WGU Pathophysiology
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, WGU Pathophysiology
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1. A patient with a viral illness and severe Metabolic alkalosis---The patient's pH and CO2
vomiting has an elevated CO2 level and level are both elevating (moving in the same di-
a blood pH of 7.53. She is breathing rection). This indicates metabolic alkalosis. The
slowly. What condition does the patient CO2 level is high because her respiratory system
have? is attempting to compensate for the high pH by
exhaling less and retaining more CO2
2. What happens in a dehydrated patient? ADH levels increase and the RAAS is activat-
ed.---Osmoreceptors will stimulate the secretion
of ADH from the posterior pituitary. In addition,
low circulation stimulates the kidney to make
renin, and the RAAS will increase blood volume
and blood pressure by promoting the reabsorp-
tion of water and sodium in the kidney and
through vasoconstriction.
3. ACE (angiotensin converting enzymes) The decrease in the production of angiotensin
inhibitors are used to treat abnormal II keeps blood vessels more dilated. This lowers
blood pressure. They prevent the con- blood pressure.---Normally, the RAAS is activated
version of angiotensin I to angiotensin in response to low blood pressure, and its ac-
II. What explains how ACE inhibitors tions help to raise blood pressure. As part of this
work? response, angiotensin II is produced and causes
vasoconstriction. Vasoconstriction increases blood
pressure. Sometimes individuals with high blood
pressure are prescribed ACE inhibitors. Because
ACE inhibitors prevent the production of an-
giotensin II, blood vessels will remain more dilat-
ed, lowering blood pressure.
4. In diabetic ketoacidosis, what happens Because HCO3- is consumed, the anion gap in-
to the anion gap? creases.---The anion gap is the difference be-
tween measured cations (Na+ and K+) and mea-
| Complete Exam Study
Guide, Practice Questions,
Answers & ATI Review
Material
, WGU Pathophysiology
Study online at https://quizlet.com/_hz8fue
1. A patient with a viral illness and severe Metabolic alkalosis---The patient's pH and CO2
vomiting has an elevated CO2 level and level are both elevating (moving in the same di-
a blood pH of 7.53. She is breathing rection). This indicates metabolic alkalosis. The
slowly. What condition does the patient CO2 level is high because her respiratory system
have? is attempting to compensate for the high pH by
exhaling less and retaining more CO2
2. What happens in a dehydrated patient? ADH levels increase and the RAAS is activat-
ed.---Osmoreceptors will stimulate the secretion
of ADH from the posterior pituitary. In addition,
low circulation stimulates the kidney to make
renin, and the RAAS will increase blood volume
and blood pressure by promoting the reabsorp-
tion of water and sodium in the kidney and
through vasoconstriction.
3. ACE (angiotensin converting enzymes) The decrease in the production of angiotensin
inhibitors are used to treat abnormal II keeps blood vessels more dilated. This lowers
blood pressure. They prevent the con- blood pressure.---Normally, the RAAS is activated
version of angiotensin I to angiotensin in response to low blood pressure, and its ac-
II. What explains how ACE inhibitors tions help to raise blood pressure. As part of this
work? response, angiotensin II is produced and causes
vasoconstriction. Vasoconstriction increases blood
pressure. Sometimes individuals with high blood
pressure are prescribed ACE inhibitors. Because
ACE inhibitors prevent the production of an-
giotensin II, blood vessels will remain more dilat-
ed, lowering blood pressure.
4. In diabetic ketoacidosis, what happens Because HCO3- is consumed, the anion gap in-
to the anion gap? creases.---The anion gap is the difference be-
tween measured cations (Na+ and K+) and mea-
