and Answers 100% Correct
Saper Paper mice make UCP's in hypothalamus by attaching hte coding region of an
uncoupler gene to regulatory region of an orexion gene
-UCP mouse>body weight than WT, lower body temp, same food intake/activity, greater life
span
Kleibers Law BMR=mass^2/3 but originally tried to prove it was mass^3/4 but was wrong
Goose Paper Purpse The blood of the bar headed goose would have an increased temp
dependence of O2
Bohr Effectt pH decreases and the partial pressure of CO2 increases, resulting in lower
affinity of Hb for O2
-results in a shift to the right of coorperative kinetics curve Hb binding of O2
Goose Paper(Temp affect on Hb affinity for O2) Lung Temp decreases, O2 affinity
increases
muscle temp incrases, O2 affinity decreases
, BIO 212 Final University of Oregon Questions
and Answers 100% Correct
Graded Potential membrane potential change that diminishes with distance and time when
propogated
Funny channel -voltage gated cation channels that remain open when membrane potential
is below threshold potential
-generate pacemaker potential that brings membrane potential from resting to threshold
EPSP Interaction that causes an increase in K+ movement through ligand gated channels
AcetylCholine (Ach) -Does not open or close funny channels, but reduces flow through
them
-Leads to opening of ligand gated K+ channels
-Slows pacemaker potential
ATP and PFK relationship -ATP is a substrate of PFk
-ATP is a direct competitive (allosteric) inhibitor of PFK
, BIO 212 Final University of Oregon Questions
and Answers 100% Correct
-Increase PFK activity-->increase ATP production
-incrase AMP concentration would lead to increase in PFK activity bc as AMP increase, ATP
decreases
HT1 Opens stomata when CO2 levels are low, when it is missing stomata fail to open in
respponse to low CO2
-inhibited by bicarbonate, which is made by CA
Affinity for O2 in RBS's high affinity----->low affinity
lung body tissue
high O2 low O2
low CO2 high CO2
low H+ high H+
-RBC's carry O2 from lung to body tissye
People with FH mutations lack functional LDL receptors