Test Bank for Rubin’s Pathology 7th Edition all chapters 1-34 with answers keys

TESTBANK n




RUBIN'S PATHOLOGY: CLINICOPATHOLOGIC FOUNDATIONS OF MEDICIN
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E 7TH EDITION
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DAVID S. STRAYER, EMANUEL RUBIN
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,TestnBanknRubin'snPathology:nClinicopathologicnFoundationsnofnMedicinen7thnEdition

TablenofnContents:
Chaptern1:nCellnAdaptation,nInjurynandnDeath
Chaptern2:nInflammation
Chaptern3:nRepair,nRegenerationnandnFibrosis
Chaptern4:nImmunopathology
Chaptern5:nNeoplasia
Chaptern6:nDevelopmentalnandnGeneticnDiseases
Chaptern7:nHemodynamicnDisorders
Chaptern8:nEnvironmentalnandnNutritionalnPathology
Chaptern9:nInfectiousnandnParasiticnDiseases
SectionnII:nPathogenesisnofnSystemicnConditionsnExpandablensection
Chaptern10:nAging
Chaptern11:nSystemicnAutoimmunenDiseases
Chaptern12:nSepsis
Chaptern13:nObesitynandnDiabetesnMellitus
Chaptern14:nThenPathologynofnPregnancy
Chaptern15:nThenAmyloidoses
SectionnIII:nDiseasesnofnIndividualnOrgannSystemsExpandablensection
Chaptern16:nBloodnVessels
Chaptern17:nThenHeart
Chaptern18:nThenRespiratorynSystem
Chaptern19:nThenGastrointestinalnTract
Chaptern20:nThenLivernandnBiliarynSystem
Chaptern21:nThenPancreas
Chaptern22:nThenKidney
Chaptern23:nThenLowernUrinarynTractnandnMalenReproductivenSystem
Chaptern24:nThenFemalenReproductivenSystemnandnPeritoneum
Chaptern25:nThenBreast
Chaptern26:nHematopathology
Chaptern27:nThenEndocrinenSystem
Chaptern28:nThenSkin
Chaptern29:nThenHeadnandnNeck
Chaptern30:nBones,nJointsnandnSoftnTissue
Chaptern31:nSkeletalnMusclenandnPeripheralnNervousnSystem
Chaptern32:nThenCentralnNervousnSystem
Chaptern33:nThenEye
Chaptern34:nForensicnPathology

,Rubin'snPathology:nClinicopathologicnFoundationsnofnM
edicineChaptern1:nCellnAdaptation,nInjurynandnDeath

Ischemianandnotherntoxicninjuriesnincreasenthenaccumulationnofnintracellularncalciumnasnanresult
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1. of:
A) releasenofnstoredncalciumnfromnthenmitochondria.
B) improvednintracellularnvolumenregulation.
C) decreasedninfluxnacrossnthencellnmembrane.
D) attractionnofncalciumntonfattyninfiltrates.
Thenpatientnisnfoundntonhavenliverndisease,nresultingninnthenremovalnofnanlobenofnhisnliver.
2. Adaptationntonthenreducednsizenofnthenlivernleadsnton_ ofnthenremainingnliverncells.
A) metaplasia
B) organnatrophy
C) compensatorynhyperplasia
D) physiologicnhypertrophy
Anpersonneatingnpeanutsnstartsnchokingnandncollapses.nHisnairwaynobstructionnisnpartiallyncleared
,nbutnhenremainsnhypoxicnuntilnhenreachesnthenhospital.nThenprolongedncellnhypoxia
3. causednancerebralninfarctionnandnresulting _ninnthenbrain.
A) caspasenactivation
B) coagulationnnecrosis
C) rapidnphagocytosis
D) proteinnp53ndeficiency
Bacterianandn virusesncausencellndamagenby ,nwhichnisnuniquenfromnthenintracellular
4. damagencausednbynotherninjuriousnagents.
A) disruptingnthensodium/potassiumnATPasenpump
B) interruptingnoxidativenmetabolismnprocesses
C) replicatingnandnproducingncontinuedninjury
D) decreasingnproteinnsynthesisnandnfunction
Thenpatientnhasnanprolongedninterruptionninnarterialnbloodnflowntonhisnleftnkidney,ncausing
5. hypoxicncellninjurynandnthenreleasenofnfreenradicals.nFreenradicalsndamagencellsnby:
A) destroyingnphospholipidsninnthencellnmembrane.
B) alteringnthenimmunenresponsenofnthencell.
C) disruptingncalciumnstorageninnthencell.
D) inactivationnofnenzymesnandnmitochondria.

, 6. Injuredncellsnhavenimpairednflownofnsubstancesnthroughnthencellnmembranenasnanresultnof:
A) increasednfatnload.
B) alterednpermeability.
C) alterednglucosenutilization.
D) increasednsurfacenreceptors.
7. Reversiblenadaptivenintracellularnresponsesnareninitiatednby:
A) stimulusnoverload.
B) geneticnmutations.
C) chemicalnmessengers.
D) mitochondrialnDNA.
8. Injuredncellsnbecomenverynswollennasnanresultnof:
A) increasedncellnproteinnsynthesis.
B) alteredncellnvolumenregulation.
C) passivenentrynofnpotassiumnintonthencell.
D) blebnformationninnthenplasmanmembrane.
Andiabeticnpatientnhasnimpairednsensation,ncirculation,nandnoxygenationnofnhisnfeet.nHenstepsnonnan
piecenofnglass,nthenwoundndoesnnotnheal,nandnthenareantissuenbecomesnnecrotic.nThennecrotic
9. cellndeathnisn characterizednby:
A) rapidnapoptosis.
B) cellularnrupture.
C) shrinkagenandncollapse.
D) chronicninflammation.
An99-year-oldnwomannhasnexperiencednthendeclinenofncellnfunctionnassociatednwithnage.nA
10. groupnofntheoriesn ofncellularnagingnfocusnonnprogrammed:
A) changesnwithngeneticninfluences.
B) eliminationnofncellnreceptornsites.
C) insufficientntelomerasenenzyme.
D) DNAnmutationnornfaultynrepair.
Ann89-year-
oldnfemalenpatientnhasnexperiencednsignificantndecreasesninnhernmobilitynandnstaminanduringnan3
-
weeknhospitalnstaynfornthentreatmentnofnanfemoralnheadnfracture.nWhichnofnthenfollowingnphenom
enanmostnlikelynaccountsnfornthenpatientsndecreaseninnmusclenfunction
11. thatnunderliesnhernreducednmobility?
A) Impairednmusclencellnmetabolismnresultingnfromnmetaplasia
B) Dysplasianasnanconsequencenofninflammationnduringnbonenremodeling
C) Disusenatrophynofnmusclen cellsnduringnanprolongednperiodnofnimmobility