• Wrong document? Swap it for free
  • Written by students who passed
  • Immediately available after payment
  • Read online or as PDF
Sell
Where do you study
Your language
Document preview thumbnail
Preview 2 out of 14 pages
Exam (elaborations)

NUR 226 Latest Exam 2 Questions And Actual Answers.

Document preview thumbnail
Preview 2 out of 14 pages

RAAS regulates long-term BP and extracellular volume 1. Angiotemsionogen released by the liver in response to low BP and changes in blood volume 2. Low fluid volume stimulates the kidney to release renin which causes the liver to convert angiotensinogen to angiotensin I 3. Angiotensin I travels to the lung where it is converted by Angiotensin II by ACE (angiotensin-converting enzyme) 4. Angiotensin II acts on the adrenal glands to cause the release of aldosterone, which then causes fluid retention 5. Angiotensin II is a potent vasoconstrictor-- it also seems to cause inappropriate remodeling of the heart after a heart attack 6. This ultimately causes the nephron to retain fluid and the BP goes up - Answer Renin Angiotensin Aldosterone System - receptors in the carotid sinus, aorta, and left ventricle - sense BP and can alter the BP by altering heart rate - can also impact vasodilation and vasoconstriction - Answer Arterial baroreceptors - helps maintain consistent levels of tissue perfusion - regulates based on mean arterial pressure - alters the resistance (diameter) in arterioles - helps keep consistent BP at the tissue levels despite changes that are occurring in other mechanisms - Answer Vascular autoregulation Systolic— less than 120 AND Diastolic— less than 80 - Answer Normal BP Systolic— 120-129 AND Diastolic— less than 80 - Answer Elevated BP Systolic— 130-139 OR Diastolic— 80-89 - Answer High BP Stage 1 Systolic— 140 or higher OR Diastolic— 90 or higher - Answer High BP Stage 2

Content preview

NUR 226 Latest Exam 2 Questions And
Actual Answers.
RAAS regulates long-term BP and extracellular volume

1. Angiotemsionogen released by the liver in response to low BP and changes in blood volume

2. Low fluid volume stimulates the kidney to release renin which causes the liver to convert
angiotensinogen to angiotensin I

3. Angiotensin I travels to the lung where it is converted by Angiotensin II by ACE (angiotensin-
converting enzyme)

4. Angiotensin II acts on the adrenal glands to cause the release of aldosterone, which then
causes fluid retention

5. Angiotensin II is a potent vasoconstrictor-- it also seems to cause inappropriate remodeling of
the heart after a heart attack

6. This ultimately causes the nephron to retain fluid and the BP goes up - Answer Renin
Angiotensin Aldosterone System



- receptors in the carotid sinus, aorta, and left ventricle

- sense BP and can alter the BP by altering heart rate

- can also impact vasodilation and vasoconstriction - Answer Arterial baroreceptors



- helps maintain consistent levels of tissue perfusion

- regulates based on mean arterial pressure

- alters the resistance (diameter) in arterioles

- helps keep consistent BP at the tissue levels despite changes that are occurring in other
mechanisms - Answer Vascular autoregulation



Systolic— less than 120 AND Diastolic— less than 80 - Answer Normal BP



Systolic— 120-129 AND

Diastolic— less than 80 - Answer Elevated BP



Systolic— 130-139 OR

Diastolic— 80-89 - Answer High BP Stage 1

, Systolic— higher than 180 AND/OR

Diastolic— higher than 120 - Answer Hypertensive crisis



- Occurs when there is NO KNOWN CAUSE of hypertension— usually because of an absence of
underlying disease process

- Occurs due to complicated interactions of genetics and the environment, and involving several
neurohormonal effects (SNS, RAAS, Natriuretic peptides) - Answer Primary/Essential
hypertension



- smoking

- excess sodium intake

- sedentary lifestyle

- hyperlipidemia

- stress

- family history and genetics

- obesity

- age >60

- African Americans

- high alcohol consumption - Answer Risk factors for primary hypertension



- hypertension with a KNOWN cause

- usually related to underlying disease or disorder: health conditions, certain medicines,
pregnancy, hormonal therapy, renal disease, tumors, drugs such as corticosteroids,
antihistamines, cocaine, and amphetamines

- treatment is to treat the underlying cause - Answer Secondary hypertension



NO! "silent killer"

- must look for signs of end-organ damage: chest pain (heart), headache (brain), visual changes
(eyes), weakness or pain in extremities (brain/stroke) - Answer Are there signs and symptoms
of hypertension?



- increased left ventricular work

Document information

Uploaded on
March 27, 2025
Number of pages
14
Written in
2024/2025
Type
Exam (elaborations)
Contains
Questions & answers
$10.99

Wrong document? Swap it for free Within 14 days of purchase and before downloading, you can choose a different document. You can simply spend the amount again.
Written by students who passed
Immediately available after payment
Read online or as PDF

Seller avatar
Reputation scores are based on the amount of documents a seller has sold for a fee and the reviews they have received for those documents. There are three levels: Bronze, Silver and Gold. The better the reputation, the more your can rely on the quality of the sellers work.
TestSolver9
3.6
(182)
Sold
1035
Followers
131
Items
32587
Last sold
1 day ago




Why students choose Stuvia

Created by fellow students, verified by reviews

Quality you can trust: written by students who passed their tests and reviewed by others who've used these notes.

Didn't get what you expected? Choose another document

No worries! You can instantly pick a different document that better fits what you're looking for.

Pay as you like, start learning right away

No subscription, no commitments. Pay the way you're used to via credit card and download your PDF document instantly.

Student with book image

“Bought, downloaded, and aced it. It really can be that simple.”

Alisha Student

Working on your references?

Create accurate citations in APA, MLA and Harvard with our free citation generator.

Working on your references?

Frequently asked questions