NURl 201/l NUR201l Examl 1l –l Medical-
Surgicall Nursingl Il Guidel |l Fortisl (Latestl
2026/l 2027l Update)l 100%l Verifiedl
Questionsl &l Answersl |l Gradel A
Q:l Typesl ofl inflammation
Answer:
acute,l subacute,l chronic
Q:l acutel inflammation
Answer:
shortl terml responsel tol injury
-l Lastsl 2l tol 3l weeks,l usuallyl nol residuall damagel afterl healingl -l -l Neutrophils—
predominantl celll typel atl sitel ofl inflammation
Q:l subacutel inflammation
Answer:
Hasl samel featuresl asl acutel inflammationl butl persistsl longerl thanl 4l weeks.
Q:l chronicl inflammation
Answer:
Mayl lastl weeks,l months,l orl years
Injuriousl agentl persistsl orl repeatsl injuryl tol tissues
Predominantl celll types—lymphocytesl andl macrophages
Mayl resultl froml changesl inl immunel response(e.g.,l autoimmunel disease)
,Q:l Nursingl implementationl forl inflammation:
Answer:
Healthl promotion
Preventionl ofl injury
Adequatel nutritionl andl hydration
Earlyl recognitionl ofl inflammation
Immediatel treatment
Q:l Nursingl implementationl forl acutel carel inflammation:
Answer:
Observationl andl vitall signs
Immunosuppressed—classicl manifestationsl masked;
justl "don'tl feell well"
Q:l Feverl management:
Answer:
Moderatel fevers,l upl tol 103,l usuallyl producel fewl problems.l Considerl antipyreticl usel ifl
greatl discomfort.
Immunocompromisedl patientsl shouldl bel treatedl immediatelyl forl fever.
Feverl greaterl thanl 104l canl damagel bodyl cells
Olderl adultsl mayl havel bluntedl febrilel responsel tol infection
Spongel bathsl andl coolingl blanketsl mayl bel usefull ifl thermostaticl setl pointl loweredl withl
antipyretics;l avoidl shivering
Q:l antipyretic
Answer:
Medicationl usedl tol reducel al fever
,Q:l Drugl therapyl forl woundl care:
Answer:
Antipyreticl andl Anti-inflammatory
Salicylatesl (Aspirin)l
NSAIDSl (Ibuprofen)
Antipyretic
Acetaminophen
Antil Inflammatory
Corticosteroids
Q:l NSAIDsl helpl decrease
Answer:
inflammation,l pain,l andl fever.l
Theyl dol thisl byl blockingl thel body'sl productionl ofl al substancel calledl prostaglandin:l
Thisl substancel promotesl inflammationl andl feverl byl causingl thel releasel ofl variousl
chemicall signalsl inl yourl body.
Q:l Corticosteroidsl pharml namesl endl with
Answer:
-sone
-lone
Q:l RICE
Answer:
Rest:l O2l andl nutrientsl forl repair;l woundl healing
Ice:l (firstl 24l hours)—reducedl swellingl andl pain
Compression:l andl immobilization—reducel edemal andl stabilize
, Elevation:l reducel edemal andl pain
Q:l Healingl isl thel finall phasel ofl the
Answer:
inflammatoryl response.
Q:l Thel 2l majorl componentsl forl healing:
Answer:
1.l Regeneration:l
Replacel cellsl andl tissuesl withl samel type.
2.l Repair:
(mostl common)
Connectivel tissuel replacesl lostl cells
Primary,l secondary,l orl tertiaryl intention
Q:l Primaryl intentionl woundl healing
Answer:
woundsl thatl heall underl conditionsl ofl minimall tissuel loss.
Q:l secondaryl intentionl woundl healing
Answer:
-l Woundsl thatl occurl froml trauma,l ulceration,l andl infectionl havel extensivel tissuel loss.
-l Wide,l irregularl margins—edgesl cannotl bel approximated
-l Morel debris,l cells,l andl exudate—mayl needl debrided
-l Granulatesl froml edgesl inl andl froml bottoml up;largerl scars
Q:l Thel primaryl intentionl woundl healingl includesl 3l phases
Surgicall Nursingl Il Guidel |l Fortisl (Latestl
2026/l 2027l Update)l 100%l Verifiedl
Questionsl &l Answersl |l Gradel A
Q:l Typesl ofl inflammation
Answer:
acute,l subacute,l chronic
Q:l acutel inflammation
Answer:
shortl terml responsel tol injury
-l Lastsl 2l tol 3l weeks,l usuallyl nol residuall damagel afterl healingl -l -l Neutrophils—
predominantl celll typel atl sitel ofl inflammation
Q:l subacutel inflammation
Answer:
Hasl samel featuresl asl acutel inflammationl butl persistsl longerl thanl 4l weeks.
Q:l chronicl inflammation
Answer:
Mayl lastl weeks,l months,l orl years
Injuriousl agentl persistsl orl repeatsl injuryl tol tissues
Predominantl celll types—lymphocytesl andl macrophages
Mayl resultl froml changesl inl immunel response(e.g.,l autoimmunel disease)
,Q:l Nursingl implementationl forl inflammation:
Answer:
Healthl promotion
Preventionl ofl injury
Adequatel nutritionl andl hydration
Earlyl recognitionl ofl inflammation
Immediatel treatment
Q:l Nursingl implementationl forl acutel carel inflammation:
Answer:
Observationl andl vitall signs
Immunosuppressed—classicl manifestationsl masked;
justl "don'tl feell well"
Q:l Feverl management:
Answer:
Moderatel fevers,l upl tol 103,l usuallyl producel fewl problems.l Considerl antipyreticl usel ifl
greatl discomfort.
Immunocompromisedl patientsl shouldl bel treatedl immediatelyl forl fever.
Feverl greaterl thanl 104l canl damagel bodyl cells
Olderl adultsl mayl havel bluntedl febrilel responsel tol infection
Spongel bathsl andl coolingl blanketsl mayl bel usefull ifl thermostaticl setl pointl loweredl withl
antipyretics;l avoidl shivering
Q:l antipyretic
Answer:
Medicationl usedl tol reducel al fever
,Q:l Drugl therapyl forl woundl care:
Answer:
Antipyreticl andl Anti-inflammatory
Salicylatesl (Aspirin)l
NSAIDSl (Ibuprofen)
Antipyretic
Acetaminophen
Antil Inflammatory
Corticosteroids
Q:l NSAIDsl helpl decrease
Answer:
inflammation,l pain,l andl fever.l
Theyl dol thisl byl blockingl thel body'sl productionl ofl al substancel calledl prostaglandin:l
Thisl substancel promotesl inflammationl andl feverl byl causingl thel releasel ofl variousl
chemicall signalsl inl yourl body.
Q:l Corticosteroidsl pharml namesl endl with
Answer:
-sone
-lone
Q:l RICE
Answer:
Rest:l O2l andl nutrientsl forl repair;l woundl healing
Ice:l (firstl 24l hours)—reducedl swellingl andl pain
Compression:l andl immobilization—reducel edemal andl stabilize
, Elevation:l reducel edemal andl pain
Q:l Healingl isl thel finall phasel ofl the
Answer:
inflammatoryl response.
Q:l Thel 2l majorl componentsl forl healing:
Answer:
1.l Regeneration:l
Replacel cellsl andl tissuesl withl samel type.
2.l Repair:
(mostl common)
Connectivel tissuel replacesl lostl cells
Primary,l secondary,l orl tertiaryl intention
Q:l Primaryl intentionl woundl healing
Answer:
woundsl thatl heall underl conditionsl ofl minimall tissuel loss.
Q:l secondaryl intentionl woundl healing
Answer:
-l Woundsl thatl occurl froml trauma,l ulceration,l andl infectionl havel extensivel tissuel loss.
-l Wide,l irregularl margins—edgesl cannotl bel approximated
-l Morel debris,l cells,l andl exudate—mayl needl debrided
-l Granulatesl froml edgesl inl andl froml bottoml up;largerl scars
Q:l Thel primaryl intentionl woundl healingl includesl 3l phases
