Stroke Pathophysiology
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Guide, Practice Questions,
Answers & ATI Review
Material
, Stroke Pathophysiology
Study online at https://quizlet.com/_2cng4b
1. Where is the most MCA
common site of an
ischemic stroke
2. An embolic stroke cardiac origin, carotid artery
usually has its ori-
gin where
3. Specific locations small vessel/lacunar arteries, large vessels (anterior and posterior systems affet-
for atherosclerotic ed)
plaques to build up
over time
4. Which type of Hemorrhagic d/t increased pressure
stroke is more dan-
gerous
5. Clinical pathology Cellular level events include: depletion of energy source, loss of ion pump,
of an ischemic glutamate release, CA excitotoxicity, apoptosis, neuroprotection.--> ischemic
stroke penumbra--> cerebral edema
6. Clinical patholo- ruptured vessel creates havoc on brain--> increased ICP, constrict cerebral flow,
gy of hemorrhagic increase brain tissue compression--> inflammatory response progresses CVA
stroke impairment bc of cerebral ischemia d/t hemorrhage (C reactive protein starts
inf. process)
7. Changes in brain 1. protease release (b/d blood brain barrier, central edema)
structure in an is- 2. Angiogenesis
chemic stroke 3. Immune response
8. Best imaging for CT is gold standard b/c it detects blood flow and perfusion.
an intracranial he-
morrhage
1/8
| Complete Exam Study
Guide, Practice Questions,
Answers & ATI Review
Material
, Stroke Pathophysiology
Study online at https://quizlet.com/_2cng4b
1. Where is the most MCA
common site of an
ischemic stroke
2. An embolic stroke cardiac origin, carotid artery
usually has its ori-
gin where
3. Specific locations small vessel/lacunar arteries, large vessels (anterior and posterior systems affet-
for atherosclerotic ed)
plaques to build up
over time
4. Which type of Hemorrhagic d/t increased pressure
stroke is more dan-
gerous
5. Clinical pathology Cellular level events include: depletion of energy source, loss of ion pump,
of an ischemic glutamate release, CA excitotoxicity, apoptosis, neuroprotection.--> ischemic
stroke penumbra--> cerebral edema
6. Clinical patholo- ruptured vessel creates havoc on brain--> increased ICP, constrict cerebral flow,
gy of hemorrhagic increase brain tissue compression--> inflammatory response progresses CVA
stroke impairment bc of cerebral ischemia d/t hemorrhage (C reactive protein starts
inf. process)
7. Changes in brain 1. protease release (b/d blood brain barrier, central edema)
structure in an is- 2. Angiogenesis
chemic stroke 3. Immune response
8. Best imaging for CT is gold standard b/c it detects blood flow and perfusion.
an intracranial he-
morrhage
1/8
