episodic
- very common
headaches:
- most patients obtain relief with OTC analgesics
and do not seek medical attention.
Source: Neurology 1.pptx/ Slide Notes
- may vary in intensity throughout the day but are almost
always present.
True about Chronic
- occur ≥ 15 days/mo.
headaches -
Source: Neurology 1.pptx/ Slide Notes
- NSAIDS, ASA, Tylenol or Fioricet (Tylenol &
barbiturate);
- some pts respond to Triptans as well- good
product is Treximet (Imitrex & Aleve mix)
- Prevention: Tricyclic antidepressants
- Cool compresses
Treatment for Tension HA's:
- Hydration
- Stress reduction: restorative yoga, deep
breathing, relaxation, sleep
Source: Neurology 1.pptx
True
- If severe headaches are thought to be tension-type
Reconsider the diagnosis of
headaches, the diagnosis should be reconsidered
tension-type headache if
because severe tension-type headaches are rare.
headache is severe, T/F?
Source: Neurology 1.pptx/ Slide Notes
African Americans and True
Hispanics are twice as likely
to have Migraines than Source: Neurology 1.pptx
Caucasians or Asians, T/F?
, True
Migraines are Hereditary,
T/F?
Source: Neurology 1.pptx
________________ is thought to be Migraine
a neurovascular pain
syndrome with altered Source: Neurology 1.pptx/ Migraine Slide Notes
central neuronal
processing (activation of
brain stem nuclei, cortical
hyperexcitability, and
spreading cortical
depression) and
involvement of the
trigeminovascular system
(triggering neuropeptide
release, which causes
painful inflammation in
cranial vessels and the dura
mater).
