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NSG 120 Final Test Questions with Verified Correct Answers

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NSG 120 Final Test Questions with Verified Correct Answers

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NSG 120 Final Test Questions with Verified
Correct Answers
Pathogenesis

The development of the disease or the sequence of events involved in the tissue changes

related to the specific disease process.

Onset

sudden OR acute- An acute disease indicates a short-term illness that develops quickly with

marked signs such as high fever or severe pain—for example, acute appendicitis; onset may

be insidious, best described as a gradual progression with only vague or very mild signs.

Chronic

often a milder condition developing gradually, such as rheumatoid arthritis, but it persists for

a long time and usually causes more permanent tissue damage. Often a chronic disease is

marked by intermittent acute episodes.

Subclinical (stage/phase)

in which pathologic changes occur but the patient exhibits no obvious manifestations,

perhaps because of the great reserve capacity of some organs.

Latent stage

"silent" stage, in which no clinical signs are evident, characterizes some diseases. In

infectious diseases this stage may be referred to as the incubation period, which is the time

between exposure to the microorganism and the onset of signs or symptoms; it may last for a

day or so or may be prolonged, perhaps for days or weeks.

Disease transmission of an insect bite

,The injury occurs when the insect either bites or stings the patient. Venom is injected into the

tissue, resulting in the body's response to a foreign protein.

Rocky Mountain Spotted Fever/Tick Bite disease transmission

The causative agent, Rickettsia rickettsii, is transmitted by the wood tick and is carried in the

feces of infected ticks. It is introduced into the bloodstream of a person during a prolonged

tick bite.

Symptoms of Rocky Mountain Spotted Fever

Class triad: headache, fever, maculopapular rash (bumps and petechial rash)

high fever

Malaria/Mosquito bite disease transmission

Malaria is caused by four species of the protozoan genus Plasmodium, which is transmitted

from infected human to human by the bite of mosquito vectors or, less commonly, by blood

transfusion or IV drug use.

Disease transmission of animal/human bites

The bite occurs when the offending animal is agitated, frightened, threatened, or angry. Bites

can be from domestic animals, such as cats and dogs; farm animals; or wild animals, such as

skunks, bats, raccoons, and foxes. Human bites have been recorded.



The pattern of the teeth marks is helpful in determining the type of bite, human or animal,

and the type of animal that did the biting if not immediately known.

Snake bite disease transmission

,Four kinds of poisonous snakes are known to inhabit the United States: rattlesnakes,

copperheads, water moccasins, and coral snakes.



Some bites are the work of nonpoisonous snakes, but all snakebites should be treated as

poisonous by the first aid provider.

Cardinal Signs of Inflammation

The cardinal signs of inflammation are redness (rubor or erythema), heat, swelling, and pain;

sometimes loss of function

Diangostic blood changes that may indicate inflammation?

*Leukocytosis (increased WBC)

*Differential count of WBC

*plasma protein (increased prothrombin and fibrinogen)

*CRP (appears with acute inflammation and necrosis)

*increased ESR

*cell enzymes present (may indicate necrosis)

Treatment of inflammation

*Drugs - Aspirin, NSAIDS, etc.

*RICE (rest, ice, compression, elevation)

*Compression stockings

*Moderate exercise

Opportunistic infection

, Microbes that are not usually pathogenic but can become pathogenic when transferred into

another location of the body, if the balance of the species is not maintained, or if the body's

defense is impaired.

Stages of Pressure Ulcers

Stage I: Intact skin with non-blanchable redness of a localized area usually over a bony

prominence. Darkly pigmented skin may not have visible blanching; its color may differ from

the surrounding area.



Stage II: Partial thickness loss of dermis presenting as a shallow open ulcer with a red pink

wound bed, without slough. May also present as an intact or open/ ruptured serum-filled

blister.



Stage III: Full thickness tissue loss. Subcutaneous fat may be visible but bone, tendon or

muscle are not exposed. Slough may be present but does not obscure the depth of tissue loss.

May include undermining and tunneling.



Stage IV: Full thickness tissue loss with exposed bone, tendon or muscle. Slough or eschar

may be present on some parts of the wound bed. Often include undermining and tunneling.

Deep Tissue Injury Purple or maroon localized area of discolored intact skin or blood-filled

blister due to damage of underlying soft tissue from pressure and/or shear. The area may be

preceded by tissue that is painful, firm, mushy, boggy, warmer or cooler as compared to

adjacent tissue.



Unstageable: Full thickness tissue loss in which the base of the ulcer is covered by slough

(yellow, tan, gray, green or brown) and/or eschar (tan, brown or black) in wound bed

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