This comprehensive HESI Pathophysiology Exit Exam Complete Questions Bank provides an extensive collection of high-yield pathophysiology practice questions with detailed answer rationales covering essential concepts across all major body systems and disease processes. The document is meticulously structured to facilitate critical thinking, clinical judgment, and prioritization skills development for nursing students preparing for the HESI exit exam, NCLEX, and other comprehensive nursing assessments. This question bank includes all versions of the actual exam, emphasizing the integration of pathophysiologic mechanisms with clinical manifestations, diagnostic findings, and evidence-based nursing interventions.
Key Content Areas Covered:
1. Foundational Pathophysiology Concepts
Positive feedback as an initial disturbance in a system that sets off a chain of events not favoring stability and abruptly displaces a system from its steady state
Negative feedback, disease, and homeostasis as related concepts
Homeostasis as a stable coordinated physiologic internal environment achieved through processes that oppose change
Allostasis as achieving stability through physiologic change
General adaptation syndrome (GAS) and stress response
2. Aging and Developmental Pathophysiology
Organ shrinkage with age including prostate, gums, and heart
Slower heart rate in infants compared to adults
Decreased temperature sensation in the elderly
Decreased sweat glands in the elderly
Decreased thirst perception in the elderly
Women having lower hemoglobin than men
Men having higher calcium levels than women
Women snoring less than men
Neonatal period as the first two months of life
Infancy, childhood, and prenatal periods
Acclimatization as an increase in normal adaptive response to decreased oxygen availability at high altitude
Circadian rhythms and biological clocks
3. Disease Causation and Terminology
Iatrogenic disease as unwanted medical treatment
Idiopathic, ecogenetic, and genotypical causes of disease
Sign as an objectively identifiable aberration of disease
Symptom as a subjective experience of disease
Syndrome as a collection of signs and symptoms
Stage as the extent of disease progression
Acute and chronic disease classifications
Remission and exacerbation
Complications and sequelae
4. Cellular Physiology and Pathophysiology
Lipid molecules crossing cell membranes by diffusion
Channel proteins, endocytosis, exocytosis, and voltage-gated ion channels as membrane transport mechanisms
Protein synthesis as a component of anabolic metabolism
Glycolysis, citric acid cycle, and oxidative phosphorylation as catabolic processes
Sodium-potassium pump as the electrochemical mechanism responsible for maintaining cell volume
Cellular hypoxia resulting in failure of the sodium-potassium pump
Enhanced ATP activity, loss of intracellular calcium, and cellular swelling in hypoxia
Atrophy of leg muscles after prolonged bed rest
Dysplasia, hypertrophy, and hyperplasia of leg muscles
Atrophy as the most likely cellular adaptation from chronic ischemia
Metaplasia, hypertrophy, and hyperplasia as cellular adaptations
Decompression sickness (the bends) caused by gas bubbles in the blood blocking circulation and resulting in ischemia
Free radicals forming abnormal chemical bonds destroying cellular membranes
Carbon monoxide binding tightly to hemoglobin preventing oxygen carrying
Intracellular calcium accumulation creating muscle tetany
Dysplasia as the cellular adaptation creating the greatest concern on a biopsy report
Anaplasia, proto-oncogenes, and suppressor genes
Apoptosis as normal cell suicide
Atrophy, hypertrophy, hyperplasia, metaplasia, dysplasia, and neoplasia
5. Genetics and Genetic Disorders
Transcription as the process by which mRNA is synthesized from single-stranded DNA
Sense stranding, replication, and translation
Epithelium, connective tissue, hematopoietic tissue, and nervous tissue as the four cell types found in adult humans
Recessive trait genotype as aa
Homozygous dominant (AA), heterozygous (Aa), and homozygous recessive (aa) genotypes
Chromosomal abnormalities linked with Down syndrome including translocation
Inversion, deletion, and duplication
Hemophilia as a deficiency of factor VIII causing a bleeding disorder
Marfan syndrome, phenylketonuria, and Huntington disease
Retroviruses causing cancer including Epstein-Barr virus
Rhinovirus, parvovirus, and poxvirus
BRCA1 gene contributing specifically to some breast cancer
Rb, P53, and VHL genes
Autosomal dominant, autosomal recessive, and X-linked recessive inheritance patterns
Genetic counseling and testing considerations
6. Oncologic Pathophysiology
Grading as used to describe the histologic characteristics of tumor cells
Staging, cachexia, and angiogenesis
Early detection offering the best prognosis for cure of cancer
Aggressive chemotherapy and radiation therapy
Combined 5-year survival rate for cancer being 62%
Most cancer deaths occurring in those over 75 years old
Men having 1 in 3 lifetime risk of cancer
Cancer as the leading cause of death
TNM classification system tracking the progress of disease and effectiveness of treatment
Cancer cachexia caused by cancer cells producing an enzyme that robs normal cells of nutrients
Lack of adequate normal cells to support metabolism
Increased basal metabolic rate
Cancer cell destruction utilizing most of body's nutrients
Loss or defect of tumor suppressor genes explaining genetic basis for cancer development
Exposure to ionizing radiation
Decreased immune function
Infection by oncogenic bacteria
Benign versus malignant tumors
Tumor markers including PSA, CA-125, and CEA
Chemotherapy, radiation therapy, and biotherapy
Bone marrow transplantation and stem cell therapy
7. Immunologic Pathophysiology
Bacteria categorized by shape, need for oxygen, and reaction to staining
Size and color as bacterial classification
Opportunistic infection when host's immune system is compromised and resident flora becomes pathogenic
Salmonella infection, otitis media, and myocarditis
Patient education to reduce emergence of resistant infections including taking all antibiotics ordered even when feeling recovered
Stopping antibiotics when fever is gone, taking antibiotics for viral infections, and sharing antibiotics as inappropriate practices
Vessel walls briefly constricting after cellular injury
Active immunity including pneumonia vaccine
Injection of pooled gamma globulin, breast-feeding, and placental transfer as passive immunity
Leukemia, weakness, infection, and immunosuppression
Elevated eosinophils in blood indicating allergies
Bacterial infection, viral infection, and sinus infection
Serous drainage from a ruptured vesicle
Lymphocytes associated with the immune response
Neutrophils, monocytes, and macrophages
Anaphylaxis as a severe life-threatening type 1 hypersensitivity reaction
Angioedema, urticaria, and hives
Systemic lupus erythematosus (SLE) underlying pathophysiologic abnormality as autoimmunity
Immune deficiency, type 2 hypersensitivity, and immunosuppression
Petechiae, easy bruising, bleeding gums, occult hematuria, or retinal hemorrhage as thrombocytopenia
Neutropenia, leukopenia, and anemia
Allogenic transplant from a sister
Autologous transplant and peripheral stem cell transplant
Multiple myeloma as a malignant disorder of mature antibody-secreting B lymphocytes or plasma cells
Hairy cell leukemia, ALL, and CML
Reed-Sternberg cells in Hodgkin lymphoma
B-cell lymphoma, NK-cell lymphoma, and T-cell lymphoma
Bence Jones proteins assisting in diagnosis of multiple myeloma
Elevated liver enzymes, hyperkalemia, and erythrocytosis
HIV-1 as a causative agent in the US
HIV transmission via breast milk and sharing needles
Aerosol route, pregnancy, saliva, and surface survival
Initial HIV infection with flulike symptoms and rash
Decreased CD4 T cells, seroconversion within 3-6 days, and CD4 T cell count 200 per mcg/L
HIV rapid finger stick tests giving results in 20 minutes
Electrophoresis, ELISA, and Western blot
Anergic response because of immune system damage in HIV patients with non-reactive TB skin test
Increased T cells suppressed response
Sputum testing accuracy with HIV
8. Hematologic Pathophysiology
Erythropoiesis requiring vitamin B12, folate, and ascorbic acid (Vitamin C)
Vitamin K, iron, and erythropoietin
Shift of oxyhemoglobin curve to the right caused by chronic disease
Increased pH, decreased H+ ions, and decreased temperature
Polycythemia as an excess of RBCs
Anemia, pancytopenia, and granulocytosis
Aplastic anemia running in families
Overproduction of hematopoietic tissue
Pancytopenia and viral infections
Macrocytic anemia including pernicious anemia
Thalassemia, anemia of chronic renal failure, and iron deficiency anemia
Sickle cell anemia management including acetaminophen for fever
Early spleen removal
Anemia of chronic disease caused by lack of erythropoietin
Estrogen, parathyroid hormone, and adrenaline
Signs and symptoms of anemia including pallor, shortness of breath, and reduced activity tolerance
Bradycardia and dry mucous membranes
Platelets contributing to hemostasis by clumping
Vasoconstriction, fibrinolysis stimulation, and intrinsic pathway
Petechiae in vascular and platelet disorders being flat, pinpoint, and non-blanching
Red, raised, and macular
Flat and purple with vesicles
Raised, red, and blanching
Allergic purpura lesions including fever, itching, arthralgias, very palpable lesions, and bleeding from lesions
Lesions located mainly on the trunk and face
Disseminated intravascular coagulation (DIC) as both a bleeding and clotting disorder
Acute form with more thrombotic episodes
Fibrinogen level increased and fibrin split products decreased
Lymphatic system arrangement similar to circulatory system
Reabsorbing fluid that leaks out of the lymphatic system
Lymphatic vessels being thin-walled with valves
Lymphatic tissue and lymph nodes found deep within muscles and connective tissue
Arterial thrombosis of the legs
Varicose veins, atherosclerosis, cerebral aneurysm, and cerebral AV malformation as causes of stroke signs and symptoms
9. Cardiovascular System Pathophysiology
Increased hydrostatic pressure in blood vessels resulting in fluid movement into interstitial space
Increased levels of albumin, vasoconstriction, and hypertension
Left heart failure manifestations including pulmonary edema and paroxysmal nocturnal dyspnea (PND)
Peripheral edema and increased exercise tolerance
Compartment syndrome manifestations including pain, paresthesia, ischemia, inflammation, coolness, and edema
Increased intracranial pressure (ICP) manifestations including projectile vomiting, altered mental status, and decerebrate position
Intense thirst
Unusual thirst in diabetic patient indicating hyperglycemia
Nephropathy, polyuria, and fluid overload
Chronic airway disease including COPD characterized by progressive increase in ciliary function
Asthmatic attacks producing edema and increased mucus
COPD producing reversible constriction of airways
Asthma resulting in hyperinflation of alveoli
Peripheral artery disease with intermittent pain with walking caused by arteriole occlusion resulting in ischemia to muscles
Initial vital sign alteration in acute hypovolemia as tachycardia
Tachypnea, hypotension, and elevated temperature
Stable angina with chest pain, shortness of breath with activity, and relief by rest
Unstable angina, myocardial infarction, and pericarditis
Risk factors for gastric cancer including eating bacon and other cured meats and Helicobacter pylori infection
Celiac disease, ulcerative colitis, and high carbohydrate diet
Immunologic activity in COPD including macrophages causing destruction of alveolar walls resulting in loss of overall alveolar surface area
T cells attacking healthy goblet cells
B cells producing excessive immunoglobulin
Antigen-antibody complexes deposited in small airways
Protein restriction for patients with liver and kidney disease because protein breakdown produces nitrogenous wastes that are excreted by liver and kidneys
Protein having proinflammatory effect on liver tissue
Protein excreted by damaged nephrons resulting in proteinuria
Less protein needed as organ dysfunction results in reduced metabolic needs
Mechanism of end-organ disease with diabetes mellitus including hyperglycemia resulting in microvascular damage to capillaries
Episodic hypoglycemia causing intermittent reduction of blood flow to organs
Buildup of nitrogenous wastes in blood being toxic to organs
Reduction in blood viscosity resulting in enhanced perfusion of organs
Diagnosis of pancreatitis with elevated serum lipase and amylase levels
Hard, dry stools, right lower quadrant pain, and thrombocytopenia
Elevated plasma glucose causing release of insulin
Release of glucagon, glycogen, and potassium
Normal lab values including LDL of 100 mg/dL, potassium of 3.5-5.0 mEq/L, hemoglobin of 12-17 gm/dL, HDL 40 mg/dL, and BUN of 15 mg/dL
Hematocrit 30% as abnormal
Open-angle glaucoma with trabecular meshwork becoming blocked over time
Angle of Schlemm constricted preventing drainage
Sudden and acutely painful presentation
Adult respiratory distress syndrome (ARDS) expected feature as hypoxia
Extreme hypercapnia, metabolic alkalosis, and green or yellow sputum
Chronic lymphocytic leukemia (CLL) may be suppressed but not likely cured
Acute lymphocytic leukemia (ALL), Hodgkin lymphoma, and acute myelogenous leukemia (AML)
Benign prostatic hypertrophy (BPH) causing urinary symptoms by compression of urethra causing difficulty voiding
Inflammatory changes in bladder
Metastasis of tumor cells into urinary tract
Formation of calculi resulting in urinary pain
Acute renal failure expected finding as increased serum potassium
Decreased specific gravity of urine
Reduced blood urea nitrogen (BUN)
Normal blood creatinine level
Mechanism of extreme edema after a burn
Decubitus ulcers from prolonged pressure on tissues causing inflammation and ischemia
Eczema, psoriasis, and abrasions
HIV patient with non-reactive TB skin test but active disease by sputum testing due to anergic response because of immune system damage
Presence of HIV virus interfering with skin reaction
Increase in T cells suppressed response
Sputum testing not accurate with HIV
Travel to a country with possibly contaminated water and expected infections including hepatitis A and acute amoebic dysentery
Hepatitis B, ulcerative colitis, and gluten enteropathy
Osteoporosis resulting in increased bone reabsorption and demineralization
Rheumatoid arthritis resulting in antigen-antibody deposition and inflammation of joint
Acute gout resulting in repetitive stress and destruction of articular cartilage
Osteoarthritis always affecting joints symmetrically and causing fevers and fatigue
Foam cells (fatty macrophages) associated with development of atherosclerosis
Pluripotent stem cells, reticulocytes, and myocytes
Emergency infusion of factor VII for patient with von Willebrand's disease having surgery
Patient with gunshot wound to chest
Patient with pernicious anemia
Patient with polycythemia vera
Appropriate candidate for transfusion of whole blood as trauma patient who is hemorrhaging
Elderly woman with iron deficiency anemia
Septic patient with disseminated intravascular coagulopathy (DIC)
Patient with hemophilia A getting routine infusion
Amyotrophic lateral sclerosis (ALS) expected progression as destruction of motor neurons in brain and spinal cord causing progressive muscle weakness
Dysfunction of substantia nigra in the brain causing reduction in dopamine and development of progressive movement disorder
Acute inflammation of meninges causing nuchal rigidity, pain, headache, and high fever
Loss of cholinergic receptors in muscles leading to early muscle fatigue and weakness
Abdominal aortic aneurysm (AAA) rupture causing hypotension and shock
Sudden loss of circulation to one leg
Acute respiratory distress
Proteinuria and edema
Risk factors for osteoporosis including low BMI, being Caucasian, and smoking
Repetitive stress to joint and being overweight
Hormone released in stressful situation causing alteration in glucose metabolism as cortisol
Epinephrine, renin, and erythropoietin
Difference between diabetic ketonuria (DKA) and hyperosmolar hyperglycemic state (HHS) including DKA patients having ketones in urine and blood while HHS patients do not
DKA occurring with diabetics and HHS occurring in anyone
DKA characterized by hyperglycemia and HHS characterized by hypoglycemia
DKA being very dangerous and HHS being less serious
Exophthalmos caused by increased inflammation of eyes affecting muscles and fatty tissue around eye
Decreased cardiac output resulting in activation of RAAS system and increased blood pressure
Increased pressure in anterior chamber of eye causing hardening of eyeball and blurring of vision
Inflammation of cornea causing intense pressure and pain in eye
Stress dosing of steroids for Addison's disease
Cushing's disease, acromegaly, and hypothyroidism
Early infections (shift to the left) with bandemia
Increased B cells, lymphocytosis, and thrombocytopenia
Frank-Starling law predicting more preload, increased contraction
More afterload, less perfusion
Less afterload, more ischemia
More contraction, less output
Function of glomerulus as filter blood
Activating vitamin D, actively reabsorbing glucose, and regulating sodium excretion
Neurotransmitter changes in brain associated with mood depression including low serotonin
High dopamine, increased acetylcholine (ACh), and low norepinephrine
HIV transmission via breast milk and sharing needles
Aerosol route, pregnancy, saliva, and surface survival
Initial HIV infection with flulike symptoms and rash
Decreased CD4 T cells, seroconversion within 3-6 days, and CD4 T cell count 200 per mcg/L
HIV rapid finger stick tests giving results in 20 minutes
Electrophoresis, ELISA, and Western blot
Erythropoiesis requiring vitamin B12, folate, and ascorbic acid (Vitamin C)
Vitamin K, iron, and erythropoietin
Shift of oxyhemoglobin curve to the right caused by chronic disease
Increased pH, decreased H+ ions, and decreased temperature
Polycythemia as an excess of RBCs
Anemia, pancytopenia, and granulocytosis
Aplastic anemia running in families
Overproduction of hematopoietic tissue
Pancytopenia and viral infections
Macrocytic anemia including pernicious anemia
Thalassemia, anemia of chronic renal failure, and iron deficiency anemia
Sickle cell anemia management including acetaminophen for fever
Early spleen removal
Anemia of chronic disease caused by lack of erythropoietin
Estrogen, parathyroid hormone, and adrenaline
Signs and symptoms of anemia including pallor, shortness of breath, and reduced activity tolerance
Bradycardia and dry mucous membranes
Platelets contributing to hemostasis by clumping
Vasoconstriction, fibrinolysis stimulation, and intrinsic pathway
Petechiae in vascular and platelet disorders being flat, pinpoint, and non-blanching
Red, raised, and macular
Flat and purple with vesicles
Raised, red, and blanching
Allergic purpura lesions including fever, itching, arthralgias, very palpable lesions, and bleeding from lesions
Lesions located mainly on the trunk and face
Disseminated intravascular coagulation (DIC) as both a bleeding and clotting disorder
Acute form with more thrombotic episodes
Fibrinogen level increased and fibrin split products decreased
Lymphatic system arrangement similar to circulatory system
Reabsorbing fluid that leaks out of the lymphatic system
Lymphatic vessels being thin-walled with valves
Lymphatic tissue and lymph nodes found deep within muscles and connective tissue
Arterial thrombosis of the legs
Varicose veins, atherosclerosis, cerebral aneurysm, and cerebral AV malformation as causes of stroke signs and symptoms
Upper acceptable level of cholesterol in blood including total cholesterol of 200 mg/dL, HDL of 40 mg/dL, and LDL of 100 mg/dL
Total cholesterol of 40 mg/dL, HDL of 200 mg/dL, and LDL of 160 mg/dL
Total cholesterol of 40 mg/dL, HDL of 160 mg/dL, and LDL of 200 mg/dL
Total cholesterol of 200 mg/dL, HDL of 140 mg/dL, and LDL of 60 mg/dL
Modifiable risk factor for prevention of atherosclerosis as smoking
Age as non-modifiable risk factor
Major determinant of diastolic blood pressure as systemic vascular resistance (SVR)
Heart rate (HR), stroke volume (SV), and preload
Renin release not stimulated by parasympathetic activation
Renal hypoperfusion, fight or flight response, and decreased Na+ delivery
Another name for vasopressin as antidiuretic hormone
Arterial natriuretic peptide, angiotensin, and aldosterone
Causes of increased blood pressure including intracerebral hemorrhage
Dehydration, blood loss, and prolonged bed rest
Endocrine disorder causing elevated BP as coarctation of the aorta
Renin-angiotensin system (RAS) altering blood pressure in response to decreased perfusion of the kidney
Decreased flow to the brain, arm, and liver
Arrangement in which separate cells of the myocardium can function together as syncytium
Autoregulation, ischemia, and myocyte
Electrolytes necessary for muscle contraction including sodium, calcium, and potassium
Magnesium and selenium
Blood flow in the human body from the right atrium to the right ventricle to the lungs, back to the left atrium to the left ventricle to the body, then back through the venous system
Left side of heart to lungs to right side of heart
Right ventricle to right atrium to lungs
Cardiac muscle obtaining O2 and getting rid of wastes during diastole
Systole, ventricular contraction, and atrial repolarization
Myocardial infarction described by radiating chest pain, ST segment changes, and elevated troponin
Chest pain worse with cough and deep breath
Chest pain initiated by exercise or stress
Chest pain worsened with deep breathing but relieved by rest
Congenital heart disease producing cyanosis due to right-to-left shunting of blood
Left-to-right shunting of blood
Ventricular septal obstruction
Atherosclerotic plaques
Patient with heart failure awakening intermittently during night with shortness of breath described as paroxysmal dyspnea
Dyspnea, orthopnea, and cyanosis
Left heart failure symptoms not expected including pedal edema
Cough, dyspnea, and crackles in lungs
Hypovolemic shock compensatory mechanism preserving adequate circulation as renin-angiotensin-aldosterone cascade
Increased parasympathetic nervous system activity
Vasodilation and decreased secretion of antidiuretic hormone (ADH)
Shock not characterized by vasodilation and peripheral pooling of blood as cardiogenic
Septic shock
Underlying problem in all shock states as inadequate cellular oxygenation
Tachycardia, cardiac failure, and faulty compensatory mechanisms
Cells switching to anaerobic metabolism producing lactic acid
Excess energy, carbon monoxide, and bilirubin
Types of dead space including anatomic, alveolar, and physiologic
Cartilaginous as not a type of dead space
Compliance as a measure of how easily the lung can be inflated
How easily gas exchange occurs at the alveoli
How much dead space the lung has
Shunting
Conditions increasing lung compliance including emphysema
Pneumonia
Hypoventilation with PaCO2 exceeding 45 mm Hg
Amount of air entering alveoli increasing
Normal response to high altitude
Hypocapnia occurring
Major obstructive airway diseases including bronchitis, asthma, and emphysema
Pneumonia as not a major obstructive airway disease
Pathogenic changes associated with bronchitis including inflamed airways, swelling from exudation of fluid, and loss of ciliary function
Decreased mucous production as not associated with bronchitis
Most common cause of chronic bronchitis as smoking
Viral infection, bacterial infection, and air pollution
Emphysema in a 24-year-old male who has never smoked caused by alpha-1 antitrypsin deficiency
Value consistent with acute respiratory failure including pH 7.3
PaO2 of 100 mm Hg, PaCO2 of 40 mm Hg, and hypocapnia
Abnormality characteristic of emphysema as trapping of air in distal air sacs
Extensive inflammation of lower airways
Widespread occurrence of bronchial plugs
Collapse of proximal airways
Triad suggesting epiglottitis including sore throat, difficulty swallowing, and drooling
Hoarseness, sore throat, and productive cough
Persistent productive cough, vomiting, and fever
Fever, sore throat, and increased thirst
Tachycardia, decreased or absent breath sounds on affected side, hyper-resonance, and sudden chest pain on affected side as signs of pneumothorax
Myocardial infarction
Tubercular bacilli transmitted by airborne droplet nuclei
Blood and body fluids, contaminated food, and contaminated blood
Release of ADH from posterior pituitary stimulated when patient is hypovolemic or has hyper-osmolality of extracellular fluid
Patient being hypervolemic and hypertensive
Patient having hypo-osmolality of extracellular fluid and being lethargic
Patient being febrile and restless
Clinical signs of hypocalcemia versus hypercalcemia including hypercalcemia exhibiting decreased neuromuscular irritability such as lethargy, shortened ST segment, and diminished reflexes
Hypocalcemia exhibiting increased neuromuscular irritability such as headache, renal calculi, and fatigue
Hypocalcemia exhibiting increased neuromuscular irritability such as positive Trousseau sign, positive Chvostek sign, paresthesia, muscle twitching, and cramping
Hypercalcemia exhibiting decreased neuromuscular irritability such as seizures, lethargy, nausea, vomiting, diarrhea, dizziness, disorientation, and muscle twitching
Too much carbonic acid accumulating in blood causing respirations to increase and depth to increase
Respirations increasing and depth decreasing
Respirations decreasing and depth increasing
Respirations decreasing and depth decreasing
Kidneys responsible for maintaining fluid and electrolyte homeostasis
Kidneys filtering more than 70 ml of fluid an hour
Kidneys activating vitamin C
Kidneys containing 100,000 nephrons
Calcium in chronic renal failure with low serum levels
Increased absorption from GI tract
High serum calcium
Normal parathyroid activity
Predisposing factors for pyelonephritis including pregnancy, neurogenic bladder, catheterization, and urinary obstruction
Viral infection, sexual trauma, and sepsis
Diabetes, polycystic kidney disease, and cystoscopy
Irradiation, diabetes, and trauma
Effect of urinary obstruction on glomerular filtration, urinary stasis, and infection risk including decreased glomerular filtration, urinary stasis, and predisposition to infection
Decreased hydrostatic pressure, decreased filtration, and anuria
Increased glomerular filtration rate, diuresis, and infectious process
No effect on glomerular filtration, increased urinary stasis, and infection
Glomerulonephritis resulting from antigen-antibody complexes
Trauma to kidney, infection caused by staphylococci, and obstructive pathology
Risk factors for acute renal failure including atherosclerosis, hypertension, and diabetes mellitus
Youth as not a risk factor
Classic finding in renal failure as increased phosphorus
Crystals in urine, increased calcium, and protein in urine
Patient presenting with jugular venous distention, bounding pulses, rales in lungs, and peripheral edema in chronic renal failure caused by inability to excrete fluid
Azotemia, hypocalcemia, and hypertension
Nutrition not restricted in renal failure including carbohydrates
Protein, sodium, and fluid
Incontinence manifested by constant or intermittent dribbling as overflow incontinence
Urge incontinence, mixed incontinence, and stress incontinence
Predisposing factors for cystitis in elderly including benign prostatic hypertrophy, diabetes mellitus, and bowel incontinence
Obesity, chronic illness such as diabetes, and sexual relations
Lack of exercise, arthritis, and decreased estrogen in women
Urinary incontinence, neurologic illness, and thirst
Normal cells committing suicide as apoptosis
Anaplasia, proto-oncogene, and suppressor genes
Chronic stress expected findings including reduced productivity
Increased energy, reduced urine output, and heightened awareness
Conductive hearing loss caused by bug in ear
Removal of dry cerumen
Disease of cochlear system
Listening to iPod set at 10
Sensorineural hearing loss resulting from cochlear damage
Congenital absence of outer ear
Impacted cerumen in ears
Bean inserted into external auditory canal
Patient with diabetic ketonuria developing hypokalemia during treatment due to K+ following glucose into cells
Decrease in serum pH
Activation of active transport mechanism in kidneys
Increase in cardiac output