NURS 5315 Endocrine – Questions With Expert
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Terms in this set (62)
Thyroid hormones T3, T4 and calcitonin
Pancreas hormones Insulin and glucagon
Made up of the cortex which secretes steroids such
Adrenal Glands
as cortisone and aldosterone and the medulla which
hormones
secretes catecholamines such as epi and norepi
result in alterations of thyroid hormone (TH) levels
Primary thyroid disorders with secondary feedback effects on pituitary thyroid
stimulating hormone (TSH)
Subclinical Thyroid Thyroid disease that presents with minimal to no
disease symptoms but with abnormal lab values
Secondary Thyroid conditions that results from the dysfunction of the
disorders pituitary gland TSH production
(hyperthyroidism) a condition that results from any
cause of increased TH levels. Will how low TSH
levels and high T4 level. S&S: increased metabolic
rate, heat intolerance, goiter, menstrual
Thyrotoxicosis
irrregularities, weight loss, diaphoresis, fine tremor,
tachycardia, frequent bowel movements,
restlessness, short attention span, hair loss, anorexia,
exophthalmos, pretibial edema, and heart failure.
, worsening hyperthyroid state triggered by an
igniting even such as infection, trauma,
cardiopulmonary disorder, burns, seizures surgery,
Thyrotoxic Crisis(thyroid
or spontaneously. S&S: extreme restlessness and
storm)
agitation, delirium, seizures, coma, severe
tachycardia, heart failure, hyperthermia, delirium,
volume depletion, NVD and death if not treated.
Most common cause of hyperthyroidism and is an
autoimmune disorder. Antibodies attach to the
thyroid cells and mimic the function of TSH which
results in an increased secretion of T3 and T4 and
overrides the negative feedback mechanisms which
Grave's disease
regulate TSH secretion. The stimulation of the
receptors by the antibodies results in the
development of goiter. May also experience
exophthalmos, periorbital edema, and extraocular
muscle weakness leading to strabismus and diplopia
Follicular hypertrophy of the thyroid cells is
responsible for the formation of the thyroid nodules
which secrete extra hormones. Nodules develop bc
Hyperthyroidism from of normal changes during pregnancy or puberty or
nodular thyroid disease as a result of an autoimmune issue, viral infection or
genetic influence. Symptoms develop slowly and
will not display exophthalmos or pretibial
myxedema
Defect is in the thyroid gland itself which causes
insufficient amounts of thyroid hormone. Causes
include congenital defects, thyroidectomy, thyroid
Primary hypothyroidism
radiation, iodine deficiency, anti-thyroid
medications, or impairment in thyroid hormone
synthesis
Malfunction in the pituitary or hypothalamus glands
Secondary which leads to a lack of TSH. Most common cause is
hypothyroidism pituitary tumors. Other causes include TBI,
subarachnoid hemorrhage, or pituitary infarction
Subclinical mild thyroid failure. defined by elevated TSH level
hypothyroidism with normal TH level.
Guided Solutions
Save
Terms in this set (62)
Thyroid hormones T3, T4 and calcitonin
Pancreas hormones Insulin and glucagon
Made up of the cortex which secretes steroids such
Adrenal Glands
as cortisone and aldosterone and the medulla which
hormones
secretes catecholamines such as epi and norepi
result in alterations of thyroid hormone (TH) levels
Primary thyroid disorders with secondary feedback effects on pituitary thyroid
stimulating hormone (TSH)
Subclinical Thyroid Thyroid disease that presents with minimal to no
disease symptoms but with abnormal lab values
Secondary Thyroid conditions that results from the dysfunction of the
disorders pituitary gland TSH production
(hyperthyroidism) a condition that results from any
cause of increased TH levels. Will how low TSH
levels and high T4 level. S&S: increased metabolic
rate, heat intolerance, goiter, menstrual
Thyrotoxicosis
irrregularities, weight loss, diaphoresis, fine tremor,
tachycardia, frequent bowel movements,
restlessness, short attention span, hair loss, anorexia,
exophthalmos, pretibial edema, and heart failure.
, worsening hyperthyroid state triggered by an
igniting even such as infection, trauma,
cardiopulmonary disorder, burns, seizures surgery,
Thyrotoxic Crisis(thyroid
or spontaneously. S&S: extreme restlessness and
storm)
agitation, delirium, seizures, coma, severe
tachycardia, heart failure, hyperthermia, delirium,
volume depletion, NVD and death if not treated.
Most common cause of hyperthyroidism and is an
autoimmune disorder. Antibodies attach to the
thyroid cells and mimic the function of TSH which
results in an increased secretion of T3 and T4 and
overrides the negative feedback mechanisms which
Grave's disease
regulate TSH secretion. The stimulation of the
receptors by the antibodies results in the
development of goiter. May also experience
exophthalmos, periorbital edema, and extraocular
muscle weakness leading to strabismus and diplopia
Follicular hypertrophy of the thyroid cells is
responsible for the formation of the thyroid nodules
which secrete extra hormones. Nodules develop bc
Hyperthyroidism from of normal changes during pregnancy or puberty or
nodular thyroid disease as a result of an autoimmune issue, viral infection or
genetic influence. Symptoms develop slowly and
will not display exophthalmos or pretibial
myxedema
Defect is in the thyroid gland itself which causes
insufficient amounts of thyroid hormone. Causes
include congenital defects, thyroidectomy, thyroid
Primary hypothyroidism
radiation, iodine deficiency, anti-thyroid
medications, or impairment in thyroid hormone
synthesis
Malfunction in the pituitary or hypothalamus glands
Secondary which leads to a lack of TSH. Most common cause is
hypothyroidism pituitary tumors. Other causes include TBI,
subarachnoid hemorrhage, or pituitary infarction
Subclinical mild thyroid failure. defined by elevated TSH level
hypothyroidism with normal TH level.
