QUESTIONS & ANSWERS | FETAL MONITORING, EFM &
C-EFM PRACTICE QUESTIONS WITH RATIONALES 2026/2027
90 Questions with Answers and Detailed Rationales
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AWHONN ADVANCED FETAL HEART MONITORING EXAM QUESTIONS & ANSWERS | FETAL
MONITORING, EFM & C-EFM PRACTICE QUESTIONS WITH RATIONALES 2026/2027. It contains 90 carefully
selected questions that reflect the most current exam content and testing strategies. Each question is
accompanied by a correct answer and a detailed rationale that explains the underlying pathophysiology,
pharmacology, or clinical reasoning.
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identify areas requiring further question format and content
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Review Summary 90 Questions
Foundations - Application - Awhonn Advanced Fetal Heart Monitoring & Fetal Monitoring EFM & C-efm
WITH Rationales 2026/2027 Fetal Heart Monitoring EFM & C-efm Graduate / Advanced
All answers with rationales
,Table of Contents
Content Area Questions Key Topics
Fundamentals OF Electronic 1-15 Fetal, Tracing, Baseline, Heart RATE, Decelerations
Fetal Monitoring
Fetal Heart RATE Pattern 16-30 Fetal, Tracing, Heart RATE, Variability, Baseline
Interpretation
Uterine Contraction 31-45 Fetal, Tracing, Heart RATE, Variability, Baseline
Assessment
Oxygenation AND Acid-base 46-60 Fetal, Decelerations, Variability, Heart RATE, Baseline
Balance
Category I II AND III Tracing 61-75 Tracing, Fetal, Variability, Weeks, Heart RATE
Management
Antepartum AND Intrapartum 76-90 Fetal, Heart RATE, Tracing, Appropriate, Baseline
Fetal Surveillance
TOTAL 90 All questions include answers and detailed rationales
,Section A - Fundamentals OF Electronic Fetal Monitoring
Q1.
A patient at 39 weeks gestation has a Category I tracing. The nurse notes a decrease in
baseline variability from moderate to minimal over 30 minutes, followed by a single late
deceleration. Which pathophysiologic mechanism best explains this sequence?
A. Acute cord compression leading to B. Progressive fetal hypoxemia causing
reflexive peripheral vasoconstriction chemoreceptor-mediated autonomic
dysfunction
C. Maternal position change causing D. Fetal sleep cycle transitioning to a quiet
aortocaval compression and decreased state with reduced metabolic demand
preload
Correct: B - Progressive fetal hypoxemia causing chemoreceptor-mediated autonomic
dysfunction
Rationale:Loss of variability followed by late decelerations indicates progressive fetal
hypoxemia and metabolic acidemia. The chemoreceptor response to hypoxia initially causes
increased variability, but as acidemia worsens, autonomic dysfunction leads to decreased
variability. Late decelerations result from myocardial depression due to hypoxemia.
Why the other answers are wrong:
A. Cord compression typically causes variable decelerations, not late decelerations, and does
not explain the loss of variability.
C. Aortocaval compression would cause a sudden decrease in baseline, not a gradual loss of
variability and late decelerations.
D. Fetal sleep cycles cause decreased variability but not late decelerations.
Reference: AWHONN, Fetal Heart Monitoring, 5th Ed., Ch. 6
Q2.
A patient at 34 weeks gestation with preterm premature rupture of membranes develops a
fever of 38.9°C and a fetal heart rate of 180 bpm with moderate variability and no
decelerations. Which intervention is most consistent with current evidence-based
management?
A. Administer antipyretics and initiate B. Perform an amnioinfusion to reduce cord
broad-spectrum antibiotics compression
C. Administer terbutaline for fetal D. Immediate cesarean birth without delay
tachycardia
Correct: A - Administer antipyretics and initiate broad-spectrum antibiotics
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, Section A - Fundamentals OF Electronic Fetal Monitoring
Rationale: Maternal fever and fetal tachycardia suggest chorioamnionitis. Evidence supports
antibiotic therapy and antipyretics to reduce maternal fever and associated fetal tachycardia.
Immediate birth may be indicated if acidemia is suspected, but with moderate variability and
no decelerations, a non-acidotic fetus is likely, and antibiotics can be initiated while
evaluating.
Why the other answers are wrong:
B. Amnioinfusion is not indicated for fever or tachycardia; it is used for variable decelerations
due to cord compression.
C. Terbutaline is a tocolytic, not appropriate for fetal tachycardia; it could mask signs of
acidemia.
D. Immediate cesarean is not indicated without evidence of fetal acidemia; the tracing is
reassuring.
Reference: ACOG Practice Bulletin No. 116, Management of Intrapartum Fetal Heart Rate Tracings
Q3.
Which of the following best describes the primary mechanism by which intermittent
auscultation (IA) identifies fetal compromise compared to continuous electronic fetal
monitoring (EFM)?
A. IA detects early signs of hypoxemia B. IA evaluates fetal heart rate response to
through baseline variability assessment contractions at scheduled intervals
C. IA measures fetal oxygen saturation D. IA provides continuous beat-to-beat
noninvasively variability data
Correct: B - IA evaluates fetal heart rate response to contractions at scheduled intervals
Rationale:IA is performed at regular intervals (e.g., every 15-30 minutes in active labor) and
assesses FHR rate, rhythm, and response to contractions. It does not provide variability or
detailed pattern analysis. It can detect bradycardia or tachycardia but lacks the specificity of
continuous EFM for detecting subtle decelerations.
Why the other answers are wrong:
A. IA cannot assess variability because it does not provide a continuous tracing.
C. Fetal oxygen saturation monitoring is a different technology (FSpO2), not IA.
D. IA does not provide continuous data; it is intermittent.
Reference: AWHONN, Fetal Heart Monitoring, 5th Ed., Ch. 4
Q4.
A patient in active labor has recurrent variable decelerations with loss of variability and a
baseline of 160 bpm. An amnioinfusion is initiated. Which finding indicates the
amnioinfusion is most effective?
A. Decreased frequency of decelerations B. Increased baseline variability and
and improved variability increased number of accelerations
Page 4