NUR 207 Exam 4: Critical Care & Med-
Surg exam (2026/2027) 70+ Actual
Questions & Answers | Comprehensive
Study Guide – WVJC
Ace your upcoming Critical Care & Med-Surg exam with this premium, student-verified study
guide! This comprehensive resource includes 70+ actual, high-yield questions and
detailed rationales pulled directly from recent NUR 207 Test 4 assessments.
Whether you are cramming for a 12-lead EKG analysis or reviewing multisystem trauma, this
targeted Q&A bank will help you master core concepts and identify the exact distractors and
correct answers used by professors.
What’s Included:
• Real Exam Questions: Over 70 frequently repeated multiple-choice questions from
actual NUR 207 Exam 4 tests.
• Detailed Rationales: Not just "A, B, or C"—you get step-by-step explanations for why
the correct answer is right and why the others are wrong.
• Format: Clean, easily readable, and formatted as a print-ready PDF for quick
memorization and review.
Key Topics Covered:
• Advanced Cardiac Diagnostics: 12-lead EKG interpretation, ST segment
elevation/depression in myocardial infarctions (MI), and cardiac enzymes.
• Heart Failure Management: Right-sided vs. Left-sided heart failure presentations,
pulmonary edema, and pharmacology (e.g., Morphine, Oxygen, Nitroglycerin, Aspirin).
• Pulmonary Emergencies: Tension pneumothorax, pulmonary embolisms, chest tube
management, and interventions for respiratory distress.
• Critical Care & Hemodynamics: Primary/secondary surveys for trauma, shock, and
basic life support resuscitation protocols.
Why Choose This Study Guide?
• Save Time: Cut down your study hours by focusing on the exact concepts most likely
to appear on your test.
1
, • Boost Confidence: Practice with questions designed to mirror the cognitive level
required by your nursing program.
• Zero Guesswork: Clear, verified answer keys eliminate the frustration of searching for
correct textbook rationales on your own.
Perfect for last-minute review sessions, building, and passing your critical care exams with an A!
Download your copy today and secure your grade.
Q1. Which tasks are part of nursing care for a patient with Parkinson's disease?
[Multiple Choice]
A) Prescribing entacapone and adjusting levodopa dosing without provider input
B) Performing DaTscan imaging and interpreting MRI results independently
C) Conducting CSF antibody analysis to confirm diagnosis
D) Medication administration education, optimizing mobility, assisting with ADLs,
supporting bowel elimination and nutrition, and promoting effective communication
Answer: Medication administration education, optimizing mobility, assisting with ADLs,
supporting bowel elimination and nutrition, and promoting effective communication
Explanation: Nursing care centers on education about medications, helping patients maintain
mobility and independence with activities of daily living, managing bowel and nutrition needs,
and supporting communication. Imaging interpretation and diagnostic procedures are performed
by providers/radiologists, not by nurses alone. Medication prescription and dose changes are
provider responsibilities. CSF lab analysis is a diagnostic test, not a nursing intervention.
Q2. Which features are associated with rigidity in Parkinson's disease? [Multiple
Choice]
A) White matter plaques and increased CSF antibodies
B) Peripheral nerve sensory loss and areflexia
C) Flaccid weakness and positive Babinski sign
D) Cogwheel phenomenon (jerky movements) and a stooped posture
Answer: Cogwheel phenomenon (jerky movements) and a stooped posture
Explanation: Parkinsonian rigidity produces a steady resistance to passive movement that can
appear as a cogwheel (ratchety) quality and often accompanies a stooped posture. Flaccid
weakness and a positive Babinski reflect lower and upper motor neuron pathologies respectively
and are not features of Parkinsonian rigidity. Peripheral sensory loss and absent reflexes point to
2
, peripheral neuropathy rather than central rigidity. White matter plaques and CSF antibody
increases are findings in multiple sclerosis, not rigidity in Parkinson's.
Q3. According to the source, which factors increase risk for Parkinson's disease?
[Multiple Choice]
A) Directly caused by acute viral infection in all cases
B) Average age of onset 60 years, men 50% more likely than women, and about 25%
inherited
C) Typical onset in childhood, women more affected than men, and 100% inherited
D) Associated primarily with increased CSF antibodies and white matter plaques
Answer: Average age of onset 60 years, men 50% more likely than women, and about
25% inherited
Explanation: The source lists risk factors as average onset around 60 years, higher incidence in
men (about 50% more), and a portion (~25%) with hereditary influence. Childhood onset and
female predominance are incorrect for typical Parkinson's. Increased CSF antibodies and white
matter plaques are features of multiple sclerosis, not Parkinson's. Parkinson's is not described as
directly caused by an acute viral infection in every case.
Q4. What does deep brain stimulation (DBS) involve? [Multiple Choice]
A) Injection of loflupane I-123 to image dopamine transporters
B) Implanting a neurostimulator that sends electrical signals to areas of the brain
C) Removal of the substantia nigra to halt disease progression
D) Systemic administration of MAO-B inhibitors to increase dopamine
Answer: Implanting a neurostimulator that sends electrical signals to areas of the brain
Explanation: Deep brain stimulation involves surgical implantation of a neurostimulator that
delivers electrical impulses to targeted brain regions to modulate abnormal activity. MAO-B
inhibitors are pharmacologic, not a neurosurgical stimulation. Removing the substantia nigra is
not a treatment and would cause severe deficits. Injecting loflupane I-123 refers to a DaTscan
imaging procedure used in diagnosis, not a therapeutic electrical stimulation.
Q5. Which description best matches relapsing-remitting multiple sclerosis
(RRMS)? [Multiple Choice]
A) Attacks lasting days to months with symptom-free periods that can last months or
years and often increasing disability with each relapse
B) Always becomes constant without full remission immediately after the first attack
3
, C) Progressive worsening from the start with no flare-ups
D) Steady worsening from the start with no relapses or remissions
Answer: Attacks lasting days to months with symptom-free periods that can last months
or years and often increasing disability with each relapse
Explanation: RRMS is defined by discrete attacks (days to months) followed by remission periods
that can last months or years; disability tends to accumulate with repeated relapses. Steady
deterioration from onset without relapses describes primary progressive MS, not RRMS.
Becoming constant without remission after the first attack more closely resembles transition to
secondary progressive MS but is not the defining RRMS pattern. Progressive worsening without
flare-ups is primary progressive or other progressive forms, not relapsing-remitting.
Q6. What elements of nervous tissue are damaged in multiple sclerosis (MS)?
[Multiple Choice]
A) Peripheral motor endplates and muscle fibers exclusively
B) Dopamine-producing neurons in the substantia nigra only
C) Vascular endothelium causing repeated ischemic strokes
D) Myelin, nerve fibers, and the specialized cells that make myelin, leading to scar
tissue in the CNS
Answer: Myelin, nerve fibers, and the specialized cells that make myelin, leading to scar
tissue in the CNS
Explanation: MS is characterized by immune-mediated damage to CNS myelin, the underlying
nerve fibers, and oligodendrocytes (cells that produce myelin), which leads to scarred areas
(sclerosis). Damage limited to substantia nigra dopamine neurons describes Parkinson's, not MS.
Peripheral motor endplates and muscle fibers are outside the CNS and not the primary targets in
MS. Vascular endothelial injury and ischemic strokes are a separate vascular pathology, not the
autoimmune demyelination seen in MS.
Q7. Which statement correctly matches the listed Parkinson's medications with
their actions? [Multiple Choice]
A) Amantadine blocks dopamine breakdown; entacapone mimics dopamine;
pramipexole increases dopamine release; anticholinergics increase acetylcholine activity
B) All listed drugs only work by restoring levodopa levels and have the same mechanism
C) Amantadine is a COMT inhibitor; entacapone increases dopamine release;
pramipexole blocks MAO-B; anticholinergics block dopamine receptors
4
Surg exam (2026/2027) 70+ Actual
Questions & Answers | Comprehensive
Study Guide – WVJC
Ace your upcoming Critical Care & Med-Surg exam with this premium, student-verified study
guide! This comprehensive resource includes 70+ actual, high-yield questions and
detailed rationales pulled directly from recent NUR 207 Test 4 assessments.
Whether you are cramming for a 12-lead EKG analysis or reviewing multisystem trauma, this
targeted Q&A bank will help you master core concepts and identify the exact distractors and
correct answers used by professors.
What’s Included:
• Real Exam Questions: Over 70 frequently repeated multiple-choice questions from
actual NUR 207 Exam 4 tests.
• Detailed Rationales: Not just "A, B, or C"—you get step-by-step explanations for why
the correct answer is right and why the others are wrong.
• Format: Clean, easily readable, and formatted as a print-ready PDF for quick
memorization and review.
Key Topics Covered:
• Advanced Cardiac Diagnostics: 12-lead EKG interpretation, ST segment
elevation/depression in myocardial infarctions (MI), and cardiac enzymes.
• Heart Failure Management: Right-sided vs. Left-sided heart failure presentations,
pulmonary edema, and pharmacology (e.g., Morphine, Oxygen, Nitroglycerin, Aspirin).
• Pulmonary Emergencies: Tension pneumothorax, pulmonary embolisms, chest tube
management, and interventions for respiratory distress.
• Critical Care & Hemodynamics: Primary/secondary surveys for trauma, shock, and
basic life support resuscitation protocols.
Why Choose This Study Guide?
• Save Time: Cut down your study hours by focusing on the exact concepts most likely
to appear on your test.
1
, • Boost Confidence: Practice with questions designed to mirror the cognitive level
required by your nursing program.
• Zero Guesswork: Clear, verified answer keys eliminate the frustration of searching for
correct textbook rationales on your own.
Perfect for last-minute review sessions, building, and passing your critical care exams with an A!
Download your copy today and secure your grade.
Q1. Which tasks are part of nursing care for a patient with Parkinson's disease?
[Multiple Choice]
A) Prescribing entacapone and adjusting levodopa dosing without provider input
B) Performing DaTscan imaging and interpreting MRI results independently
C) Conducting CSF antibody analysis to confirm diagnosis
D) Medication administration education, optimizing mobility, assisting with ADLs,
supporting bowel elimination and nutrition, and promoting effective communication
Answer: Medication administration education, optimizing mobility, assisting with ADLs,
supporting bowel elimination and nutrition, and promoting effective communication
Explanation: Nursing care centers on education about medications, helping patients maintain
mobility and independence with activities of daily living, managing bowel and nutrition needs,
and supporting communication. Imaging interpretation and diagnostic procedures are performed
by providers/radiologists, not by nurses alone. Medication prescription and dose changes are
provider responsibilities. CSF lab analysis is a diagnostic test, not a nursing intervention.
Q2. Which features are associated with rigidity in Parkinson's disease? [Multiple
Choice]
A) White matter plaques and increased CSF antibodies
B) Peripheral nerve sensory loss and areflexia
C) Flaccid weakness and positive Babinski sign
D) Cogwheel phenomenon (jerky movements) and a stooped posture
Answer: Cogwheel phenomenon (jerky movements) and a stooped posture
Explanation: Parkinsonian rigidity produces a steady resistance to passive movement that can
appear as a cogwheel (ratchety) quality and often accompanies a stooped posture. Flaccid
weakness and a positive Babinski reflect lower and upper motor neuron pathologies respectively
and are not features of Parkinsonian rigidity. Peripheral sensory loss and absent reflexes point to
2
, peripheral neuropathy rather than central rigidity. White matter plaques and CSF antibody
increases are findings in multiple sclerosis, not rigidity in Parkinson's.
Q3. According to the source, which factors increase risk for Parkinson's disease?
[Multiple Choice]
A) Directly caused by acute viral infection in all cases
B) Average age of onset 60 years, men 50% more likely than women, and about 25%
inherited
C) Typical onset in childhood, women more affected than men, and 100% inherited
D) Associated primarily with increased CSF antibodies and white matter plaques
Answer: Average age of onset 60 years, men 50% more likely than women, and about
25% inherited
Explanation: The source lists risk factors as average onset around 60 years, higher incidence in
men (about 50% more), and a portion (~25%) with hereditary influence. Childhood onset and
female predominance are incorrect for typical Parkinson's. Increased CSF antibodies and white
matter plaques are features of multiple sclerosis, not Parkinson's. Parkinson's is not described as
directly caused by an acute viral infection in every case.
Q4. What does deep brain stimulation (DBS) involve? [Multiple Choice]
A) Injection of loflupane I-123 to image dopamine transporters
B) Implanting a neurostimulator that sends electrical signals to areas of the brain
C) Removal of the substantia nigra to halt disease progression
D) Systemic administration of MAO-B inhibitors to increase dopamine
Answer: Implanting a neurostimulator that sends electrical signals to areas of the brain
Explanation: Deep brain stimulation involves surgical implantation of a neurostimulator that
delivers electrical impulses to targeted brain regions to modulate abnormal activity. MAO-B
inhibitors are pharmacologic, not a neurosurgical stimulation. Removing the substantia nigra is
not a treatment and would cause severe deficits. Injecting loflupane I-123 refers to a DaTscan
imaging procedure used in diagnosis, not a therapeutic electrical stimulation.
Q5. Which description best matches relapsing-remitting multiple sclerosis
(RRMS)? [Multiple Choice]
A) Attacks lasting days to months with symptom-free periods that can last months or
years and often increasing disability with each relapse
B) Always becomes constant without full remission immediately after the first attack
3
, C) Progressive worsening from the start with no flare-ups
D) Steady worsening from the start with no relapses or remissions
Answer: Attacks lasting days to months with symptom-free periods that can last months
or years and often increasing disability with each relapse
Explanation: RRMS is defined by discrete attacks (days to months) followed by remission periods
that can last months or years; disability tends to accumulate with repeated relapses. Steady
deterioration from onset without relapses describes primary progressive MS, not RRMS.
Becoming constant without remission after the first attack more closely resembles transition to
secondary progressive MS but is not the defining RRMS pattern. Progressive worsening without
flare-ups is primary progressive or other progressive forms, not relapsing-remitting.
Q6. What elements of nervous tissue are damaged in multiple sclerosis (MS)?
[Multiple Choice]
A) Peripheral motor endplates and muscle fibers exclusively
B) Dopamine-producing neurons in the substantia nigra only
C) Vascular endothelium causing repeated ischemic strokes
D) Myelin, nerve fibers, and the specialized cells that make myelin, leading to scar
tissue in the CNS
Answer: Myelin, nerve fibers, and the specialized cells that make myelin, leading to scar
tissue in the CNS
Explanation: MS is characterized by immune-mediated damage to CNS myelin, the underlying
nerve fibers, and oligodendrocytes (cells that produce myelin), which leads to scarred areas
(sclerosis). Damage limited to substantia nigra dopamine neurons describes Parkinson's, not MS.
Peripheral motor endplates and muscle fibers are outside the CNS and not the primary targets in
MS. Vascular endothelial injury and ischemic strokes are a separate vascular pathology, not the
autoimmune demyelination seen in MS.
Q7. Which statement correctly matches the listed Parkinson's medications with
their actions? [Multiple Choice]
A) Amantadine blocks dopamine breakdown; entacapone mimics dopamine;
pramipexole increases dopamine release; anticholinergics increase acetylcholine activity
B) All listed drugs only work by restoring levodopa levels and have the same mechanism
C) Amantadine is a COMT inhibitor; entacapone increases dopamine release;
pramipexole blocks MAO-B; anticholinergics block dopamine receptors
4
