Nutrition And Metabolism Exam 3 Study Guide (APHY 102) Verified Questions And Answers

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Nutrition And Metabolism Exam 3 Study Guide (APHY 102) Verified Questions And Answers

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Nutrition And Metabolism Exam 3 Study Guide (APHY 102) Verified Questions And Answers Nutrition And Metabolism Exam 3 Study Guide (APHY 102) Verified Questions And Answers Nutrition And Metabolism Exam 3 Study Guide (APHY 102) Verified Questions And Answers Nutrition And Metabolism Exam 3 Study Guide (APHY 102) Verified Questions And Answers Nutrition And Metabolism Exam 3 Study Guide (APHY 102) Verified Questions And Answers

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Nutrition And Metabolism Exam 3 Study
ll ll ll ll ll ll

Guide (APHY 102) Verified Questions And
ll ll ll ll ll ll

ll
Answers ll

ll

Where llis llSGLT1 llpredominantly ll expressed? -
ll ll CORRECT ll ANSWER-Sodium
lldependent ll
ll
glucose lltransporter: llbrain, heart,
ll

llenterocytes
ll

What llare llthe llpathophysiological llconditions llthat llaffect llfructose lluptake llrate llin llthe
llbody? ll
ll
- llCORRECT llANSWER-Fructosuria, llhereditary llfructose llintolerance, lland ll1-
6
ll
biphosphatase
lldeficiency
ll

How llare llnutrient llmolecules lltaken llin llenterocytes? ll- llCORRECT llANSWER-
Sugars: ll
ll ll
Transporters
ll
Protein: llBroken lldown llinto llamino llacids lland llabsorbed llwith llsodium
llcotransport
ll
Lipids: llBroken lldown llinto llfatty llacids lland
llabsorbed
ll

4) Which llglucose lltransport llsystem llis llused llfor llfructose, lland llwhere llis llit
llexpressed llin llthe llbody? ll- llCORRECT llANSWER-GLUT5, llprimarily llexpressed llon llthe

llapical llsurface llof llthe llsmall llintestine llbrush llborder llcells, llbut llalso llat lllower lllevels llin

lltestis, llkidney, llbrain, llskeletal llmuscle lland llwhite lladipose lltissue ll
ll

5) What llis llprimary lllactase lldeficiency? ll- llCORRECT llANSWER-Inherited
lldeficiency llin lllactase llenzyme, lllactase lldecreases llas llyour lldiet llbecomes llless llreliant

llon llmilk lland lldairy llproducts. ll
ll

6) What llis llthe llfate llof lldietary lllipids? llHow llare llthey lldigested, llabsorbed, lland
lltransferred llin llthe llbody? ll- llCORRECT llANSWER-Digested ll

Emulsified llby llbile llsalts ll
Degraded llinto lltriglycerols llby llintestinal lllipases.
llAbsorbed ll

Resulting llfatty llacids llare lltaken llup llby llintestinal llmucosa lland llturned llinto lltriglycerols.
llTriglycerols llare llcombined llwith llcholesterol lland llapoproteins llto llform llchylomicrons ll

Transferred ll
Chylomycrons llmove llthrough lllacteal llsystem llto llorgans lland lltissues. ll

, Lipoprotein lllipases llactivated llby llapoC llII llin llthe llcapillaries llrelease llfatty llacids lland
llglycerol llFatty llacids llenter llcells ll(in llimage llmyocytes llor lladipocytes. llFatty llacids llare

lloxidized llas llfuel llor llreestrified llfor llstorage. ll
ll

How llare lllipids lltaken llinto lladipocytes? ll- llCORRECT llANSWER-They llare llbroken lldown
llby ll
ll
apoC llII llinto llfatty llacids llbefore llentering llvia llfacilitated
lltransport.
ll

What llis llCori llcycle? ll- llCORRECT llANSWER-A lllittle llgame llthe llmuscle lland llliver llplay,
llmuscle llis llworking llso llits llusing llglucose lland llmaking lllactase llas lla llbyproduct, lltoo

llmuch lllactate llis lla llbad llthing llso llthis lllactate llis llshuttled lloff llto llthe llliver ll(mom!). llThe

llliver llconverts lllactate llback llinto llpyruvate lland llturns llpyruvate llback llinto llglucose llvia

llgluconeogenesis, llit llthen llsends llthis llglucose llback llto llthe llmuscles llso llthey llcan

llcontinue llworking. ll
ll

9) llWhat llis llglucose-alanine llcycle? ll- llCORRECT llANSWER-Basically lllike llCori llcycle,
llbut ll
ll
alanine llinstead llof lllactate. llStill lla llgame llbetween llmuscles lland
llliver.
ll
Pyruvate ll+ llNH2 ll= llAlanine

ll(Transamination)
ll

0) llHow lldoes llexercise llintensity llaffect llcarbohydrate lland llfat llmetabolism? ll(Which
llone ll
ll
increases llwith llexercise llintensity) ll- llCORRECT llANSWER-When llexercise
llintensity
ll
increases, llcarbohydrate llmetabolism llincreases lland llfat llmetabolism
lldecreases.
ll

11) llHow llis llmeal llorder ll(carbohydrate llfirst llor llprotein llfirst) llaffects llpost llprandial
llglucose ll
ll
and llpost llprandial llinsulin lllevels? ll- llCORRECT llANSWER-Eating llprotein
llthen
ll
carbohydrate llwill llyield lllower llpost llprandial llinsulin lllevels llthen llvise
llversa.
ll

12) What llis llthe lldifference llin llcontraction lland llmetabolic llfuel llpreference llbetween
llwhite llmuscle lland llred/dark? ll- llCORRECT llANSWER-Fast/white llmuscle llactivity llhas

llfaster lland llstronger llcontractions llthat llrequire llATP llproduced llfrom llaerobic llglycolysis

llas lla llfuel llsource. ll

Slow/red llmuscle llis llslower llbut llcan lllast llfor lla lllot lllonger lldue llto llgetting llATP llfrom llthe
llmore llefficient llaerobic llmetabolism llpathways. ll
ll

13) What llis llthe lloverall llbenefit llof llregulating llthe llrate llof llconversion llof llglucose llinto
llpyruvate? ll- llCORRECT llANSWER-Can llmake llsure llwe llhave llenough llintermediates

llthat llgo llinto llthe llmetabolic llpathways. ll

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