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Pharm 2 Final Exam- Drugs For Dysrhythmias Test: Questions With Accurate Solutions

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Pharm 2 Final Exam- Drugs For Dysrhythmias Test: Questions With Accurate Solutions

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Pharm 2 Final Exam- Drugs For Dysrhythmias
Test: Questions With Accurate Solutions
In the normal heart, in which location does the contraction of heart
muscle begin?
1. Sinoatrial (SA) node
2. Atrioventricular (AV) node
3. Bundle of His
4. Purkinje fibers Correct Answer - 1. Sinoatrial (SA) node

Which laboratory parameter is monitored in a patient taking
amiodarone for the development of hepatotoxicity? Select all that
apply.
1. Serum bilirubin
2. Serum creatinine
3. Serum creatine kinase
4. Alanine aminotransferase
5. Aspartate aminotransferase Correct Answer - 4. Alanine
aminotransferase
5. Aspartate aminotransferase

Which finding would lead the nurse to suspect a possible
dysrhythmia when completing a routine assessment of a patient?
1. Irregular pulse
2. Heart rate 66 beats/min
3. Pulse pressure 6 mm Hg
4. Capillary refill 2 seconds Correct Answer - 1. Irregular pulse

Administering lidocaine at too high a dose can cause which effect?
1. Seizures
2. Hypertension
3. Increased respirations.
4. Tachycardia Correct Answer - 1. Seizures

, For which rhythm is the use of amiodarone, a K channel blocker, be
contraindicated?
1. Supraventricular tachycardia
2. Atrial Fibrillation
3. Atrial Flutter
4. Third-degree heart block Correct Answer - 4. Third degree
heart block

Class IV antiarrhythmics or calcium channel blockers have what
action on the cardiovascular system?
1. Increase heart rate
2. Decrease refractory period
3. Increase blood vessel spasm
4. Decrease flow of calcium ions Correct Answer - 4. Decrease
flow of calcium ions

Which complication might be anticipated in a patient taking a
potassium channel blocker?
1. Asthma
2. Edema
3. Pneumonitis
4. Myocardial toxicity Correct Answer - 3. Pneumonitis

By which mechanism of action does propafenone work?
1. Slowing the conduction rate through the atria and ventricles of
the heart.
2. Shortening the duration of the electrical stimulation and the time
between the electrical impulses.
3. Prolonging the duration of the electrical stimulation on cells and
the refractory time between electrical impulses.
4. Blocking calcium ion flow, prolonging duration of the electrical
stimulation, and slowing atrioventricular (AV) node conduction.

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