Solutions And 100% Correct Verified Answers| Well
Graded A+
Type II antiarrhythmics - CORRECT ANSWERS-Beta-blockers: reduce rate and
conduction
Type III antiarrhythmics - CORRECT ANSWERS-potassium channel blockers: prolong
the refractory period
**may produce PR arrhythmias like torsade’s de pointes
i.e. amiodarone, sotalol
Type IV antiarrhythmics - CORRECT ANSWERS-calcium channel blockers: slow
conduction velocity, (prolongs QRS) and lengthens the refractory period (prolongs QT
interval)
i.e. verapamil, diltiazem
Nitrates - CORRECT ANSWERS-decrease the heart's oxygen demand (reduce
afterload) i.e. nitroglycerin, isosorbide mononitrate, isosorbide dinitrate
Epinephrine - CORRECT ANSWERS-causes vasoconstriction, increased HR and
contractility
Atropine - CORRECT ANSWERS-antimuscarinic agent: blocks the effect of
acetylcholine at the sinus and AV nodes
Lack of oxygen to the brain will cause... - CORRECT ANSWERS-the brain to become
acidic and will cause permanent damage or death within 5-6 minutes
Isuprel - CORRECT ANSWERS-adrenergic agonist: stimulates beta receptors causing
increased HR and force of contraction as well as slight vasodilation
Alpha blockers - CORRECT ANSWERS-block alpha-1 receptors to inhibit
norepinephrine at the receptor site
i.e. doxazosin, prazosin, terazosin
Verapamil - CORRECT ANSWERS-antianginal, antihypertensive, antiarrhythmic --
smooth muscle relaxation, widening of blood vessels to increase oxygen delivery to
heart. Decreases conduction through the AV node.