This expertly curated Q&A document for NUR 370 Learning Module 3 features over 60 concise, exam-aligned questions and answers, focused on critical pathophysiology and pharmacologic concepts in tissue repair, infection, inflammation, autoimmune diseases, and antibiotic therapy. Tailored for BSN and ABSN nursing students, this guide supports understanding of complex disease processes and nursing interventions foundational to clinical and NCLEX success.
The wound healing section breaks down all phases:
Inflammatory phase (pathogen control, immune activation)
Proliferative phase (tissue rebuilding)
Maturation/remodeling phase (scar strengthening and remodeling)
It also identifies factors impairing healing: malnutrition, poor oxygenation, infection, foreign bodies, aging, and inflammatory dysfunction.
Infectious disease content includes:
Urinary Tract Infections (UTIs): symptoms (cloudy urine, dysuria, nocturia), anatomy (ureters, bladder, urethra), and interventions (hydration, antibiotic adherence, acidification of urine)
Cellulitis: deeper bacterial skin infection with swelling, redness, drainage
Sepsis and Septic Shock: progression from local infection to systemic response, symptoms (fever, AMS, hypotension), and treatment priorities (antibiotics, fluids, oxygen)
The module includes a microbiology primer on:
Gram-positive vs. gram-negative bacteria
Aerobic vs. anaerobic organisms
Bactericidal vs. bacteriostatic agents
Key antibiotic classes: penicillins, cephalosporins, lipoglycopeptides
A major section covers autoimmune diseases, particularly:
Rheumatoid Arthritis (RA): pathophysiology (pannus formation, TNF activation), symptoms, and drug treatments including DMARDs, NSAIDs, corticosteroids, antimalarials
Systemic Lupus Erythematosus (SLE): B-cell hyperactivity, triggers (stress, UV, hormones), and treatments (glucocorticoids, immunosuppressants, antimalarials)
Pharmacologic detail includes:
DMARDs (TNF blockers) – action and adverse effects
NSAIDs – COX-1 vs. COX-2 targeting, GI and renal risks
Glucocorticoids – adrenal suppression, anti-inflammatory effect, long-term complications
Ideal for students in:
BSN/ABSN nursing programs focused on pathophysiology, microbiology, and pharmacology
Preparing for NUR 370 assessments, midterms, and NCLEX prep covering infection, healing, immunity, and autoimmune disorders
Structured for clarity and exam retention, this document builds strong clinical foundations in wound care, infection control, inflammation, and immune-modulating therapies.
Keywords:
wound healing phases, inflammatory response, sepsis symptoms, septic shock, UTI treatment, cellulitis, gram-positive bacteria, antibiotics, DMARDs, rheumatoid arthritis, systemic lupus, glucocorticoids, NSAIDs, immune suppression, NUR 370
Content preview
NUR 370 LM 3
tissue regeneration - 🧠 ANSWER ✔✔replacement of connective tissue to
create a permanent scar
inflammatory phase (WH) - 🧠 ANSWER ✔✔prepares wound environment
for healing, all the things show up to get rid of the injurious thing and
control the infection
proliferate phase (WH) - 🧠 ANSWER ✔✔build new tissue to fill the wound
Maturational/Remodeling Phase (WH) - 🧠 ANSWER ✔✔continued
remodeling of scar tissue, takes the longest
Factors that promote wound healing - 🧠 ANSWER ✔✔Increased blood flow
done via capillary permeability and vasodilation, increased oxygen to
increase the blood flow
, Impaired wound healing - 🧠 ANSWER ✔✔Malnutrition
Impaired blood flow and O2,
Impaired inflammatory and immune responses
Infection
Wound Separation
Foreign Bodies
Aging Effects
Cellulitis - 🧠 ANSWER ✔✔-deeper skin infection caused by a bacterial
infection
-preexisting wounds can be portals
-red, swollen, tender plaque
-malodorous drainage
Urinary Tract Infection - 🧠 ANSWER ✔✔Bacterial infection, can affect
bladder (cystitis) or kidneys (pyelonephritis)
Symptoms: cloudy foul smelling urine, 100000 colony forming units/mL,
treated by antibiotics