10/15/24, 8:42 PM NUR 529 Exam 2/2024-2025 update Set Test Questions And Revised Correct Answers | Already Passed/get it 100% accurate…
NUR 529 Exam 2/2024-2025 update Set Test
Questions And Revised Correct Answers |
Already Passed/get it 100% accurate!!
Terms in this set (200)
is the main source of afterload on the left heart, Left
Systemic arterial pressure ventricle afterload is increased with narrowing
(stenosis) of the aortic valve
Vessel Spasm, formation of the platelet plug, blood
Stages of Hemostasis
coagulation (clot retraction, clot dissolution)
First stage of hemostasis, constricts the vessel and
Vessel Spasm reduces blood flow. It is a transient event that
usually lasts minutes or hours.
Second stage of hemostasis, 1. platelets are
attracted to damaged vessel wall. 2. activation by
sub-endothelial tissue. 3. change from smooth disks
Formation of the platelet to spiny spheres. 4. exposing glycoprotein
plug receptors on their surfaces. This step requires a
protein molecule called von Willebrand factor that
leaks into the injured tissue from the plasma.
(adhesion, activation, aggregation)
During Hemostasis, Smooth disks to spiny spheres
platelets change from .....
Formation of the platelet plug requires a protein
von Willebrand factor
molecule called (helps platelets stick together)
Exposed during the formation of the platelet plug
Glycoprotein Receptors when platelets change from smooth disks to spiny
spheres
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, 10/15/24, 8:42 PM NUR 529 Exam 2/2024-2025 update Set Test Questions And Revised Correct Answers | Already Passed/get it 100% accurate…
Third step of hemostasis: stabilization of the plug
1.Blood coagulation or
occurs as the coagulation pathway is activated on
development of an
the platelet surface, and fibrinogen is converted to
insoluble fibrin clot
fibrin.
Results from a decrease in platelet production,
Thrombocytopenia increased sequestration of platelets in the spleen, or
decreased platelet survival
Drug induced can be from aspirin, atorvastatin, antibiotics and of
thrombocytopenia course Heparin.
the consolidation or tightening of the fibrin clot
Clot retraction pulls the edges of the damaged vessel close
together, allowing for repair of the vessel lining.
fibrinolysis occurs through conversion of
plasminogen to plasmin by tissue plasminogen
activator (tPA), which is released from damaged
Clot dissolution
endothelial cells. This conversion results in digestion
of fibrin fibers within the clot and the production of
fibrin degradation products.... Clot dissolves.
was formally called idiopathic thrombocytopenia
Immune purpura. ITP is the most common thrombocytopenic
thrombocytopenia disorder among children and often follows an upper
respiratory infection.
rare and caused by a deficiency of the gene
responsible for severing large von Willebrand factor
Thrombotic
proteins. The unchecked platelet aggregation
Thrombocytopenic
results in microvascular occlusions...end organ
failure
Most often involves small vessels of mucous
Spontaneous bleeding
membranes and skin, common sites are nose,
from platelet disorders
mouth, GI, uterine
seen as pinpoint hemorrhages (petechiae), or
Cutaneous bleeding
purple areas of bruising (purpura)
Occur almost exclusively in conditions of platelet
Petechiae
deficiency, not dysfunction
120 days RBC lifespan
https://quizlet.com/957631555/nur-529-exam-22024-2025-update-set-test-questions-and-revised-correct-answers-already-passedget-it-100-accur… 2/8
NUR 529 Exam 2/2024-2025 update Set Test
Questions And Revised Correct Answers |
Already Passed/get it 100% accurate!!
Terms in this set (200)
is the main source of afterload on the left heart, Left
Systemic arterial pressure ventricle afterload is increased with narrowing
(stenosis) of the aortic valve
Vessel Spasm, formation of the platelet plug, blood
Stages of Hemostasis
coagulation (clot retraction, clot dissolution)
First stage of hemostasis, constricts the vessel and
Vessel Spasm reduces blood flow. It is a transient event that
usually lasts minutes or hours.
Second stage of hemostasis, 1. platelets are
attracted to damaged vessel wall. 2. activation by
sub-endothelial tissue. 3. change from smooth disks
Formation of the platelet to spiny spheres. 4. exposing glycoprotein
plug receptors on their surfaces. This step requires a
protein molecule called von Willebrand factor that
leaks into the injured tissue from the plasma.
(adhesion, activation, aggregation)
During Hemostasis, Smooth disks to spiny spheres
platelets change from .....
Formation of the platelet plug requires a protein
von Willebrand factor
molecule called (helps platelets stick together)
Exposed during the formation of the platelet plug
Glycoprotein Receptors when platelets change from smooth disks to spiny
spheres
https://quizlet.com/957631555/nur-529-exam-22024-2025-update-set-test-questions-and-revised-correct-answers-already-passedget-it-100-accur… 1/8
, 10/15/24, 8:42 PM NUR 529 Exam 2/2024-2025 update Set Test Questions And Revised Correct Answers | Already Passed/get it 100% accurate…
Third step of hemostasis: stabilization of the plug
1.Blood coagulation or
occurs as the coagulation pathway is activated on
development of an
the platelet surface, and fibrinogen is converted to
insoluble fibrin clot
fibrin.
Results from a decrease in platelet production,
Thrombocytopenia increased sequestration of platelets in the spleen, or
decreased platelet survival
Drug induced can be from aspirin, atorvastatin, antibiotics and of
thrombocytopenia course Heparin.
the consolidation or tightening of the fibrin clot
Clot retraction pulls the edges of the damaged vessel close
together, allowing for repair of the vessel lining.
fibrinolysis occurs through conversion of
plasminogen to plasmin by tissue plasminogen
activator (tPA), which is released from damaged
Clot dissolution
endothelial cells. This conversion results in digestion
of fibrin fibers within the clot and the production of
fibrin degradation products.... Clot dissolves.
was formally called idiopathic thrombocytopenia
Immune purpura. ITP is the most common thrombocytopenic
thrombocytopenia disorder among children and often follows an upper
respiratory infection.
rare and caused by a deficiency of the gene
responsible for severing large von Willebrand factor
Thrombotic
proteins. The unchecked platelet aggregation
Thrombocytopenic
results in microvascular occlusions...end organ
failure
Most often involves small vessels of mucous
Spontaneous bleeding
membranes and skin, common sites are nose,
from platelet disorders
mouth, GI, uterine
seen as pinpoint hemorrhages (petechiae), or
Cutaneous bleeding
purple areas of bruising (purpura)
Occur almost exclusively in conditions of platelet
Petechiae
deficiency, not dysfunction
120 days RBC lifespan
https://quizlet.com/957631555/nur-529-exam-22024-2025-update-set-test-questions-and-revised-correct-answers-already-passedget-it-100-accur… 2/8
