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NURS 3550 - WEEK 6 EXAM QUESTIONS WITH 100% CORRECT ANSWERS | LATEST VERSION 2025/2026.

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Hemostasis - ANS The process that prevents and stops bleeding, involving the formation of a platelet plug. Platelet Plug Formation - ANS Occurs when blood vessel damage exposes collagen, activating platelets to change shape, release ADP and TXA2, and aggregate to stop bleeding. Coagulation: Extrinsic Pathway - ANS Activated by tissue factor outside the blood, leading to the activation of factor VII, which activates factor X and catalyzes prothrombin into thrombin. Thrombin - ANS An enzyme that catalyzes fibrinogen into fibrin, activates factor V into Va, and converts factor VIII into its active form VIIIa. Coagulation: Intrinsic Pathway - ANS Activated by factors within the blood, starting with factor XII exposed to collagen, leading to the activation of factor X. Thrombosis - ANS The formation of a blood clot within a blood vessel. Anticoagulants - ANS Drugs that disrupt the coagulation cascade and suppress fibrin production, primarily used for v

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NURS 3550 - WEEK 6 EXAM QUESTIONS
WITH 100% CORRECT ANSWERS |
LATEST VERSION 2025/2026.




Hemostasis - ANS The process that prevents and stops bleeding, involving the formation of a
platelet plug.


Platelet Plug Formation - ANS Occurs when blood vessel damage exposes collagen, activating
platelets to change shape, release ADP and TXA2, and aggregate to stop bleeding.


Coagulation: Extrinsic Pathway - ANS Activated by tissue factor outside the blood, leading to
the activation of factor VII, which activates factor X and catalyzes prothrombin into thrombin.


Thrombin - ANS An enzyme that catalyzes fibrinogen into fibrin, activates factor V into Va,
and converts factor VIII into its active form VIIIa.


Coagulation: Intrinsic Pathway - ANS Activated by factors within the blood, starting with
factor XII exposed to collagen, leading to the activation of factor X.


Thrombosis - ANS The formation of a blood clot within a blood vessel.


Anticoagulants - ANS Drugs that disrupt the coagulation cascade and suppress fibrin
production, primarily used for venous thrombosis.


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