Articles Psychobiology of Food Choice and Eating
Behaviour
Lecture 1: Models of food choice
Book chapters 1, 2, 3, p1-58.
Lecture 2: Appetite and Satiety
Rolls, 2009, The relationship between dietary energy density and
energy intake.
http://www.sciencedirect.com/science/article/pii/S003193840900122X
Lowering the ED (kcal/g) by increasing the volume of preloads without changing
macronutrient content can enhance satiety and reduce subsequent energy intake
at a meal. Since people tend to eat a consistent weight of food, when the ED of
the available foods is reduced, energy intake is reduced.
Blundell et al. 2010, Appetite control: methodological aspects of the
evaluation of foods.
http://onlinelibrary.wiley.com/doi/10.1111/j.1467-
789X.2010.00714.x/abstract;jsessionid=9C30248D43313A033FAAAF85AFBE3D82
.f01t01
Not important
De Graaf and Kok, 2010, Slow food, fast food and the control of food
intake
https://edu6.wur.nl/bbcswebdav/pid-912149-dt-content-rid-
5453139_1/courses/HNE30306_2015_5/DeGraafnrendo%202010%2041.pdf
Two elements of our food supply and eating environment that facilitate high
energy intake: a high eating rate and distraction of attention from eating. These
two elements are believed to undermine our body’s capacity to regulate its
energy intake at healthy levels because they impair the congruent association
between sensory signals and metabolic consequences.
Foods that can be eaten quickly lead to high food intake and low satiating effects
—the reason being that these foods only provide brief periods of sensory
exposure, which give the human body insufficient cues for satiation.
Lecture 3: Physiological signals of satiety
Again Blundell et al. 2010, see lecture 2.
Delzenne et al., 2010, Gastrointestinal targets of appetite regulation in
humans.
http://onlinelibrary.wiley.com/doi/10.1111/j.1467-789X.2009.00707.x/abstract
- Gastric emptying, stomach distension and gut motility influence appetite;
Direct, inverse, and causal relation between gastric distension and
appetite (mainly satiation).
Weaker relationship between gastric emptying and appetite (mainly
satiety).
, - GI peptides are involved in the control of satiety and appetite (ghrelin,
cholecystokinin, glucagon-like peptide, peptide tyrosin-tyrosin) and can be
used as potential biomarkers.
- New developments in techniques and methods for the assessment of
physiological targets involved in appetite regulation (including brain
imaging, interesting new experimental approaches, targets and markers).
- Selected biomarkers becoming essential as representative markers of
appetite regulation.
Selection of arguments supporting a role for ghrelin as a key factor in preprandial
hunger:
1. Produced by organs recently exposed to food (stomach and duodenum).
2. Triggers eating when administered at times of minimal spontaneous food
intake.
3. Extremely rapid and short-lived orexigenic actions, as required for a signal
influencing individual meal-related behaviour.
Behaviour
Lecture 1: Models of food choice
Book chapters 1, 2, 3, p1-58.
Lecture 2: Appetite and Satiety
Rolls, 2009, The relationship between dietary energy density and
energy intake.
http://www.sciencedirect.com/science/article/pii/S003193840900122X
Lowering the ED (kcal/g) by increasing the volume of preloads without changing
macronutrient content can enhance satiety and reduce subsequent energy intake
at a meal. Since people tend to eat a consistent weight of food, when the ED of
the available foods is reduced, energy intake is reduced.
Blundell et al. 2010, Appetite control: methodological aspects of the
evaluation of foods.
http://onlinelibrary.wiley.com/doi/10.1111/j.1467-
789X.2010.00714.x/abstract;jsessionid=9C30248D43313A033FAAAF85AFBE3D82
.f01t01
Not important
De Graaf and Kok, 2010, Slow food, fast food and the control of food
intake
https://edu6.wur.nl/bbcswebdav/pid-912149-dt-content-rid-
5453139_1/courses/HNE30306_2015_5/DeGraafnrendo%202010%2041.pdf
Two elements of our food supply and eating environment that facilitate high
energy intake: a high eating rate and distraction of attention from eating. These
two elements are believed to undermine our body’s capacity to regulate its
energy intake at healthy levels because they impair the congruent association
between sensory signals and metabolic consequences.
Foods that can be eaten quickly lead to high food intake and low satiating effects
—the reason being that these foods only provide brief periods of sensory
exposure, which give the human body insufficient cues for satiation.
Lecture 3: Physiological signals of satiety
Again Blundell et al. 2010, see lecture 2.
Delzenne et al., 2010, Gastrointestinal targets of appetite regulation in
humans.
http://onlinelibrary.wiley.com/doi/10.1111/j.1467-789X.2009.00707.x/abstract
- Gastric emptying, stomach distension and gut motility influence appetite;
Direct, inverse, and causal relation between gastric distension and
appetite (mainly satiation).
Weaker relationship between gastric emptying and appetite (mainly
satiety).
, - GI peptides are involved in the control of satiety and appetite (ghrelin,
cholecystokinin, glucagon-like peptide, peptide tyrosin-tyrosin) and can be
used as potential biomarkers.
- New developments in techniques and methods for the assessment of
physiological targets involved in appetite regulation (including brain
imaging, interesting new experimental approaches, targets and markers).
- Selected biomarkers becoming essential as representative markers of
appetite regulation.
Selection of arguments supporting a role for ghrelin as a key factor in preprandial
hunger:
1. Produced by organs recently exposed to food (stomach and duodenum).
2. Triggers eating when administered at times of minimal spontaneous food
intake.
3. Extremely rapid and short-lived orexigenic actions, as required for a signal
influencing individual meal-related behaviour.
