NU 327 Pathophysiology Exam 6 Questions and
Answers with Complete Solutions (Graded A+) |
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Practice questions for this set
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Excessive medication
Fasciculations (especially around mouth)
Difficulty chewing, swallowing, speaking
Advancing muscle weakness
Increased secretions
Respiratory distress
Choose an answer
NHHC- Nonketotic hyperglycemic
1 Glucose 2
hyperosmolar coma
3 Glycolysis 4 Cholinergic Crisis
Don't know?
,Terms in this set (78)
Glucose broken down carbohydrates for energy; sugar
Glucagon hormone produced by alpha cells of pancreas.
Stimulates glycogenolysis and gluconeogenesis
in liver
Glycogen carbohydrate consists of glucose produced in
muscle and liver from stored glucose
Insulin hormone produced by beta cells of pancreas.
Energy metabolism
stimulates the diffusion of glucose into adipose
and muscle tissue and inhibits the production of
glucose by the liver
Glycolysis breakdown of sugar so cells can use
Glycogenolysis production of glucose from breakdown of
glycogen in liver and muscle tissue
Glycogenesis production of glycogen from glucose in liver in
muscle tissue
, Gluconeogenesis production of glucose from amino acids in liver
Fed state dominated by insulin
After eating, rise in blood glucose and insulin
Presence of insulin stimulates glucose diffusion
into adipose and muscle tissue and inhibits
production of glucose by liver
Glycolysis -energy use for cell
Glycogenesis-glycogen production muscle/liver
Fasting state dominated by glucagon
Glucose is produced
by glycogenolysis
By gluconeogenesis
Glucagon-responsible for 75% of glucose
production
Lipolysis is primary source of energy to muscles
Type 1 Diabetes Mellitus Thin
Absolute insulin deficiency- Loss of beta cells
Overproduction of glucagon by pancreatic alpha
cells
hypovolemia
diabetic ketoacidosis
lactic acid
Kussmaul respirations; fruity breath
Polydipsia, Polyuria, Polyphagia
Type 2 Diabetes Mellitus Obese, sedentary lifestyle, abdominal adipose
insulin resistance/insufficient insulin- beta cell
dysfunction
decreased number of insulin receptors
DKA uncommon
Polydipsia, Polyuria, Polyphagia
NHHC- Nonketotic hyperglycemic hyperosmolar
coma
Answers with Complete Solutions (Graded A+) |
New Update 2026
Save
Practice questions for this set
Learn 1 /7 Study with Learn
Excessive medication
Fasciculations (especially around mouth)
Difficulty chewing, swallowing, speaking
Advancing muscle weakness
Increased secretions
Respiratory distress
Choose an answer
NHHC- Nonketotic hyperglycemic
1 Glucose 2
hyperosmolar coma
3 Glycolysis 4 Cholinergic Crisis
Don't know?
,Terms in this set (78)
Glucose broken down carbohydrates for energy; sugar
Glucagon hormone produced by alpha cells of pancreas.
Stimulates glycogenolysis and gluconeogenesis
in liver
Glycogen carbohydrate consists of glucose produced in
muscle and liver from stored glucose
Insulin hormone produced by beta cells of pancreas.
Energy metabolism
stimulates the diffusion of glucose into adipose
and muscle tissue and inhibits the production of
glucose by the liver
Glycolysis breakdown of sugar so cells can use
Glycogenolysis production of glucose from breakdown of
glycogen in liver and muscle tissue
Glycogenesis production of glycogen from glucose in liver in
muscle tissue
, Gluconeogenesis production of glucose from amino acids in liver
Fed state dominated by insulin
After eating, rise in blood glucose and insulin
Presence of insulin stimulates glucose diffusion
into adipose and muscle tissue and inhibits
production of glucose by liver
Glycolysis -energy use for cell
Glycogenesis-glycogen production muscle/liver
Fasting state dominated by glucagon
Glucose is produced
by glycogenolysis
By gluconeogenesis
Glucagon-responsible for 75% of glucose
production
Lipolysis is primary source of energy to muscles
Type 1 Diabetes Mellitus Thin
Absolute insulin deficiency- Loss of beta cells
Overproduction of glucagon by pancreatic alpha
cells
hypovolemia
diabetic ketoacidosis
lactic acid
Kussmaul respirations; fruity breath
Polydipsia, Polyuria, Polyphagia
Type 2 Diabetes Mellitus Obese, sedentary lifestyle, abdominal adipose
insulin resistance/insufficient insulin- beta cell
dysfunction
decreased number of insulin receptors
DKA uncommon
Polydipsia, Polyuria, Polyphagia
NHHC- Nonketotic hyperglycemic hyperosmolar
coma
