TEST BANK FOR NEUROSCIENCE
FUNDAMENTALS FOR REHABILITATION
LATEST UPDATED 2025 EXAM WITH
COMPLETE QUESTIONS AND CORRECT
ANSWERS ALREADY GRADED A+
Neuroplasticity includes:
ANSWER-1. Habituation
2. Experience-dependent neuroplasticity: learning and
memory
3. Cellular recovery after injury
Habituation
ANSWER-*Non-associative learning
*Decreased response to repeated benign stimulus
*Decrease in synaptic activity between sensory neurons
*with prolonged stimulation-#of synapses decrease
,*PT Habituation-intended to decr neural response to a
stimulus
Experience dependent Plasticity
ANSWER--with repetitions of a task, # of active regions
decrease
-eventually, when a tasks is learned, only a small /distint
region is active during task
-Plasticity of the intrinsic excitability of neurons by
functional change in ion channels
-Long term Potentiation-conversion of silent synapses to
active
-Long Term Depression-conversion of active synapses to
silent
TMS
ANSWER-transcranial magnetic stimulation; use of strong
magnets to briefly interrupt normal brain activity as a
way to study brain regions
,*can enhance or inhibit
Axonal injury in periphery-sprouting
ANSWER-1. Collateral Sprouting: 1 axon sprouting to
multiple cell bodies
2. Regenerative Sprouting: Multiple axons leading to 1
cell body
**Exercise begun 5 days after a peripheral nerve lesion
increases regeneration
**Synkinesis- improperly re-innervated mm =
unintended movement
Axonal injury in CNS-sprouting
ANSWER--Functional axon regeneration does not occur
in CNS axons
-Glial scars physically block axonal regeneration
-Denervation hypersensitivity-new receptor sites develop
in response to decreased neurotransmitter released
, -when neurotransmitters are released from other nearby
axons, increased hypersensitive response occurs
CNS Axonal injury, functional recovery
ANSWER-1. Synaptic hyper-effectiveness--only some
branches of a presynaptic axons are destroyed--release
of larger than normal amounts of transmitters
2. Unmasking of silent synapses
3. Neurogenesis-stem cells in brain are capable of
creating new neurons
4. Excitotoxicity-cell death caused by overexcitation of
neurons
Development of nervous system: 0-6+ weeks
ANSWER-Neural tube
chromosomes
-drugs
-chemicals
FUNDAMENTALS FOR REHABILITATION
LATEST UPDATED 2025 EXAM WITH
COMPLETE QUESTIONS AND CORRECT
ANSWERS ALREADY GRADED A+
Neuroplasticity includes:
ANSWER-1. Habituation
2. Experience-dependent neuroplasticity: learning and
memory
3. Cellular recovery after injury
Habituation
ANSWER-*Non-associative learning
*Decreased response to repeated benign stimulus
*Decrease in synaptic activity between sensory neurons
*with prolonged stimulation-#of synapses decrease
,*PT Habituation-intended to decr neural response to a
stimulus
Experience dependent Plasticity
ANSWER--with repetitions of a task, # of active regions
decrease
-eventually, when a tasks is learned, only a small /distint
region is active during task
-Plasticity of the intrinsic excitability of neurons by
functional change in ion channels
-Long term Potentiation-conversion of silent synapses to
active
-Long Term Depression-conversion of active synapses to
silent
TMS
ANSWER-transcranial magnetic stimulation; use of strong
magnets to briefly interrupt normal brain activity as a
way to study brain regions
,*can enhance or inhibit
Axonal injury in periphery-sprouting
ANSWER-1. Collateral Sprouting: 1 axon sprouting to
multiple cell bodies
2. Regenerative Sprouting: Multiple axons leading to 1
cell body
**Exercise begun 5 days after a peripheral nerve lesion
increases regeneration
**Synkinesis- improperly re-innervated mm =
unintended movement
Axonal injury in CNS-sprouting
ANSWER--Functional axon regeneration does not occur
in CNS axons
-Glial scars physically block axonal regeneration
-Denervation hypersensitivity-new receptor sites develop
in response to decreased neurotransmitter released
, -when neurotransmitters are released from other nearby
axons, increased hypersensitive response occurs
CNS Axonal injury, functional recovery
ANSWER-1. Synaptic hyper-effectiveness--only some
branches of a presynaptic axons are destroyed--release
of larger than normal amounts of transmitters
2. Unmasking of silent synapses
3. Neurogenesis-stem cells in brain are capable of
creating new neurons
4. Excitotoxicity-cell death caused by overexcitation of
neurons
Development of nervous system: 0-6+ weeks
ANSWER-Neural tube
chromosomes
-drugs
-chemicals
