Simulation Objectives
By the end of this simulation, students will:
1. Identify changes in client's condition and collaborate appropriately with health team members.
2. Perform/Verbalize an appropriate focused assessment for the clinical situation.
3. Demonstrate/Verbalize safe medication administration as appropriate.
4. Develop individualized plan of care and teaching with appropriate goals, interventions and evaluations.
Required Activities Prior to Simulation:
1. Review Lewis 12th text
a. Upper GI Problems
2. Review Skills Pertinent to this Simulation
a. Medication administration – IV push and IV continuous
b. IV medication administration using a carpuject
c. IV maintenance
3. Review SBAR Guidelines and Form.
4. Determine and review appropriate focused assessments.
5. TYPE and COMPLETE the Clinical Preparation Sheet below without the use of AI or ChatGPT type of websites. Put this in your own
words.
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, VALERIE JONES SIMULATION PREP SHEET
Date:________________ Student Name: __________________________ Assigned Simulated Patient: ____________________________
Noticing: Recognizing Cues
Age: Diagnosis: Provider: Isolation: IV Type: PMH:
Gender: Fall Risk Indicators: Location:
Length of Stay: Allergies:
Code Status: Fluid/Rate: Social history:
Mobility Needs:
Explain the history of present illness in narrative shift handoff format in your own words:
Valerie Jones, 78-year-old Hispanic female, admitted from home with a two-month history of epigastric discomfort that worsens after
meals. This morning, pain intensified and was followed by vomiting of dark, coffee-ground material, indicating a possible upper GI
bleed. She has been taking aspirin 325 mg daily since her husband’s recent death from a heart attack. Her daughter brought her to the
ED. Currently NPO, receiving IV fluids, esomeprazole infusion, and PRN IV morphine for pain. Awaiting GI consult and endoscopy.
Condition stable but requires close monitoring.
Define and describe the pathophysiology of the primary problem:
Peptic ulcers are open sores in the stomach or duodenal lining caused by erosion of the protective mucosal barrier. Normally, this
barrier shields the gastrointestinal tract from stomach acid and digestive enzymes. When compromised—due to factors like NSAID use
(e.g., aspirin), Helicobacter pylori infection, or severe stress—acid and pepsin penetrate the tissue, leading to ulcer formation.
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