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NSG 533 Exam 1 – GI & Diabetes Management 2026 | 133 Verified Q&A | PUD, GERD, Metformin & ADA Guidelines | Wilkes University | Graded A+

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NSG 533 Advanced Pharmacology GI & Diabetes Management Exam 1 | Wilkes University 2026 | Questions & Verified Answers | Latest Update | Graded A+ 133 questions with the correct answer marked on every item, covering GI and diabetes pharmacology for NSG 533 Exam 1 at Wilkes University. What's covered: - GI disorders and treatment – Helicobacter pylori's role in peptic ulcer disease, GERD diet modifications, self-care duration limits for OTC GERD medications, stepping up to a PPI when H2 blockers fail, alarm symptoms requiring GERD work-up, how obesity-related intra-abdominal pressure contributes to reflux, causes of chronic diarrhea - Diabetes management – overall treatment goals, ADA-recommended HgbA1c monitoring frequency and target (7%), priority monitoring parameters, adjusting treatment for consistently elevated blood glucose - Antidiabetic pharmacology – thiazolidinedione (TZD) mechanism of action, metformin contraindications including eGFR 30 mL/min/1.73m² - Medication safety – why understanding drug interactions matters specifically in GI treatment regimens Format: 62-page PDF, question-and-answer layout with correct responses highlighted. Instant download after purchase.

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NSG 533 Advanced Pharmacology GI & Diabetes Management
Exam 1 | Wilkes University 2026 | Questions & Verified Answers
| Latest Update | Graded A+
1. What bacterium is primarily associated with causing Peptic Ulcer Disease
(PUD)?

Streptococcus pneumoniae

Helicobacter pylori

Escherichia coli

Clostridium difficile

2. What are the goals of treatment for diabetes?

To check blood glucose once a day.

To prevent or delay complications and maintain quality of life

To cure diabetes

To maintain fasting blood glucose levels of 180-200

3. In the community setting how long should self-care with OTC meds for GERD
be limited to?

treat OTC indefinitely

3-4 days

1 week

2 weeks

4. What is the primary reason for understanding medication interactions in the
treatment of gastrointestinal conditions?

, To avoid adverse effects and ensure the effectiveness of treatment
regimens.

To eliminate the need for monitoring.

To simplify the treatment process.

To increase the dosage of medications.

5. Which of the following diet modifications is recommended to manage GERD
symptoms?

Elevate the head of the bed

All the above

Eating smaller more frequent meals

Avoid trigger foods

6. Monitoring a patient's hemoglobin A1c every months, until patient is
well-controlled on an established regimen, allows us to gauge their long-
term blood glucose control. The American Diabetes Association (ADA) states
the HgbA1c goal as _.

6 months, less than 7%

3 months, less than 7%

12 months, less than 6%

6 months, less than 6%

7. If a patient with diabetes is consistently experiencing high blood glucose
levels, what should be the primary focus of their treatment plan?

Adjusting their medication and lifestyle to maintain blood glucose
levels within target ranges.

Encouraging them to stop all medications.

, Focusing solely on weight loss without addressing blood glucose.

Increasing their carbohydrate intake significantly.


8. Describe the potential causes of chronic diarrhea as mentioned in the
context of gastrointestinal conditions.

Chronic diarrhea is only associated with medication side effects.

Chronic diarrhea can be caused by conditions like inflammatory
bowel disease or irritable bowel syndrome.

Chronic diarrhea is caused solely by dietary factors.

Chronic diarrhea is primarily due to infections.

9. What is the therapeutic action of the antidiabetic agent type,
thiazolidinediones (TZDs)?

Increase muscle, liver and adipose tissue sensitivity to insulin

Stimulate the release of insulin from functioning pancreatic beta-cells

Prolong gastric emptying and reduce glucagon secretion after a meal

Exert similar effects to GLP-1

10. For GERD, what agent would you recommend for a patient seeking relief
from frequent GERD but is non-responsive to H2 blockers?

H-2 blockers

PPIs

Antacids

11. Describe how increased intra-abdominal pressure from obesity affects the
lower esophageal sphincter.

, Increased intra-abdominal pressure has no effect on the lower
esophageal sphincter.

Increased intra-abdominal pressure decreases gastric acid
production, reducing reflux.

Increased intra-abdominal pressure enhances the function of the
lower esophageal sphincter, preventing reflux.

Increased intra-abdominal pressure can lead to lower esophageal
sphincter dysfunction, which allows for increased reflux.

12. Which of the following is most important to monitor closely when managing
diabetes?

Cholesterol

Blood sugar

Sodium

Blood pressure

13. Which of the following is a contraindication to metformin use?

Estimated glomerular filtration rate [eGFR] <30 mL/min/1.73 m2

Morbid obesity

Obesity

History of hypoglycemia

History of pancreatitis

14. Which symptoms of GERD would be a cause for alarm and further work-up
of the patient?

Belching

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