INTENSIVE REVIEW, FOURTH EDITION BY MARIA T. CODINA
LEIK 270 CORRECTLY ANSWERED QUESTIONS | ANSWERS IN NON
- BOLD SUCCESSMAESTRO 2 /229 SUCCESSMAESTRO 3 /229TEST BANK FOR FAMILY NURSE PRACTITIONER CERTIFICATION
INTENSIVE REVIEW, FOURTH EDITION BY MARIA T. CODINA
LEIK 270 CORRECTLY ANSWERED QUESTIONS | ANSWERS IN NON
- BOLD
1.In the person with reactive thrombocytosis, clotting risk is typically absent until a platelet count of:
a.e400,000 mm3
b.e800,000 mm3.
c.e1 million mm3.
d.e2 million mm3:.C. e1 million mm3.
Reactive thrombocytosis is an increase in platelet count in response to a stimulus. There are several causes of reactive thrombocytosis, including acute bleeding and blood loss, allergic reactions, cancer, infections, some types of anemia, major surgery,
splenectomy, and some types of medications. Treatment of reactive throm- bocytosis is directed at the underlying cause and platelet counts usually return
to normal once the underlying condition is brought under control. An increased risk of blood clots can occur once the platelet count is e1 million mm3 (NL = 150,000-
450,000 mm3). Topic: Assessment and Intervention in Select Hematologic Disorders SUCCESSMAESTRO 4 /2292.Antibodies are primarily formed by:
a.T-cell lymphocytes.
b.B-cell lymphocytes.
c.Mast cells.
d.Eosinophils.: B. B-cell lymphocytes.
B cells are involved in antibody formation. Delaying the hypersensitivity response is a primary function of T-cell lymphocytes. Granulocytes and monocytes are involved in the phagocytosis of circulating debris and pathogens, such as bacteria or fungi. Topic: Assessment and Intervention in Select Hematologic Disorders
3.Constitutional neutropenia is most often found in:
a.Those of African ancestry.
b.Sepsis recovery.
c.Post transfusion states.
d.Patients who have undergone splenectomy.: A. Those of African ancestry.
Constitutional neutropenia (also called benign ethnic neutropenia or benign familial neutropenia) is a condition more often observed in people with African and Middle Eastern ancestry. The condition is potentially caused by a defect in the release of mature WBCs from the bone marrow to peripheral circulation. Despite a consistently