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Test bank to Neuroscience of clinical psychiatry 4th edition by Higgins

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TEST BANK

, CHAPTER LIST

o SECTION 1: The Neuroscience Model
 Chapter 1: Introduction
 Chapter 2: Neuroanatomy
 Chapter 3: Neural Cells
 Chapter 4: Networks
 Chapter 5: Neurotransmitters
 Chapter 6: Receptors and Signaling the Nucleus
 Chapter 7: Genetics and Epigenetics
o SECTION 2: Modulators
 Chapter 8: Hormones and the Brain
 Chapter 9: Plasticity and Adult Development
 Chapter 10: Immunity and Inflammation
 Chapter 11: The Electrical Brain
o SECTION 3: Behaviors
 Chapter 12: Pain
 Chapter 13: Wanting and Liking
 Chapter 14: Appetite
 Chapter 15: Anger and Aggression
 Chapter 16: Sleep and Rhythms
 Chapter 17: Sex and the Brain
 Chapter 18: Social Attachment
 Chapter 19: Memory
 Chapter 20: Intelligence
 Chapter 21: Attention
o SECTION 4: Disorders
 Chapter 22: Depression
 Chapter 23: Anxiety
 Chapter 24: Schizophrenia
 Chapter 25: Addiction
 Chapter 26: Alzheimer Diseas

,SECTION 1: THE NEUROSCIENCE MODEL




Chapter 1 — Introduction: The

Neuroscience Model and Neuroimaging



1. How did early twentieth-century theorists primarily view heredity in mental illness?

A. As an environmental trigger acting on uniform cellular biology

B. As an unlinked set of spontaneous somatic chromosome errors

C. As an inevitable inherited degeneration across family generations

D. As an unpredictable trait independent of biological lineage

Answer: C

Rationale: Early twentieth-century degeneration models conceptualized psychiatric illness as an

unavoidable, inherited biological vulnerability that worsened across successive family

generations. This deterministic view preceded modern quantitative genetics and neglected

gene-environment interactions. Understanding this history clarifies the shift toward complex

polygenic models in contemporary clinical neuroscience.

Keywords: heredity, historical psychiatry, degeneration theory




2. What was the main contribution of early brain dissection studies to psychiatry?

A. Demonstrating that localized tissue damage produces discrete functional deficits

B. Identifying cellular targets for selective psychotropic medications

C. Proving that psychiatric disorders arise primarily from diffuse metabolic toxins

D. Establishing whole-brain electrical synchronization during deep natural sleep

, Answer: A

Rationale: Early clinicopathological correlation studies demonstrated that focal anatomical

lesions correspond to specific behavioral and cognitive deficits. This established the principle of

functional localization, moving psychiatry away from vague humoral theories. It laid the

groundwork for modern structural and functional neuroanatomy.

Keywords: brain dissection, lesion localization, functional neuroanatomy




3. Which historical neuroscience concept represents a flawed localization doctrine?

A. Cortical cytoarchitecture mapping

B. Reflexive spinal connectivity

C. Transduction of neural impulses

D. Cranial surface phrenology

Answer: D

Rationale: Phrenology incorrectly posited that complex personality traits and psychiatric

tendencies could be mapped directly from cranial surface contours. Although methodologically

invalid, it popularized the concept that mental faculties reside in specialized cerebral

compartments. Modern neuroscience replaced these superficial claims with empirically verified

cortical circuity.

Keywords: phrenology, pseudoscience, cortical localization




4. What is the primary diagnostic limitation of plain cranial radiographs in psychiatry?

A. Total absence of ionizing radiation during procedural acquisition

B. Inability to distinguish soft brain parenchyma from surrounding tissues

C. Extreme sensitivity to microvascular white matter ischemic lesions

D. Prohibitive financial expense compared to advanced functional scanning

Answer: B

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Publisher: Unknown ISBN: 9781975212223 Edition: Unknown

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