Examl 2:l NRl 324/l NR324l (Latestl 2026/l
2027l Update)l Adultl Healthl Il Guide|l Q/Al
|l Gradel A|l 100%l Correctl (Verifiedl
Answers)l -Chamberlain
Q:l Hemophilia
Answer:
anl X-linkedl recessivel geneticl disorderl causedl byl al defectivel orl deficientl coagulationl
factor
-Hemophilial Al (mostl common)l andl hemophilial B
Q:l Clinicall manifestationsl ofl hemophilial andl complications
Answer:
slow,l persistent,l prolongedl bleedingl froml minorl traumal orl cuts,l delayedl bleedingl afterl
minorl injuriesl (hrsl orl days),l epistaxis,l GIl bleeding,l hematurial andl potentiall renall
failure,l echymosesl andl subcutaneousl hematomas,l bleedingl intol thel jointsl (hemarthrosis)
Q:l Diagnosticl studiesl forl hemophilial include
Answer:
Laboratoryl studiesl
-Prothrombinl time
-Thrombinl time
-Plateletl count
-Partiall thromboplastinl time
-Bleedingl time
-Factorsl assays
Q:l Managementl forl hemophilia
Answer:
,-preventivel care
-stopl topicall bleedingl ASAPl w/l directl pressurel orl icel
-administerl specificl coagulationl factorl tol raisel ptsl level
-Ifl jointl isl involvedl totallyl restl involvedl joint
-replacementl therapyl duringl acutel episodesl asl prophylaxisl
-treatmentl ofl complicationsl ofl diseasel orl therapy
Q:l Hemochromatosis
Answer:
Ironl overloadl disorder
-causedl primarilyl byl al geneticl defect
-mayl alsol occurl secondaryl tol diseasesl suchl asl sideroblasticl anemia
-mayl alsol bel causedl byl liverl diseasel andl chronicl bloodl transfusionsl usedl tol treatl
thalassemial andl SCD
Q:l Normall rangel forl totall bodyl iron
Answer:
2l tol 6g
Q:l Someonel withl hemochromatosisl mayl accumulatel ironl atl al ratel of..
Answer:
0.5l tol 1.0l al yearl andl mayl exceedl totall ironl concentrationsl ofl 50g
Q:l Physicall examinationl ofl al patientl withl hemochromatosisl willl reveal
Answer:
anl enlargedl liverl andl spleenl withl pigmentationl changesl inl thel skin
Q:l S/Sl ofl hemochromatosis
Answer:
Normallyl developl betweenl agesl 40-60
Earlyl symptoms:l fatigue,l arthralgia,l impotence,l abdominall pain,l andl weightl loss
Later:l liverl enlargementl andl eventuallyl cirrhosis
,-diabetesl mellitus,l skinl pigmentl changesl (bronzing),l cardiacl changesl (e.g.,l
cardiomyopathy),l arthritis,l andl testicularl atrophy
Q:l Disseminatedl Intravascularl Coagulationl (DIC)
Answer:
al seriousl bleedingl andl thromboticl disorderl thatl resultsl froml abnormallyl initiatedl andl
acceleratedl clotting
-Subsequentl decreasesl inl clottingl factorsl andl plateletsl ensue,l whichl mayl leadl tol
uncontrollablel hemorrhage
Q:l Truel orl false:l DICl canl justl occurl withl aging
Answer:
FALSE:l DICl isl alwaysl causedl byl anl underlyingl diseasel orl condition
-underlyingl conditionl mustl bel treatedl forl DICl tol resolve
Q:l Riskl factorsl forl DIC
Answer:
Shock
Septicemia
Hemolyticl processes
Obstetricl conditions
Malignancies
Tissuel damage
Q:l S/Sl ofl DIC
Answer:
Delirium,l coma,l ischemia
Gangrene,l pulmonaryl ARDS
oliguria,l azotemia
paralyticl ileusl
intracerebrall bleeding
petechiae
hematuria
dyspnea
, hemoptysis
massivel bleeding
epistaxis
gingivall bleeding
Q:l Diagnosticl studiesl forl DIC
Answer:
-Bloodl testl -l PT,l PTT,l aPTT,l thrombinl time,l fibrinogen,l platelets
-fibrinl splitl products
-Factorl assays
-Antithrombinl 3
-Proteinsl Cl andl S
-Plasminogen,l tissuel plasminogenl activator
-Peripherall bloodl smear
Q:l Managementl ofl DIC
Answer:
Diagnosel itl quickly
-stabilizel patient
-treatl underlyingl causativel disease
-controll ongoingl thrombosisl andl bleeding
Q:l Pacemakerl indications
Answer:
-usedl tol pacel thel heartl whenl thel normall conductionl pathwayl isl damaged
-treatl chronicl heartl problemsl (AVl heartl blocks,l sickl sinusl syndrome,l atriall fibrillationl
withl slowl ventricularl response,l severel heartl failure,l cardiomyopathy,l bundlel branchl
block).
Q:l Examplel ofl al pacemakerl spike
Answer:
Q:l Nursingl Managementl forl al patientl withl al pacel maker
2027l Update)l Adultl Healthl Il Guide|l Q/Al
|l Gradel A|l 100%l Correctl (Verifiedl
Answers)l -Chamberlain
Q:l Hemophilia
Answer:
anl X-linkedl recessivel geneticl disorderl causedl byl al defectivel orl deficientl coagulationl
factor
-Hemophilial Al (mostl common)l andl hemophilial B
Q:l Clinicall manifestationsl ofl hemophilial andl complications
Answer:
slow,l persistent,l prolongedl bleedingl froml minorl traumal orl cuts,l delayedl bleedingl afterl
minorl injuriesl (hrsl orl days),l epistaxis,l GIl bleeding,l hematurial andl potentiall renall
failure,l echymosesl andl subcutaneousl hematomas,l bleedingl intol thel jointsl (hemarthrosis)
Q:l Diagnosticl studiesl forl hemophilial include
Answer:
Laboratoryl studiesl
-Prothrombinl time
-Thrombinl time
-Plateletl count
-Partiall thromboplastinl time
-Bleedingl time
-Factorsl assays
Q:l Managementl forl hemophilia
Answer:
,-preventivel care
-stopl topicall bleedingl ASAPl w/l directl pressurel orl icel
-administerl specificl coagulationl factorl tol raisel ptsl level
-Ifl jointl isl involvedl totallyl restl involvedl joint
-replacementl therapyl duringl acutel episodesl asl prophylaxisl
-treatmentl ofl complicationsl ofl diseasel orl therapy
Q:l Hemochromatosis
Answer:
Ironl overloadl disorder
-causedl primarilyl byl al geneticl defect
-mayl alsol occurl secondaryl tol diseasesl suchl asl sideroblasticl anemia
-mayl alsol bel causedl byl liverl diseasel andl chronicl bloodl transfusionsl usedl tol treatl
thalassemial andl SCD
Q:l Normall rangel forl totall bodyl iron
Answer:
2l tol 6g
Q:l Someonel withl hemochromatosisl mayl accumulatel ironl atl al ratel of..
Answer:
0.5l tol 1.0l al yearl andl mayl exceedl totall ironl concentrationsl ofl 50g
Q:l Physicall examinationl ofl al patientl withl hemochromatosisl willl reveal
Answer:
anl enlargedl liverl andl spleenl withl pigmentationl changesl inl thel skin
Q:l S/Sl ofl hemochromatosis
Answer:
Normallyl developl betweenl agesl 40-60
Earlyl symptoms:l fatigue,l arthralgia,l impotence,l abdominall pain,l andl weightl loss
Later:l liverl enlargementl andl eventuallyl cirrhosis
,-diabetesl mellitus,l skinl pigmentl changesl (bronzing),l cardiacl changesl (e.g.,l
cardiomyopathy),l arthritis,l andl testicularl atrophy
Q:l Disseminatedl Intravascularl Coagulationl (DIC)
Answer:
al seriousl bleedingl andl thromboticl disorderl thatl resultsl froml abnormallyl initiatedl andl
acceleratedl clotting
-Subsequentl decreasesl inl clottingl factorsl andl plateletsl ensue,l whichl mayl leadl tol
uncontrollablel hemorrhage
Q:l Truel orl false:l DICl canl justl occurl withl aging
Answer:
FALSE:l DICl isl alwaysl causedl byl anl underlyingl diseasel orl condition
-underlyingl conditionl mustl bel treatedl forl DICl tol resolve
Q:l Riskl factorsl forl DIC
Answer:
Shock
Septicemia
Hemolyticl processes
Obstetricl conditions
Malignancies
Tissuel damage
Q:l S/Sl ofl DIC
Answer:
Delirium,l coma,l ischemia
Gangrene,l pulmonaryl ARDS
oliguria,l azotemia
paralyticl ileusl
intracerebrall bleeding
petechiae
hematuria
dyspnea
, hemoptysis
massivel bleeding
epistaxis
gingivall bleeding
Q:l Diagnosticl studiesl forl DIC
Answer:
-Bloodl testl -l PT,l PTT,l aPTT,l thrombinl time,l fibrinogen,l platelets
-fibrinl splitl products
-Factorl assays
-Antithrombinl 3
-Proteinsl Cl andl S
-Plasminogen,l tissuel plasminogenl activator
-Peripherall bloodl smear
Q:l Managementl ofl DIC
Answer:
Diagnosel itl quickly
-stabilizel patient
-treatl underlyingl causativel disease
-controll ongoingl thrombosisl andl bleeding
Q:l Pacemakerl indications
Answer:
-usedl tol pacel thel heartl whenl thel normall conductionl pathwayl isl damaged
-treatl chronicl heartl problemsl (AVl heartl blocks,l sickl sinusl syndrome,l atriall fibrillationl
withl slowl ventricularl response,l severel heartl failure,l cardiomyopathy,l bundlel branchl
block).
Q:l Examplel ofl al pacemakerl spike
Answer:
Q:l Nursingl Managementl forl al patientl withl al pacel maker
