CLT-LANA
Study online at https://quizlet.com/_hsd8nr
1. Stage 0 CVI: No symptoms, norm protein
2. Stage I cvi: Mild edema, normal protein, norm lymph vessels, phlebolymphodynamic insufficiency
3. Stage 2 cvi: Mod symptoms ( pigmentation, viscosity, pain), high protein, vessels morphologically changed, TC
reduced, phlebolymphostatic insufficiency
4. Stage 3 cvi: Severe symptoms( hypoxia, necrosis, pain), very high protein load, lymph vessels morphologically
change, TC reduced, severe phlebolymphostatic insufficiency
5. Treatment stage 0 cvi: Compression therapy, elevation, exercise
6. Treatment stage 1 cvi: Compression therapy, elevation, exercise
7. Treatment stage 2 cvi: CDT
8. Treatment stage 3 cvi: CDT and wound care
9. Ambulatory venous hypertension: Failure of venous valves such that blood flow bidirectional
resulting in increased pressure to the capillaries
10. What happens with low flow states in blood capillaries to leukocytes: They be-
come trapped and release proteolytic enzymes and oxygen free radicals which damage capillary basement membranes
11. What happens to plasma proteins with low flow states in blood capillaries: -
Plasma proteins leak into leak into the surrounding tissue creating a fibrin cuff, edema, and hypoxia
12. What causes reddish brown skin associated with edema/ lymphedema: High
intracApillary pressure stretches endothelial cells away from each other and eurythrocytes leave the blood capillaries
staining skin with hemosiderin deposits
13. Does ambulatory venous hypertension cause an increase or decrease in net
filtration: Increase
14. What causes mechanical insufficiency in cvi: Fibrin leakage into the tissue as well as an
inflammatory reaction overall increase in protein and water load
15. What is not considered with staging lymphedema: Limb volume
16. Mild lymphedema severity volume increase: <20%
17. Moderate lymphedema severity volume increase: 20-40%
18. Severe lymphedema severity increase: >40%
19. Where do DVT form: Sub fascial veins
20. What is the most common ulceration and where do they form: Venous stasis and
they form in the distal 1/3 of the leg usually around the malleoli
21. vascular venous wounds: Shallow, irregular, on anterior/ medial leg with peri wound with hemosiderin
staining,scaly, weepy, warm skin
1/3
Study online at https://quizlet.com/_hsd8nr
1. Stage 0 CVI: No symptoms, norm protein
2. Stage I cvi: Mild edema, normal protein, norm lymph vessels, phlebolymphodynamic insufficiency
3. Stage 2 cvi: Mod symptoms ( pigmentation, viscosity, pain), high protein, vessels morphologically changed, TC
reduced, phlebolymphostatic insufficiency
4. Stage 3 cvi: Severe symptoms( hypoxia, necrosis, pain), very high protein load, lymph vessels morphologically
change, TC reduced, severe phlebolymphostatic insufficiency
5. Treatment stage 0 cvi: Compression therapy, elevation, exercise
6. Treatment stage 1 cvi: Compression therapy, elevation, exercise
7. Treatment stage 2 cvi: CDT
8. Treatment stage 3 cvi: CDT and wound care
9. Ambulatory venous hypertension: Failure of venous valves such that blood flow bidirectional
resulting in increased pressure to the capillaries
10. What happens with low flow states in blood capillaries to leukocytes: They be-
come trapped and release proteolytic enzymes and oxygen free radicals which damage capillary basement membranes
11. What happens to plasma proteins with low flow states in blood capillaries: -
Plasma proteins leak into leak into the surrounding tissue creating a fibrin cuff, edema, and hypoxia
12. What causes reddish brown skin associated with edema/ lymphedema: High
intracApillary pressure stretches endothelial cells away from each other and eurythrocytes leave the blood capillaries
staining skin with hemosiderin deposits
13. Does ambulatory venous hypertension cause an increase or decrease in net
filtration: Increase
14. What causes mechanical insufficiency in cvi: Fibrin leakage into the tissue as well as an
inflammatory reaction overall increase in protein and water load
15. What is not considered with staging lymphedema: Limb volume
16. Mild lymphedema severity volume increase: <20%
17. Moderate lymphedema severity volume increase: 20-40%
18. Severe lymphedema severity increase: >40%
19. Where do DVT form: Sub fascial veins
20. What is the most common ulceration and where do they form: Venous stasis and
they form in the distal 1/3 of the leg usually around the malleoli
21. vascular venous wounds: Shallow, irregular, on anterior/ medial leg with peri wound with hemosiderin
staining,scaly, weepy, warm skin
1/3
