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HESI Health Assessment and Physical Examination 2025 [ With Questions & Answers]

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HESI Health Assessment and Physical Examination 2025 [ With Questions & Answers]

Institution
HESI Health Assessment And Physical
Course
HESI Health Assessment and Physical

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HESI Health Assessment and Physical Examination
2025 [ With Questions & Answers]

A decrease in the serum sodium level is typically followed , - ANSWER-
by a decrease in serum osmolality (example <270 mOsm/L)

metabolic acidosis results in hyperkalemia as H ions are shifted into the
cell - ANSWER-to raise the pH and potassium leaves the cell and enters
the bloodstream

Catecholamines are released from - ANSWER-adrenal medulla

steroid hormones (cortisol and aldosterone) are released from -
ANSWER-adrenal cortex

are exchanged for potassium ions during alkalosis or acidosis -
ANSWER-are exchanged for potassium ions during alkalosis or acidosis

Nerve route injury could lead to - ANSWER-allodynia (pain that is
associated with a nonnoxious stimulus)

Potassium - ANSWER-is necessary for neuromuscular and
cardiovascular function

Calcium helps to regulate muscle contraction and relaxation - ANSWER-
helps to regulate muscle contraction and relaxation

hypercalcemia - ANSWER-contributes to muscle weakness and coma

hypocalcemia - ANSWER-contributes to muscle irritability and tetany

,Magnesium - ANSWER-helps with carbohydrate and protein
metabolism, affects neuromuscular function and produces vasodilation

Hypokalemia - ANSWER-lowers the resting membrane potential and
makes cells less irritable which could result in an ileus.

Hyperkalemia - ANSWER-may cause diarrhea, irritability, muscle
weakness and EKG changes

Hyperkalemia - ANSWER-tall tented T waves, absent p waves,
prolonged PR interval and QRS duration

Hypokalemia - ANSWER-may cause muscle weakness, EKG changes
(inverted T waves and ST depression), a weak irregular pulse,

Hypokalemia - ANSWER-paralytic ileus, tachydysrhythmias (premature
ventricular contractions, ventricular tachycardia), constipation and u
waves

hypernatremia - ANSWER-thirst, dry mucous membranes, lethargy,
restlessness, tachycardia and
hypertension

hypocalcemia - ANSWER-tetany, muscle twitching, bronchospasms,
laryngeal spasms, seizures and hyperirritability

A classic sign for low magnesium is - ANSWER-tetany (like
hypocalcemia)

Clinical manifestations of hyponatremia consist of - ANSWER-
headaches, seizures, lethargy, tachycardia, decrease in blood pressure,
thready pulse, hyperactive bowel sounds and abdominal cramp

, Hyponatremia - ANSWER-slows the depolarization of the cell
membrane

Hyponatremia - ANSWER-shifts fluid from the extracellular to the
intracellular compartment

Sodium could be loss from - ANSWER-the gastrointestinal (vomiting,
diarrhea, suctioning), renal (diuretics, adrenal insufficiency,

Sodium could be loss from - ANSWER-kidney disease) and
integumentary system )(ascites, burns, peripheral edema)

In hyponatremia there is a reduction in intravascular volume - ANSWER-
so blood pressure decreases

Restrict fluids in the setting of - ANSWER-hyponatremia with fluid
volume overload

Monitor level of consciousness, vital signs, intake and output, and
weight in the setting of - ANSWER-hyponatremia

Severe hyponatremia - ANSWER-could result in coma, respiratory arrest
or seizures

Hypernatremia increases - ANSWER-the serum osmolality and pulls
water out of the cells

Excessive sodium intake, excessive sodium retention and a loss in fluid
from being - ANSWER-NPO or from illness (hyperglycemia,
watery diarrhea, diabetes insipidus or diarrhea) puts the client at risk
for hypernatremia

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HESI Health Assessment and Physical
Course
HESI Health Assessment and Physical

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