1|Page
NURS 8022 EXAM 1 NEWEST 2024
ACTUAL EXAM COMPLETE EXAM
QUESTIONS AND CORRECT ANSWERS
(VERIFIED ANSWERS) |ALREADY
GRADED A+||BRAND NEW!!
Current flow from one excitable cell to the next through space
between cells called gap junctions. This location is called
_________. - ANSWER- Electrical synapse
Gap between the presynaptic membrane and the postsynaptic
cell membrane called the synaptic cleft. This location is
called_______. - ANSWER- Chemical synapse
AP in presynaptic cells causes________ - ANSWER- Ca2+
channels to open
__________ and diffuses across the synaptic cleft - ANSWER-
Neurotransmitter is released
,2|Page
Neurotransmitters bind to_____ - ANSWER- Receptors on
postsynaptic membrane
What action causes depolarization of postsynaptic cell (more
positive) - ANSWER- Excitatory
What action will cause hyper polarization of the postsynaptic
cell (making the cell negative) - ANSWER- Inhibitory
Specialized synapse between a motor neuron and a muscle fiber
is called _________. - ANSWER- Neuromuscular Transmission
Where does synapse occur? - ANSWER- On the motor end plate
What occurs to produce: An end plate potential; which will
normally initiate an AP if the local spread of current is sufficient
to open voltage sodium channels. - ANSWER- Opening of the
nACh receptor channels produces:
Inhibitor drug: _____________ Block nicotinic Ach channels by
competing for AcH binding site
reduces amplitude of end plate potential therefore no AP -
ANSWER- Curariform drugs (D-turbocurarie)
, 3|Page
Inhibitor drug: ____________ Decreases the release of AcH
from nerve terminals
Insufficient stimulus to initiate an AP. - ANSWER- Botulinum
toxin
_________ bind and activate nicotinic AcH receptors
Not destroyed by AcHE- prolonged effect - ANSWER- AcH
like drugs (Methacholine, carbachol, nicotine)
___________ inactivate acetylcholinesterase; can treat MG -
ANSWER- Anti AcHe drugs (neostigmine, physostigmine,
diisopropyl flurophosphate or "nerve gas")
Which Autoimmune disorder that attacks Ach nicotinic
cholinergic receptors at NM junction. This results in weakness
in face and legs. Treatments include anti-cholinesterase's and
immunosuppressive drugs. - ANSWER- Myesthenia Gravis
a _________ is a functional contracting unit of the muscle fiber.
- ANSWER- Sarcomere
myosin is a ________ - ANSWER- Thick filament
NURS 8022 EXAM 1 NEWEST 2024
ACTUAL EXAM COMPLETE EXAM
QUESTIONS AND CORRECT ANSWERS
(VERIFIED ANSWERS) |ALREADY
GRADED A+||BRAND NEW!!
Current flow from one excitable cell to the next through space
between cells called gap junctions. This location is called
_________. - ANSWER- Electrical synapse
Gap between the presynaptic membrane and the postsynaptic
cell membrane called the synaptic cleft. This location is
called_______. - ANSWER- Chemical synapse
AP in presynaptic cells causes________ - ANSWER- Ca2+
channels to open
__________ and diffuses across the synaptic cleft - ANSWER-
Neurotransmitter is released
,2|Page
Neurotransmitters bind to_____ - ANSWER- Receptors on
postsynaptic membrane
What action causes depolarization of postsynaptic cell (more
positive) - ANSWER- Excitatory
What action will cause hyper polarization of the postsynaptic
cell (making the cell negative) - ANSWER- Inhibitory
Specialized synapse between a motor neuron and a muscle fiber
is called _________. - ANSWER- Neuromuscular Transmission
Where does synapse occur? - ANSWER- On the motor end plate
What occurs to produce: An end plate potential; which will
normally initiate an AP if the local spread of current is sufficient
to open voltage sodium channels. - ANSWER- Opening of the
nACh receptor channels produces:
Inhibitor drug: _____________ Block nicotinic Ach channels by
competing for AcH binding site
reduces amplitude of end plate potential therefore no AP -
ANSWER- Curariform drugs (D-turbocurarie)
, 3|Page
Inhibitor drug: ____________ Decreases the release of AcH
from nerve terminals
Insufficient stimulus to initiate an AP. - ANSWER- Botulinum
toxin
_________ bind and activate nicotinic AcH receptors
Not destroyed by AcHE- prolonged effect - ANSWER- AcH
like drugs (Methacholine, carbachol, nicotine)
___________ inactivate acetylcholinesterase; can treat MG -
ANSWER- Anti AcHe drugs (neostigmine, physostigmine,
diisopropyl flurophosphate or "nerve gas")
Which Autoimmune disorder that attacks Ach nicotinic
cholinergic receptors at NM junction. This results in weakness
in face and legs. Treatments include anti-cholinesterase's and
immunosuppressive drugs. - ANSWER- Myesthenia Gravis
a _________ is a functional contracting unit of the muscle fiber.
- ANSWER- Sarcomere
myosin is a ________ - ANSWER- Thick filament
