COMAT FBS T4 100% VERIFIED QUESTIONS AND ACCURATE
DETAILED SOLUTIONS
Window period - --Answers----after hbsag disappears but
before Anti-hbs appears
(HBV DNA is the only positive marker)
Hepatitis diagnostic markers:
What is the only positive marker during the window period - --
Answers----HBV DNA
(after hbsag disappears but before Anti-hbs appears)
Hepatitis diagnostic markers:
Hx of past infection - --Answers----positive Anti-hbe
Positive Anti-hbc
(usually) positive Anti-hbs
Hepatitis diagnostic markers:
Carrier state (high infectivity) - --Answers----positive hbsag
Positive hbeag
Positive Anti-hbc
Hepatitis diagnostic markers:
,Carrier state (low infectivity) - --Answers----positive hbsag
Positive Anti-hbe
Positive Anti-hbc
Hepatitis types that are transmitted via fecal-oral / contaminated
food/water - --Answers----Hep A
Hep E
("vowels for your bowels")
Which type(s) of hepatitis CANNOT cause chronic infection - --
Answers----Hep A
(but only w/ genotype 3 for Hep E)
Which type(s) of hepatitis do NOT have a pre-exposure vaccine -
--Answers----Hep C
Hep E
E6 protein - --Answers----(HPV)
Inactivates tumor suppression gene p53
E7 protein - --Answers----(HPV)
Inactivates tumor suppression gene RB
HPV proteins - --Answers----E6 (p53)
,E7 (RB)
HPV types and associated Sx - --Answers----1-4, 7, 10:
cutaneous warts
6, 11: genital warts
16, 18: anogenital cancers (cervical, uterine, penis, anus)
Why will anogenital warts (HPV 6, 11) NOT progress to anogenital
cancer - --Answers----anogenital cancer is caused by different
serotypes (16, 18)
Hyper igm syndrome - --Answers-----Most commonly due to
defective CD40L on Th cells class switching defect; X-linked
recessive.
-Severe pyogenic infections early in life; opportunistic infection
with Pneumocystis, Cryptosporidium, CMV.
-Increased igm, double decreased igg, iga, ige
Hyper igm syndrome - --Answers----Th CD40L mutation =
inability of B-lymphos to DIFFERENTIATE from igm to other
isotypes such as igd, igg, etc.
= high igm, low everything else
Hyperkalemia causes - --Answers----"MACHHINE"
M - Medications - ACE inhibitors, NSAIDS
A - Acidosis - Metabolic and respiratory
C - Cellular destruction - Burns, traumatic injury
, H - Hypoaldosteronism / Hemolysis
I - Intake - Excessive
N - Nephrons, renal failure
E - Excretion - Impaired
Hyperkalemia cardiac symptoms - --Answers----peaked T
waves (compare to broad U waves in hypokalemia)
(also wide QRS)
Hyperlipidemia drugs - --Answers----1. 3-Hydroxy-3-
Methylglutaryl Coenzyme A Reductase Inhibitors (-statins)
atorvastatin (Lipitor), simvastatin (Zocor)
2. Niacin *preferred
3. Vitamin E
4. Ezetimibe (Zetia)
5. Cholestyramine
6. Fibric Acid Drug -gemfibrozil (Lopid)
Lyme disease mechanism - --Answers----organism uses
surface proteins to localize the skin, nerve, or joint tissue, and
heart. Neutralizes complement, enzyme is able to break down
components of connective tissue
DETAILED SOLUTIONS
Window period - --Answers----after hbsag disappears but
before Anti-hbs appears
(HBV DNA is the only positive marker)
Hepatitis diagnostic markers:
What is the only positive marker during the window period - --
Answers----HBV DNA
(after hbsag disappears but before Anti-hbs appears)
Hepatitis diagnostic markers:
Hx of past infection - --Answers----positive Anti-hbe
Positive Anti-hbc
(usually) positive Anti-hbs
Hepatitis diagnostic markers:
Carrier state (high infectivity) - --Answers----positive hbsag
Positive hbeag
Positive Anti-hbc
Hepatitis diagnostic markers:
,Carrier state (low infectivity) - --Answers----positive hbsag
Positive Anti-hbe
Positive Anti-hbc
Hepatitis types that are transmitted via fecal-oral / contaminated
food/water - --Answers----Hep A
Hep E
("vowels for your bowels")
Which type(s) of hepatitis CANNOT cause chronic infection - --
Answers----Hep A
(but only w/ genotype 3 for Hep E)
Which type(s) of hepatitis do NOT have a pre-exposure vaccine -
--Answers----Hep C
Hep E
E6 protein - --Answers----(HPV)
Inactivates tumor suppression gene p53
E7 protein - --Answers----(HPV)
Inactivates tumor suppression gene RB
HPV proteins - --Answers----E6 (p53)
,E7 (RB)
HPV types and associated Sx - --Answers----1-4, 7, 10:
cutaneous warts
6, 11: genital warts
16, 18: anogenital cancers (cervical, uterine, penis, anus)
Why will anogenital warts (HPV 6, 11) NOT progress to anogenital
cancer - --Answers----anogenital cancer is caused by different
serotypes (16, 18)
Hyper igm syndrome - --Answers-----Most commonly due to
defective CD40L on Th cells class switching defect; X-linked
recessive.
-Severe pyogenic infections early in life; opportunistic infection
with Pneumocystis, Cryptosporidium, CMV.
-Increased igm, double decreased igg, iga, ige
Hyper igm syndrome - --Answers----Th CD40L mutation =
inability of B-lymphos to DIFFERENTIATE from igm to other
isotypes such as igd, igg, etc.
= high igm, low everything else
Hyperkalemia causes - --Answers----"MACHHINE"
M - Medications - ACE inhibitors, NSAIDS
A - Acidosis - Metabolic and respiratory
C - Cellular destruction - Burns, traumatic injury
, H - Hypoaldosteronism / Hemolysis
I - Intake - Excessive
N - Nephrons, renal failure
E - Excretion - Impaired
Hyperkalemia cardiac symptoms - --Answers----peaked T
waves (compare to broad U waves in hypokalemia)
(also wide QRS)
Hyperlipidemia drugs - --Answers----1. 3-Hydroxy-3-
Methylglutaryl Coenzyme A Reductase Inhibitors (-statins)
atorvastatin (Lipitor), simvastatin (Zocor)
2. Niacin *preferred
3. Vitamin E
4. Ezetimibe (Zetia)
5. Cholestyramine
6. Fibric Acid Drug -gemfibrozil (Lopid)
Lyme disease mechanism - --Answers----organism uses
surface proteins to localize the skin, nerve, or joint tissue, and
heart. Neutralizes complement, enzyme is able to break down
components of connective tissue
