NSG 555 Autoimmune Diseases + Leukemia And
Lymphoma: Questions & A+ Solutions
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inflammation of the fingers and/or toes
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1 Polymyalgia Rheumatica 2 Giant cell arteritis and PMR
3 Genetic tendency for PMR 4 dactylitis psoriatic arthritis
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Terms in this set (85)
Rare under 50, common 70-80. Aching, stiffness in
Polymyalgia Rheumatica
shoulder, neck, pelvic girdle.
, Erythrocyte Sedimentation Rate. Associated with
ESR
polymyalgia rheumatica.
C-Reactive Protein. Associated with polymyalgia
CRP
rheumatica.
Based on clinical grounds, excluding other
Diagnosis of PMR
inflammatory conditions.
Genetic tendency for Caucasian, HLA-DRB1 genotype, increased TNF A
PMR and Il-1 susceptibility.
PMR: Asymmetric, corticosteroid-responsive, non-
RA vs PMR
erosive, self-limiting.
Edema in dorsum of hands, wrists, and feet, seen in
Pitting Edema
PMR.
High ESR, CRP, leukocytes, platelets, normochromic
Lab Findings in PMR
anemia, low albumin.
Imaging technique to show joint involvement in
Musculoskeletal US
PMR.
what disease may occure giant cell arteritis.
with PMR
refer to rheumatology for definitive dx and rX.
follow up can be by PCP
systemic corticosteroids are best--12.5-25 mg
management of PMR prednisone daily. Taper doses by 5 mg/week after
4-8 weeks of therapy and continue the lowest
effective doseage. NSAIDS DO NOT HELP. labs will
improve within 4-8 weeks and so should symptoms.
steroids often continued for 1-2 years
bone protective if on corticosteroids need Ca, D and
measures for PMR biphosphonates
Giant cell arteritis and PMR patients need to report immediately if new ha,
PMR change in vision, scalp pain, pain with chewing.
Lymphoma: Questions & A+ Solutions
Save
Practice questions for this set
Learn 1 /7 Study with Learn
inflammation of the fingers and/or toes
Choose an answer
1 Polymyalgia Rheumatica 2 Giant cell arteritis and PMR
3 Genetic tendency for PMR 4 dactylitis psoriatic arthritis
Don't know?
Terms in this set (85)
Rare under 50, common 70-80. Aching, stiffness in
Polymyalgia Rheumatica
shoulder, neck, pelvic girdle.
, Erythrocyte Sedimentation Rate. Associated with
ESR
polymyalgia rheumatica.
C-Reactive Protein. Associated with polymyalgia
CRP
rheumatica.
Based on clinical grounds, excluding other
Diagnosis of PMR
inflammatory conditions.
Genetic tendency for Caucasian, HLA-DRB1 genotype, increased TNF A
PMR and Il-1 susceptibility.
PMR: Asymmetric, corticosteroid-responsive, non-
RA vs PMR
erosive, self-limiting.
Edema in dorsum of hands, wrists, and feet, seen in
Pitting Edema
PMR.
High ESR, CRP, leukocytes, platelets, normochromic
Lab Findings in PMR
anemia, low albumin.
Imaging technique to show joint involvement in
Musculoskeletal US
PMR.
what disease may occure giant cell arteritis.
with PMR
refer to rheumatology for definitive dx and rX.
follow up can be by PCP
systemic corticosteroids are best--12.5-25 mg
management of PMR prednisone daily. Taper doses by 5 mg/week after
4-8 weeks of therapy and continue the lowest
effective doseage. NSAIDS DO NOT HELP. labs will
improve within 4-8 weeks and so should symptoms.
steroids often continued for 1-2 years
bone protective if on corticosteroids need Ca, D and
measures for PMR biphosphonates
Giant cell arteritis and PMR patients need to report immediately if new ha,
PMR change in vision, scalp pain, pain with chewing.