A
VI
TU
IS
M
O
MSN 570 Questions and
A
N
Answers 2024 Version
JP
all 100% VERIFIED
, MI - ANSWER Death of myocardium from sudden blockage of coronary artery blood
flow
2. What causes cell death in MI? - ANSWER unstable plaque breaks off, triggering
platelet aggregation, coagulation cascade and thrombus formation. continued no blood
flow in 10 seconds ischemia, 20 minutes cell die
A
3. What consequence of cell death leads to CP? - ANSWER hypoxia as cannot
meet cell demand for oxygen
VI
4. How is MI diagnosed - ANSWER EKG (STEMI) or elevated Troponin
(non-STEMI)
TU
5. What might you find in blood consistent with MI? - ANSWER elevated CK MB,
Troponin
IS
6. Where does CK MB and Troponin come from? - ANSWER Release of lysosomal
enzymes; CK MB detected after myocardial necrosis, Troponin released from damaged
heart muscle
M
7. 3 mechanism of cell injury related to reperfussion injury - ANSWER ischemia,
necrosis, reperfussion l/t RAS
O
8. define reperfussion injury - ANSWER injury that occurs after oxygen is
reintroduced to tissue perfusion
A
9. how does inflammatory process contribute to reperfussion injury? - ANSWER as
N
oxygen is reintroduced it comes in contact with damaged proteins triggering
inflammatory response
JP
10. how does oxidative process contribute to RI? - ANSWER RAS causes
impairment of cell membrane and pro-apotptoic cells release. making cell unstable
11. what is primary Hypertension? - ANSWER Essential or idiopathic, elevated BP
without an identified cause, accounts for 90 - 95% of all cases of hypertension?
12. what is secondary hypertension? - ANSWER Elevated BP, labile BP; related to
conditions ie kidney dz; resistant to BP meds
VI
TU
IS
M
O
MSN 570 Questions and
A
N
Answers 2024 Version
JP
all 100% VERIFIED
, MI - ANSWER Death of myocardium from sudden blockage of coronary artery blood
flow
2. What causes cell death in MI? - ANSWER unstable plaque breaks off, triggering
platelet aggregation, coagulation cascade and thrombus formation. continued no blood
flow in 10 seconds ischemia, 20 minutes cell die
A
3. What consequence of cell death leads to CP? - ANSWER hypoxia as cannot
meet cell demand for oxygen
VI
4. How is MI diagnosed - ANSWER EKG (STEMI) or elevated Troponin
(non-STEMI)
TU
5. What might you find in blood consistent with MI? - ANSWER elevated CK MB,
Troponin
IS
6. Where does CK MB and Troponin come from? - ANSWER Release of lysosomal
enzymes; CK MB detected after myocardial necrosis, Troponin released from damaged
heart muscle
M
7. 3 mechanism of cell injury related to reperfussion injury - ANSWER ischemia,
necrosis, reperfussion l/t RAS
O
8. define reperfussion injury - ANSWER injury that occurs after oxygen is
reintroduced to tissue perfusion
A
9. how does inflammatory process contribute to reperfussion injury? - ANSWER as
N
oxygen is reintroduced it comes in contact with damaged proteins triggering
inflammatory response
JP
10. how does oxidative process contribute to RI? - ANSWER RAS causes
impairment of cell membrane and pro-apotptoic cells release. making cell unstable
11. what is primary Hypertension? - ANSWER Essential or idiopathic, elevated BP
without an identified cause, accounts for 90 - 95% of all cases of hypertension?
12. what is secondary hypertension? - ANSWER Elevated BP, labile BP; related to
conditions ie kidney dz; resistant to BP meds
