Diabetes Mellitus (Bridge to NCLEX) Ch. 49
f9 f9 f9 f9 f9 f9
Polydipsiaf9andf9Poyuriaf9relatedf9tof9diabetesf9mellitusf9aref9primarilyf9duef9t
o
a.f9thef9releasef9off9ketonesf9fromf9cellsf9duringf9fatf9metabolism
b.f9fluidf9shiftsf9resultingf9fromf9thef9osmoticf9effectf9off9hyperglycemia.
c.f9damagef9tof9thef9kidneysf9fromf9exposuref9tof9highf9levelsf9off9glucose.
d.f9changesf9inf9RBCsf9resultingf9fromf9attachmentf9off9excessivef9glucosef9to
f9hemoglobin.f9-f9correctf9answer✔ ✔
b.f9fluidf9shiftsf9resultingf9fromf9thef9osmoticf9effectf9off9hyperglycemia.
Whichf9Statementf9wouldf9bef9correctf9forf9af9patientf9withf9typef92f9diabetesf9
whof9wasf9admittedf9tof9thef9hospitalf9withf9pneumonia?
a.f9thef9patientf9mustf9receivef9insulinf9therapyf9tof9preventf9ketoacidosis.
b.f9Thef9patientf9hasf9isletf9cellf9antibodiesf9thatf9havef9destroyedf9thef9pancrea
s'sf9abilityf9tof9producef9insulin.
c.f9Thef9patientf9hasf9minimalf9orf9absentf9endogenousf9insulinf9secretionf9and
f9requiresf9dailyf9insulinf9injections.
d.f9Thef9patientf9mayf9havef9sufficientf9endogenousf9insulinf9tof9preventf9keto
sisf9butf9isf9atf9riskf9forf9hyperosmolarf9hyperglycemicf9syndrome.f9-
f9correctf9answer✔ ✔
d.f9Thef9patientf9mayf9havef9sufficientf9endogenousf9insulinf9tof9preventf9ke
tosisf9butf9isf9atf9riskf9forf9hyperosmolarf9hyperglycemicf9syndrome.
f9 f9 f9 f9 f9 f9
Polydipsiaf9andf9Poyuriaf9relatedf9tof9diabetesf9mellitusf9aref9primarilyf9duef9t
o
a.f9thef9releasef9off9ketonesf9fromf9cellsf9duringf9fatf9metabolism
b.f9fluidf9shiftsf9resultingf9fromf9thef9osmoticf9effectf9off9hyperglycemia.
c.f9damagef9tof9thef9kidneysf9fromf9exposuref9tof9highf9levelsf9off9glucose.
d.f9changesf9inf9RBCsf9resultingf9fromf9attachmentf9off9excessivef9glucosef9to
f9hemoglobin.f9-f9correctf9answer✔ ✔
b.f9fluidf9shiftsf9resultingf9fromf9thef9osmoticf9effectf9off9hyperglycemia.
Whichf9Statementf9wouldf9bef9correctf9forf9af9patientf9withf9typef92f9diabetesf9
whof9wasf9admittedf9tof9thef9hospitalf9withf9pneumonia?
a.f9thef9patientf9mustf9receivef9insulinf9therapyf9tof9preventf9ketoacidosis.
b.f9Thef9patientf9hasf9isletf9cellf9antibodiesf9thatf9havef9destroyedf9thef9pancrea
s'sf9abilityf9tof9producef9insulin.
c.f9Thef9patientf9hasf9minimalf9orf9absentf9endogenousf9insulinf9secretionf9and
f9requiresf9dailyf9insulinf9injections.
d.f9Thef9patientf9mayf9havef9sufficientf9endogenousf9insulinf9tof9preventf9keto
sisf9butf9isf9atf9riskf9forf9hyperosmolarf9hyperglycemicf9syndrome.f9-
f9correctf9answer✔ ✔
d.f9Thef9patientf9mayf9havef9sufficientf9endogenousf9insulinf9tof9preventf9ke
tosisf9butf9isf9atf9riskf9forf9hyperosmolarf9hyperglycemicf9syndrome.