URI NUR 213 FINAL QUESTIONS WITH DETAILED VERIFIED
ANSWERS (100% CORRECT ANSWERS) /ALREADY GRADED
atrophy Ans✓✓✓decrease in size of cells
hypertrophy Ans✓✓✓increase in size of cells
hyperplasia Ans✓✓✓increase in number of cells
dysplasia Ans✓✓✓abnormal change in the way cells look; often
reversible
metaplasia Ans✓✓✓reversible replacement of one cell type by another;
in response to chronic irritation or inflammation (i.e. GERD)
inflammatory response Ans✓✓✓vascular response: initial arteriolar
vasoconstriction, vasodilation, increased capillary permeability and
plasma leakage of protein rich fluids
phagocytes Ans✓✓✓monocytes and macrophages, promote the wound
healing process by phagocytosis
monocytes Ans✓✓✓produced in bone marrow, enter circulation,
migrate to inflammatory site where they develop into macrophages
,macrophages Ans✓✓✓typically arrive at the inflammatory site 24 hours
after neutrophils
phagocytosis Ans✓✓✓the process of WBCs that surround the foreign
material resulting in margination
healing by primary intention Ans✓✓✓no gap in tissue, simple
reapproximation of edges of wound, surgical incision type healing
healing by secondary intention Ans✓✓✓gap in tissue, prolonged healing
involving generation of granulation tissue and more complex tissue, scar
tissue formation
nociception Ans✓✓✓perception of pain
nociceptors Ans✓✓✓free nerve endings (pain receptors) found in skin,
muscle, CT, circulatory system, and abdominal/thoracic/pelvic viscera
neuroanatomy of pain Ans✓✓✓nociceptors, spinal cord, thalamus,
somatosensory cortex
types of acute pain Ans✓✓✓cutaneous, somatic, visceral
cutaneous pain Ans✓✓✓injury to skin or superficial tissues
, somatic pain Ans✓✓✓arises form superficial structures (skin, SC tissue,
periosteum, tendons, joints, blood vessels); localized or distributed along
dermatones; sharp, dull, aching or throbbing
visceral pain Ans✓✓✓pain in internal organs and lining of body
cavities, poorly localized; aching, gnawing, throbbing, or intermittent
cramping
neuropathic pain Ans✓✓✓pain that originates from the pathology of the
NS; primarily chronic; notable features include widespread, evidence of
sensory deficit, burning, shooting, shock-like or tingling
genetic information Ans✓✓✓stored in the structure of DNA
ribonucleic acid (RNA) Ans✓✓✓involved in actual synthesis of cellular
protein
proto-oncogenes Ans✓✓✓normal genes that control cell proliferation;
when they mutate it can lead to cancer
oncogenes Ans✓✓✓mutated genes inherited or acquired resulting in
uncontrolled proliferation
ANSWERS (100% CORRECT ANSWERS) /ALREADY GRADED
atrophy Ans✓✓✓decrease in size of cells
hypertrophy Ans✓✓✓increase in size of cells
hyperplasia Ans✓✓✓increase in number of cells
dysplasia Ans✓✓✓abnormal change in the way cells look; often
reversible
metaplasia Ans✓✓✓reversible replacement of one cell type by another;
in response to chronic irritation or inflammation (i.e. GERD)
inflammatory response Ans✓✓✓vascular response: initial arteriolar
vasoconstriction, vasodilation, increased capillary permeability and
plasma leakage of protein rich fluids
phagocytes Ans✓✓✓monocytes and macrophages, promote the wound
healing process by phagocytosis
monocytes Ans✓✓✓produced in bone marrow, enter circulation,
migrate to inflammatory site where they develop into macrophages
,macrophages Ans✓✓✓typically arrive at the inflammatory site 24 hours
after neutrophils
phagocytosis Ans✓✓✓the process of WBCs that surround the foreign
material resulting in margination
healing by primary intention Ans✓✓✓no gap in tissue, simple
reapproximation of edges of wound, surgical incision type healing
healing by secondary intention Ans✓✓✓gap in tissue, prolonged healing
involving generation of granulation tissue and more complex tissue, scar
tissue formation
nociception Ans✓✓✓perception of pain
nociceptors Ans✓✓✓free nerve endings (pain receptors) found in skin,
muscle, CT, circulatory system, and abdominal/thoracic/pelvic viscera
neuroanatomy of pain Ans✓✓✓nociceptors, spinal cord, thalamus,
somatosensory cortex
types of acute pain Ans✓✓✓cutaneous, somatic, visceral
cutaneous pain Ans✓✓✓injury to skin or superficial tissues
, somatic pain Ans✓✓✓arises form superficial structures (skin, SC tissue,
periosteum, tendons, joints, blood vessels); localized or distributed along
dermatones; sharp, dull, aching or throbbing
visceral pain Ans✓✓✓pain in internal organs and lining of body
cavities, poorly localized; aching, gnawing, throbbing, or intermittent
cramping
neuropathic pain Ans✓✓✓pain that originates from the pathology of the
NS; primarily chronic; notable features include widespread, evidence of
sensory deficit, burning, shooting, shock-like or tingling
genetic information Ans✓✓✓stored in the structure of DNA
ribonucleic acid (RNA) Ans✓✓✓involved in actual synthesis of cellular
protein
proto-oncogenes Ans✓✓✓normal genes that control cell proliferation;
when they mutate it can lead to cancer
oncogenes Ans✓✓✓mutated genes inherited or acquired resulting in
uncontrolled proliferation
