Agacnp Board Prep Questions And Answers, Rated A+| Latest Updates 2024/25 Approved|

AGACNP BOARD PREP QUESTIONS AND ANSWERS, RATED A+ 4 types of burns - -thermal chemical electrical radiation 5 Ps of pheochromacytoma - -pressure (persistent/ resistent HTN) pain (Headache) palpitations ( tachycardia and tremors) Perspiration ( profuse with flushing) Pallor ( secondary to vasccontriction) 6 Ps of compartment syndrome - -Pain Parathesia Paresis Pallor Poiklothermia pulselessness Absolute contraindications for fibrinolysis - -HX of Cerebrovascular event Non-hemorrhagic stroke of head trauma 3 months ago cranial or spinal trauma 2 months ago known bleeding siathesis active internal bleeding Acetaminophen Overdose - -Nausea, vomiting, abdominal pain Need OD timeframe APAP/ acetaminophen levels 4 hrs after injestion liver enzymes INR and coags N-acetylcysteine (NAC) [150 mg/kg MAX] to be administered within 8 hrs of overdose Acrogemagly - -piruitary problem which produces too much growth hormone Actinic keratosis - -small patches occuring on sub exposed parts of the body. can progress to squamous cell carcinoma can removed with liquid nitrogen Acute hemolytic reaction pathophysiology and treatment - -Occurs 24 hrs after transfusion Secondary to an ABO incompatainlity Develop antibodies against donor RBCs Presents as: fever, flank pain and renal failure Treat with aggressive IV fluids and maintain adequate urinary output - diuretics, mannitol Acute lymphocytic leukemia (ALL_) pathophysiology - -involved immature B and/or T cells (from bone marrow) occurs in children acute onset of symptoms related to depressed bone marrow function and CNS manifestations presence of anemia and thrombocytopenia. Variable WBCs with 30% lymphoblasts Acute myelogenous meukemia (AML) pathophysiology - -Involves imaature myeloid linage cells (from bone marrow) occurs in older adults symptoms of anemia and spontaneous bleeding, petechiae and ecchymosis presence of anemia, neutropenia and thrombocytopenia. 30% meloblasts and presence of Auer rods Addisons disease - -adrenal insufficiency/ hypocortisolism Primary causes: damage to the adrenal cortex Secondary causes: pituitary failure ro release ACT