NR 565 MIDTERM EXAM 3 LATEST VERSIONS ACTUAL EXAM 140+
QUESTIONS AND CORRECT VERIFIED ANSWERS |ALREADY GRADED A+
1. Lab testing in patients who take ACEIs & ARBs: Potassium Serum
Creatinine
2. Lab testing in patients who take Amiodarone: Liver Function Thyroid
Function
Pulmonary Function & CXR
3. Lab testing in patients who take Anticonvulsants: Serum drug levels
4. Lab testing in patients who take Antidiabetic Drugs: Serum glucose
Hemoglobin A1c
5. Lab testing in patients who take Digoxin: Digoxin level Serum
electrolytes (if at risk)
Narrow therapeutic index
Hypokalemia, hypomagnesemia & hypocalcemia can increase toxicity risk
6. Lab testing in patients who take Diuretics (K Sparing): Serum electrolytes
Hyperkalemia can reach dangerous levels
Hypocalcemia & hypomagnesemia may occur
7. Lab testing in patients who take Diuretics (Thiazide & Loop): Serum elec- trolytes
Hypokalemia, hypomagnesemia & hyponatremia are common Thiazide
Diuretics —> hypercalcemia
Loop diuretics —> hypocalcemia
8. Lab testing in patients who take Lithium: CBC
Lithium level
Thyroid function
Renal function Serum
electrolytes
9. Lab testing in patients who take Methotrexate: CBC Liver
function
,Renal function
Pancytopenia, or decrease of any of the blood cell types Hepatotoxicity = AE
Renal toxicity = AE
10. Lab testing in patients who take NSAIDs (long-term use): CBC Serum
creatinine
Liver function
Anemia may occur if bleeding (may be occult) Prostaglandin
inhibition may decrease renal perfusion Rare but serious
liver injury may occur
,11. Lab testing in patients who take Statins: Liver function
Creatine kinase
Lipid panel
Elevations in liver enzymes
Creatine kinase can determine whether muscle pain is caused by injury secondary to drug
use
Lipids are checked to determine effect
12. Lab testing in patients who take Thiazolidinediones: Liver function
Risk for hepatotoxicity
13. Lab testing in patients who take Thyroid meds: TSH T4
14. Lab testing in patients who take Warfarin: PTT/INR
15. Drugs with narrow therapeutic index: carbamazepine, digoxin, lithium, pheny- toin,
theophylline - "Can Dangerous Levels Potentiate Toxicity"
16. Adverse effects of loop diuretics (Furosemide): Hyponatremia,
hypochloremia, and dehydration
Hypokalemia, hypotension
17. Use of thiazide diuretics: Essential HTN and heart failure
18. Adverse effects of ACE inhibitors: Hypotension, hyperK, intractable cough,
angioedema
Renal failure in patients with bilateral renal artery stenosis
19. CCB uses in treatment: Angina pectoris, essential HTN & cardiac dysrhythmias (atrial
flutter, atrial fibrillation & paroxysmal SVT)
20. CCB in afib MOA:: Blocks calcium channels in the heart & blood vessels Lowers
BP thru arteriolar dilation
Direct suppressant actions are balanced by reflex cardiac stimulation —> little net effect o
, the heart
21. Diltiazem and Digoxin Interaction: Can exacerbate digoxin-induced suppres- sion of
AV conduction
Can also intensify the cardiosuppressant effects of beta blockers
Basically increases what digoxin/beta blockers do, and also increases digoxin levels
22. Nifedipine MOA: Blocks calcium channels in VSM (vascular smooth muscle) &
promotes vasodilation - very little blockade of calcium channels in contrast to
verapamil
Direct:
Limited to blockade of calcium channels in VSM
QUESTIONS AND CORRECT VERIFIED ANSWERS |ALREADY GRADED A+
1. Lab testing in patients who take ACEIs & ARBs: Potassium Serum
Creatinine
2. Lab testing in patients who take Amiodarone: Liver Function Thyroid
Function
Pulmonary Function & CXR
3. Lab testing in patients who take Anticonvulsants: Serum drug levels
4. Lab testing in patients who take Antidiabetic Drugs: Serum glucose
Hemoglobin A1c
5. Lab testing in patients who take Digoxin: Digoxin level Serum
electrolytes (if at risk)
Narrow therapeutic index
Hypokalemia, hypomagnesemia & hypocalcemia can increase toxicity risk
6. Lab testing in patients who take Diuretics (K Sparing): Serum electrolytes
Hyperkalemia can reach dangerous levels
Hypocalcemia & hypomagnesemia may occur
7. Lab testing in patients who take Diuretics (Thiazide & Loop): Serum elec- trolytes
Hypokalemia, hypomagnesemia & hyponatremia are common Thiazide
Diuretics —> hypercalcemia
Loop diuretics —> hypocalcemia
8. Lab testing in patients who take Lithium: CBC
Lithium level
Thyroid function
Renal function Serum
electrolytes
9. Lab testing in patients who take Methotrexate: CBC Liver
function
,Renal function
Pancytopenia, or decrease of any of the blood cell types Hepatotoxicity = AE
Renal toxicity = AE
10. Lab testing in patients who take NSAIDs (long-term use): CBC Serum
creatinine
Liver function
Anemia may occur if bleeding (may be occult) Prostaglandin
inhibition may decrease renal perfusion Rare but serious
liver injury may occur
,11. Lab testing in patients who take Statins: Liver function
Creatine kinase
Lipid panel
Elevations in liver enzymes
Creatine kinase can determine whether muscle pain is caused by injury secondary to drug
use
Lipids are checked to determine effect
12. Lab testing in patients who take Thiazolidinediones: Liver function
Risk for hepatotoxicity
13. Lab testing in patients who take Thyroid meds: TSH T4
14. Lab testing in patients who take Warfarin: PTT/INR
15. Drugs with narrow therapeutic index: carbamazepine, digoxin, lithium, pheny- toin,
theophylline - "Can Dangerous Levels Potentiate Toxicity"
16. Adverse effects of loop diuretics (Furosemide): Hyponatremia,
hypochloremia, and dehydration
Hypokalemia, hypotension
17. Use of thiazide diuretics: Essential HTN and heart failure
18. Adverse effects of ACE inhibitors: Hypotension, hyperK, intractable cough,
angioedema
Renal failure in patients with bilateral renal artery stenosis
19. CCB uses in treatment: Angina pectoris, essential HTN & cardiac dysrhythmias (atrial
flutter, atrial fibrillation & paroxysmal SVT)
20. CCB in afib MOA:: Blocks calcium channels in the heart & blood vessels Lowers
BP thru arteriolar dilation
Direct suppressant actions are balanced by reflex cardiac stimulation —> little net effect o
, the heart
21. Diltiazem and Digoxin Interaction: Can exacerbate digoxin-induced suppres- sion of
AV conduction
Can also intensify the cardiosuppressant effects of beta blockers
Basically increases what digoxin/beta blockers do, and also increases digoxin levels
22. Nifedipine MOA: Blocks calcium channels in VSM (vascular smooth muscle) &
promotes vasodilation - very little blockade of calcium channels in contrast to
verapamil
Direct:
Limited to blockade of calcium channels in VSM
