NUR2392 Multidimensional Care II Exam 2 –
Questions With Verified Solutions
Save
Practice questions for this set
Learn 1 /7 Study with Learn
inflammation of the gastric mucosa lining.
erosive or non-erosive.
Most common stomach disorder.
Categorized as Type A as a result of an autoimmune condition
Type B which is caused by the bacteria Helicobacter Pylori.
Caused by bacterial infections, overuse of NSAIDs, use of alcohol, stress,
andage.
Choose an answer
1 Uncompensated 2 Metabolic Alkalosis s/s
3 gastritis 4 Alkalosis
Don't know?
Terms in this set (76)
, Uncompensated pH and one other value is abnormal
Partial compensation pH, CO2, and HCO3 are all off
Full compensation pH is normal
reduces the excitability of cardiovascular muscle,
Acidosis
neurons, skeletal muscle, and smooth muscle.
increases the sensitivity of excitable tissues allowing
Alkalosis
them to OVERRESPOND without stimulation
- Assess cardiovascular w/ acidosis (cardiac arrest
from hyperkalemia)
ABG considerations
- Assess neuro status
- Fall precautions
Bicarb HC03 kidney compensation (slow and powerful)
Respiratory PaC02 Respiratory compensation (fast but limited)
DKA, Starvation, diarrhea, kidney failure,
Metabolic acidosis dehydration, liver failure, pancreatitis, heavy
causes exercise, seizure activity, fever, hypoxia, ischemia,
ethanol/methanol intoxication
bradycardia, hypotension, thready pulse, CNS
Metabolic acidosis signs
depression, hyporeflexia, kussmal resp (with resp
and symptoms
compensation), warm, flushed, dry skin.
Metabolic acidosis hydration and medication to treat underlying
treatment problems (DKA - give insulin).
antacids, blood transfusion, sodium bicarbonate,
Metabolic alkalosis total parenteral nutrition (TPN), prolonged vomiting,
causes nasogastric suctioning, hypercortisolism,
hyperaldosteronism, Loop/Thiazide diuretics.
anxiety, irritability, tetany, seizures, POSITIVE
CHVOSTEK, POSITIVE TROUSSEAU, parathesis,
hyperreflexia, muscle cramping/twitching, skeletal
Metabolic Alkalosis s/s
muscle weakness, Tachycardia, norm/low BP,
increased Digoxin toxicity, decreased respiratory
effort (muscle weakness).
Questions With Verified Solutions
Save
Practice questions for this set
Learn 1 /7 Study with Learn
inflammation of the gastric mucosa lining.
erosive or non-erosive.
Most common stomach disorder.
Categorized as Type A as a result of an autoimmune condition
Type B which is caused by the bacteria Helicobacter Pylori.
Caused by bacterial infections, overuse of NSAIDs, use of alcohol, stress,
andage.
Choose an answer
1 Uncompensated 2 Metabolic Alkalosis s/s
3 gastritis 4 Alkalosis
Don't know?
Terms in this set (76)
, Uncompensated pH and one other value is abnormal
Partial compensation pH, CO2, and HCO3 are all off
Full compensation pH is normal
reduces the excitability of cardiovascular muscle,
Acidosis
neurons, skeletal muscle, and smooth muscle.
increases the sensitivity of excitable tissues allowing
Alkalosis
them to OVERRESPOND without stimulation
- Assess cardiovascular w/ acidosis (cardiac arrest
from hyperkalemia)
ABG considerations
- Assess neuro status
- Fall precautions
Bicarb HC03 kidney compensation (slow and powerful)
Respiratory PaC02 Respiratory compensation (fast but limited)
DKA, Starvation, diarrhea, kidney failure,
Metabolic acidosis dehydration, liver failure, pancreatitis, heavy
causes exercise, seizure activity, fever, hypoxia, ischemia,
ethanol/methanol intoxication
bradycardia, hypotension, thready pulse, CNS
Metabolic acidosis signs
depression, hyporeflexia, kussmal resp (with resp
and symptoms
compensation), warm, flushed, dry skin.
Metabolic acidosis hydration and medication to treat underlying
treatment problems (DKA - give insulin).
antacids, blood transfusion, sodium bicarbonate,
Metabolic alkalosis total parenteral nutrition (TPN), prolonged vomiting,
causes nasogastric suctioning, hypercortisolism,
hyperaldosteronism, Loop/Thiazide diuretics.
anxiety, irritability, tetany, seizures, POSITIVE
CHVOSTEK, POSITIVE TROUSSEAU, parathesis,
hyperreflexia, muscle cramping/twitching, skeletal
Metabolic Alkalosis s/s
muscle weakness, Tachycardia, norm/low BP,
increased Digoxin toxicity, decreased respiratory
effort (muscle weakness).
